Too many compromises: survey research and the spectre of communism

2016 ◽  
Vol 8 (1) ◽  
pp. 197-214 ◽  
Author(s):  
Stefan Schwarzkopf
Keyword(s):  
Survey Research ◽  
High Pressures ◽  
Social Research ◽  
Marketing Research ◽  
Scientific Practices ◽  
Archival Research ◽  
Social Scientists ◽  
Content Type ◽  
The Way

Purpose – This paper aims to chart the influence of McCarthyism and of FBI surveillance practices on a number of prominent American social scientists, market researchers, opinion pollsters and survey research practitioners during the post-war years. Hitherto disparate sets of historical evidence on how Red Scare tactics influenced social researchers and marketing scientists are brought together and updated with evidence from original archival research. Design/methodology/approach – The paper draws on the existing secondary literature on how social research practitioners and social scientists reacted to the unusually high pressures on academic freedom during the McCarthy era. It supplements this review with evidence obtained from archival research, including declassified FBI files. The focus of this paper is set on prominent individuals, mainly Bernard Berelson, Samuel Stouffer, Hadley Cantril, Robert S. Lynd, Paul F. Lazarsfeld, Herta Herzog, Ernest Dichter, but also the Frankfurt School in exile. Findings – Although some of the historiography presents American social scientists and practitioners in the marketing research sector as victims of McCarthyism and FBI surveillance, it can also be shown that virtually all individuals in focus here also developed strategies of accommodation, compromise and even opportunism to benefit from the climate of suspicion brought about by the prevailing anti-Communism. Social implications – Anyone interested in questions about the morality of marketing, market research and opinion polling as part of the social sciences practiced in vivo will need to pay attention to the way these social-scientific practices became tarnished by the way prominent researchers accommodated and at times even abetted McCarthyism. Originality/value – Against the view of social scientists as harassed academic minority, evidence is presented in this paper which shows American social scientists who researched market-related phenomena, like media, voters choices and consumer behaviour, in a different light. Most importantly, this paper for the first time presents archival evidence on the scale of Paul F. Lazarsfeld’s surveillance by the FBI.

Survey Research

2021 ◽  
pp. 272-277
Author(s):  
Jean-Frédéric Morin ◽  
Christian Olsson ◽  
Ece Özlem Atikcan
Keyword(s):  
Data Collection ◽  
Survey Research ◽  
Social Research ◽  
Marketing Research ◽  
Active Role ◽  
Structured Interviews ◽  
Collection Method ◽  
Communication Approach ◽  
Public Opinion Polls ◽  
Opinion Polls

This chapter discusses survey research. Surveys are a very common method of data collection used by many social researchers. As such, they are used in public opinion polls to gauge political trends and trait, but also in marketing research examining consumer behaviour and feedback. Surveys are also a common data collection method in many social research projects. They are further used to evaluate needs, processes, and outcomes. Importantly, surveys are a unidirectional communication approach to collect data, which is very different from observational methods, semi-structured and structured interviews, or other types of data collection where the researcher takes an active role. Specifically, using surveys, participants are presented with a set of instructions and predetermined questions. The researcher is not expected to engage in any participatory interaction or in-depth conversation with participants.

Co-workers’ perceptions of and reactions to employee’s involuntary demotion

2019 ◽  
Vol 41 (4) ◽  
pp. 740-757 ◽  
Author(s):  
Sophie Hennekam ◽  
Subramaniam Ananthram ◽  
Steve McKenna
Keyword(s):  
Design Methodology ◽  
Emotional Responses ◽  
Emotional Reaction ◽  
Content Type ◽  
Informational Justice ◽  
Behavioural Responses ◽  
Behavioural Reaction ◽  
Three Stages ◽  
Depth Interviews ◽  
The Way

Purpose The purpose of this paper is to investigate how individuals perceive and react to the involuntary demotion of a co-worker in their organisation. Design/methodology/approach The authors draw on 46 semi-structured in-depth interviews (23 dyads) with co-workers of demoted individuals. Findings The findings suggest that an individual’s observation of the demotion of a co-worker has three stages: their perception of fairness, their emotional reaction and their behavioural reaction. The perception of fairness concerned issues of distributive, procedural, interpersonal and informational justice. The emotional responses identified were feelings of disappointment/disillusion, uncertainty, vulnerability and anger. Finally, the behavioural reactions triggered by their emotional responses included expressions of voice, loyalty, exit and adaptation. Originality/value Perceptions of (in)justice perpetrated on others stimulate emotional and behavioural responses, which impacts organisational functioning. Managers should therefore pay attention to the way a demotion is perceived, not only by those directly concerned, but also by co-workers as observers.

