scholarly journals TRIVALENT CHROMIUM (CR+3) IN DIETARY CARBOHYDRATE AND ITS EFFECT ON THE GROWTH OF COMMONLY CULTIVATED FISH

2015 ◽  
Vol 78 (4-2) ◽  
Author(s):  
Subandiyono Subandiyono ◽  
Sri Hastuti
Keyword(s):  
Energy Source ◽  
Blood Glucose ◽  
Dietary Protein ◽  
Feeding Habits ◽  
Somatic Growth ◽  
Trivalent Chromium ◽  
Fish Growth ◽  
Dietary Carbohydrate ◽  
Carnivorous Fish ◽  
Glucose Metabolic Rate

Trivalent chromium (Cr+3) is an essential trace mineral for fish bio-physiological functions.  Researches on Cr+3  in the form of organic compound indicated that the mineral affected the bio-activity of insulin, the blood glucose influx, and subsequently the blood glucose metabolic rate. By increasing the blood glucose metabolism, dietary carbohydrate will be more efficiently used as a main energy source, thereby, dietary protein couldbe efficiently retained as for somatic growth.  Researches on various feeding habits of fish (e.g. gouramy-herbivorous fish, tilapia-omnivorous fish, and catfish-carnivorous fish) showed that dietary Cr+3 in certain amount increased diet utilization and the fish growth.  

Has fructose a place in the diabetic diet?

1980 ◽  
Vol 18 (17) ◽  
pp. 67-68
Keyword(s):  
Energy Source ◽  
Blood Glucose ◽  
Diabetic Complications ◽  
Artificial Sweeteners ◽  
Diabetic Diet ◽  
Commercial Process

Since fructose is an energy source and is not a sugar involved in diabetes, it was suggested about 40 years ago that it might be a useful alternative to glucose. Interest in the question1–3 has been revived by development of a commercial process for making fructose from starch, by the recognition that artificial sweeteners are potentially hazardous, and by a new awareness that control of blood glucose is necessary to prevent long-term diabetic complications.4

scholarly journals A three dimensional, full life cycle, anchovy and sardine model for the North Aegean Sea (Eastern Mediterranean): Validation, sensitivity and climatic scenario simulations

2021 ◽  
Vol 22 (3) ◽  
pp. 653
Author(s):  
ATHANASIOS GKANASOS ◽  
EUDOXIA SCHISMENOU ◽  
KOSTAS TSIARAS ◽  
STYLIANOS SOMARAKIS ◽  
MARIANNA GIANNOULAKI ◽  
...  
Keyword(s):  
Temperature Increase ◽  
Eastern Mediterranean ◽  
Larval Growth ◽  
Somatic Growth ◽  
Aegean Sea ◽  
Fish Growth ◽  
Prey Concentration ◽  
The North ◽  
North Aegean ◽  
North Aegean Sea

We present the development of a 3D full-lifecycle, individual-based model (IBM) for anchovy and sardine, online coupled to an existing hydrodynamic/biogeochemical low-trophic level (LTL) model for the North Aegean Sea. It was built upon an existing 1D model for the same species and area, with the addition of a horizontal movement scheme. In the model, both species evolve from the embryonic stage (egg+yolk sac larva) to the larval, juvenile, and adult stages. Somatic growth is simulated with the use of a “Wisconsin” type bioenergetics model and fish populations with an adaptation of the ‘super individuals’ (SI) approach. For the reference simulation and model calibration, in terms of fish growth and population biomass, the 2000-2010 period was selected. Interannual biomass variability of anchovy was successfully represented by the model, while the simulated biomass of sardine exhibited low variability and did not satisfactorily reproduce the observed interannual variability from acoustic surveys. The spatial distribution of both species’ biomass was in relatively good agreement with field data. Additional single-species simulations revealed that species compete for food resources. Temperature sensitivity experiments showed that both species reacted negatively to a temperature increase. Anchovy, in particular, was more affected since its spawning and larval growth periods largely overlap with the period of maximum yearly temperature and low prey concentration. Finally, simulation experiments using IPCC climatic scenarios showed that the predicted temperature increase and zooplankton concentration decrease in the future will negatively affect anchovy, resulting in sardine prevalence.

scholarly journals Decoupling of otolith and somatic growth during anadromous migration in a northern salmonid

2019 ◽  
Vol 76 (11) ◽  
pp. 1940-1953 ◽  
Author(s):  
Christie M. Morrison ◽  
Mélodie Kunegel-Lion ◽  
Colin P. Gallagher ◽  
Rick J. Wastle ◽  
Ellen V. Lea ◽  
...  
Keyword(s):  
Life History ◽  
Critical Time ◽  
Somatic Growth ◽  
Fish Growth ◽  
Calcium Deposition ◽  
Fish Length ◽  
Length Relationship ◽  
Anadromous Migration ◽  
Back Calculation ◽  
Size At Age