Genes, cognition, and social behavior: Next steps for foundations and researchers

2011 ◽  
Vol 30 (2) ◽  
pp. 65-87 ◽  
Author(s):  
Arthur Lupia
Keyword(s):  
Social Behavior ◽  
Political Science ◽  
National Science Foundation ◽  
Scientific Practices ◽  
Social Scientists ◽  
Research Areas ◽  
Unpublished Report ◽  
Summary Statement ◽  
The One ◽  
Scientific Value

Editor's note This well circulated but heretofore unpublished report is the summary statement of an interdisciplinary meeting of scholars convened by the National Science Foundation in Arlington, Virginia on June 28, 2010. The workshop, which was funded by the NSF's Political Science Program (Social, Behavioral & Economic Sciences Grant #1037831), was convened to answer two compelling questions: Are studies of social behavior that build from discoveries about genes and/or cognition of greater social and scientific value than studies of the same topics that ignore such factors? And, how can fundable research on genes, cognition, and politics generate transformative scientific practices, infrastructure, and findings of high social value? Assembled for the workshop were a group of scholars representing diverse yet increasingly connected research areas, including genetics, cognitive science and neuroscience, decision making and risk analysis, economics, political science, and sociology. The resulting report outlines the substantial challenges facing interdisciplinary research but also describes the considerable contributions to knowledge that could result from sustained collaborations between biologists, geneticists, and brain scientists on the one hand and social scientists on the other. Following this main report are three white papers by Jeremy Freese. Elizabeth Hammock, and Rose McDermott, which address importmant considerations related to the discussion. For a download of the full report, see http://www.isr.umich.edu.cps/workshop.Welcome.html.

Why they shared: recovering early arguments for sharing social scientific data

2020 ◽  
Vol 33 (2) ◽  
pp. 101-119
Author(s):  
Emily Hauptmann
Keyword(s):  
Social Science ◽  
Data Sharing ◽  
Financial Stability ◽  
Social Research ◽  
Scientific Data ◽  
Social Scientists ◽  
Ethical Imperative ◽  
Social Scientific ◽  
The University ◽  
University Consortium

ArgumentMost social scientists today think of data sharing as an ethical imperative essential to making social science more transparent, verifiable, and replicable. But what moved the architects of some of the U.S.’s first university-based social scientific research institutions, the University of Michigan’s Institute for Social Research (ISR), and its spin-off, the Inter-university Consortium for Political and Social Research (ICPSR), to share their data? Relying primarily on archived records, unpublished personal papers, and oral histories, I show that Angus Campbell, Warren Miller, Philip Converse, and others understood sharing data not as an ethical imperative intrinsic to social science but as a useful means to the diverse ends of financial stability, scholarly and institutional autonomy, and epistemological reproduction. I conclude that data sharing must be evaluated not only on the basis of the scientific ideals its supporters affirm, but also on the professional objectives it serves.

scholarly journals Mass spectrometry based metabolomics of volume-restricted in-vivo brain samples: actual status and the way forward

2021 ◽  
pp. 116365
Author(s):  
Marlien van Mever ◽  
Karen Segers ◽  
Debby Mangelings ◽  
Thomas Hankemeier ◽  
Yvan Vander Heyden ◽  
...  
Keyword(s):  
Mass Spectrometry ◽  
The Way

scholarly journals MetR-Regulated Vibrio cholerae Metabolism Is Required for Virulence

mBio ◽  
2012 ◽  
Vol 3 (5) ◽  
Author(s):  
Ryan W. Bogard ◽  
Bryan W. Davies ◽  
John J. Mekalanos
Keyword(s):  
Amino Acid ◽  
Vibrio Cholerae ◽  
Virulence Factor ◽  
Current Knowledge ◽  
Intestinal Colonization ◽  
Wild Type ◽  
Pathogenic Potential ◽  
Content Type ◽  
Mouse Intestine

ABSTRACTLysR-type transcriptional regulators (LTTRs) are the largest, most diverse family of prokaryotic transcription factors, with regulatory roles spanning metabolism, cell growth and division, and pathogenesis. Using a sequence-defined transposon mutant library, we screened a panel ofV. choleraeEl Tor mutants to identify LTTRs required for host intestinal colonization. Surprisingly, out of 38 LTTRs, only one severely affected intestinal colonization in the suckling mouse model of cholera: the methionine metabolism regulator, MetR. Genetic analysis of genes influenced by MetR revealed thatglyA1andmetJwere also required for intestinal colonization. Chromatin immunoprecipitation of MetR and quantitative reverse transcription-PCR (qRT-PCR) confirmed interaction with and regulation ofglyA1, indicating that misregulation ofglyA1is likely responsible for the colonization defect observed in themetRmutant. TheglyA1mutant was auxotrophic for glycine but exhibited wild-type trimethoprim sensitivity, making folate deficiency an unlikely cause of its colonization defect. MetJ regulatory mutants are not auxotrophic but are likely altered in the regulation of amino acid-biosynthetic pathways, including those for methionine, glycine, and serine, and this misregulation likely explains its colonization defect. However, mutants defective in methionine, serine, and cysteine biosynthesis exhibited wild-type virulence, suggesting that these amino acids can be scavenged in vivo. Taken together, our results suggest that glycine biosynthesis may be required to alleviate an in vivo nutritional restriction in the mouse intestine; however, additional roles for glycine may exist. Irrespective of the precise nature of this requirement, this study illustrates the importance of pathogen metabolism, and the regulation thereof, as a virulence factor.IMPORTANCEVibrio choleraecontinues to be a severe cause of morbidity and mortality in developing countries. Identification ofV. choleraefactors critical to disease progression offers the potential to develop or improve upon therapeutics and prevention strategies. To increase the efficiency of virulence factor discovery, we employed a regulator-centric approach to multiplex our in vivo screening capabilities and allow whole regulons inV. choleraeto be interrogated for pathogenic potential. We identified MetR as a new virulence regulator and serine hydroxymethyltransferase GlyA1 as a new MetR-regulated virulence factor, both required byV. choleraeto colonize the infant mouse intestine. Bacterial metabolism is a prerequisite to virulence, and current knowledge of in vivo metabolism of pathogens is limited. Here, we expand the known role of amino acid metabolism and regulation in virulence and offer new insights into the in vivo metabolic requirements ofV. choleraewithin the mouse intestine.

Meet me halfway: Are in vitro 3D cancer models on the way to replace in vivo models for nanomedicine development?

2021 ◽  
Author(s):  
Sabina Pozzi ◽  
Anna Scomparin ◽  
Sahar Israeli-Dangoor ◽  
Daniel Rodriguez ◽  
Paula Ofek ◽  
...  
Keyword(s):  
In Vivo Models ◽  
Cancer Models ◽  
The Way

scholarly journals Interactions and Localization of Escherichia coli Error-Prone DNA Polymerase IV after DNA Damage

2015 ◽  
Vol 197 (17) ◽  
pp. 2792-2809 ◽  
Author(s):  
Sarita Mallik ◽  
Ellen M. Popodi ◽  
Andrew J. Hanson ◽  
Patricia L. Foster
Keyword(s):  
Dna Damage ◽  
Dna Polymerase ◽  
Dna Helicase ◽  
Dna Lesions ◽  
Replication Forks ◽  
Content Type ◽  
Pol Iv ◽  
Dna Polymerase Iv ◽  
Stalled Replication Forks

ABSTRACTEscherichia coli's DNA polymerase IV (Pol IV/DinB), a member of the Y family of error-prone polymerases, is induced during the SOS response to DNA damage and is responsible for translesion bypass and adaptive (stress-induced) mutation. In this study, the localization of Pol IV after DNA damage was followed using fluorescent fusions. After exposure ofE. colito DNA-damaging agents, fluorescently tagged Pol IV localized to the nucleoid as foci. Stepwise photobleaching indicated ∼60% of the foci consisted of three Pol IV molecules, while ∼40% consisted of six Pol IV molecules. Fluorescently tagged Rep, a replication accessory DNA helicase, was recruited to the Pol IV foci after DNA damage, suggesting that thein vitrointeraction between Rep and Pol IV reported previously also occursin vivo. Fluorescently tagged RecA also formed foci after DNA damage, and Pol IV localized to them. To investigate if Pol IV localizes to double-strand breaks (DSBs), an I-SceI endonuclease-mediated DSB was introduced close to a fluorescently labeled LacO array on the chromosome. After DSB induction, Pol IV localized to the DSB site in ∼70% of SOS-induced cells. RecA also formed foci at the DSB sites, and Pol IV localized to the RecA foci. These results suggest that Pol IV interacts with RecAin vivoand is recruited to sites of DSBs to aid in the restoration of DNA replication.IMPORTANCEDNA polymerase IV (Pol IV/DinB) is an error-prone DNA polymerase capable of bypassing DNA lesions and aiding in the restart of stalled replication forks. In this work, we demonstratein vivolocalization of fluorescently tagged Pol IV to the nucleoid after DNA damage and to DNA double-strand breaks. We show colocalization of Pol IV with two proteins: Rep DNA helicase, which participates in replication, and RecA, which catalyzes recombinational repair of stalled replication forks. Time course experiments suggest that Pol IV recruits Rep and that RecA recruits Pol IV. These findings providein vivoevidence that Pol IV aids in maintaining genomic stability not only by bypassing DNA lesions but also by participating in the restoration of stalled replication forks.

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