We assessed the fish length – otolith length relationship (FL–OL) in Dolly Varden (Salvelinus malma malma) to verify proportional growth. A decoupling was detected during first ocean migration where fish growth was occurring at a greater rate than otolith growth. Because of this decoupling, the application of traditional back-calculation models overestimated the size-at-age in premigratory char. We developed modified back-calculation equations from existing traditional models to account for this decoupling based on discontinuous piecewise regressions. The new biological intercept breakpoint method (BI–BP) provided the most accurate representation of fish size-at-age throughout all life history stages when compared with known size-at-capture values in fish. The decoupling indicates that factors other than somatic growth are important for otolith accretion. Physiological changes during smoltification likely alter calcium uptake and thereby affect calcium deposition rates on otoliths during this short but biologically critical time period of life history. It is probable that species exhibiting similar complex ontogenetic shifts in life history will likely exhibit decoupling to some extent in the FL–OL relationship.

scholarly journals Improving Dietary Protein Quality Reduces the Negative Effects of Physical Inactivity on Body Composition and Muscle Function

2019 ◽  
Vol 74 (10) ◽  
pp. 1605-1611 ◽  
Author(s):  
Emily J Arentson-Lantz ◽  
Elfego Galvan ◽  
Jennifer Ellison ◽  
Adam Wacher ◽  
Douglas Paddon-Jones
Keyword(s):  
Body Composition ◽  
Blood Glucose ◽  
G Protein ◽  
Dietary Protein ◽  
Physical Inactivity ◽  
Protein Quality ◽  
Bed Rest ◽  
Peak Torque ◽  
Knee Extension ◽  
Negative Effects

Abstract Background Brief periods of physical inactivity can compromise muscle health. Increasing dietary protein intake is potentially beneficial but complicated by difficulties reconciling anabolic potential with a realistic food volume and energy intake. We sought to determine whether increasing dietary protein quality could reduce the negative effects of physical inactivity. Methods Twenty healthy, older men and women completed 7 days of bed rest followed by 5 days of rehabilitation. Volunteers consumed a mixed macronutrient diet (MIXED: N = 10; 68 ± 2 years; 1,722 ± 29 kcal/day; 0.97 ± 0.01 g protein/kg/day) or an isoenergetic, whey-augmented, higher protein quality diet (WHEY: N = 10; 69 ± 1 years; 1,706 ± 23 kcal/day; 0.90 ± 0.01 g protein/kg/day). Outcomes included body composition, blood glucose, insulin, and a battery of physical function tests. Results During bed rest, both groups experienced a 20% reduction in knee extension peak torque (p < .05). The WHEY diet partially protected leg lean mass (−1,035 vs. −680 ± 138 g, MIXED vs. WHEY; p = .08) and contributed to a greater loss of body fat (−90 vs. −233 ± 152 g, MIXED vs. WHEY; p < .05). Following rehabilitation, knee extension peak torque in the WHEY group fully recovered (−10.0 vs. 2.2 ± 4.1 Nm, MIXED vs. WHEY; p = .05). Blood glucose, insulin, aerobic capacity, and Short Physical Performance Battery (SPPB) changes were similar in both dietary conditions (p > .05). Conclusions Improving protein quality without increasing total energy intake has the potential to partially counter some of the negative effects of bed rest in older adults.

scholarly journals Incorporating temporal heterogeneity in environmental conditions into a somatic growth model

2017 ◽  
Vol 74 (3) ◽  
pp. 316-326 ◽  
Author(s):  
M.C. Dzul ◽  
C.B. Yackulic ◽  
J. Korman ◽  
M.D. Yard ◽  
J.D. Muehlbauer
Keyword(s):  
Food Availability ◽  
Growth Model ◽  
Environmental Conditions ◽  
Colorado River ◽  
Growth Models ◽  
Somatic Growth ◽  
Environmental Data ◽  
Fish Growth ◽  
Positive Temperature ◽  
Environmental Controls

Evaluating environmental effects on fish growth can be challenging because environmental conditions may vary at relatively fine temporal scales compared with sampling occasions. Here we develop a Bayesian state-space growth model to evaluate effects of monthly environmental data on growth of fish that are observed less frequently (e.g., from mark–recapture data where time between captures can range from months to years). We assess effects of temperature, turbidity, food availability, flow variability, and trout abundance on subadult humpback chub (Gila cypha) growth in two rivers, the Colorado River (CR) and the Little Colorado River (LCR), and we use out-of-sample prediction to rank competing models. Environmental covariates explained a high proportion of the variation in growth in both rivers; however, the best growth models were river-specific and included either positive temperature and turbidity duration effects (CR) or positive temperature and food availability effects (LCR). Our approach to analyzing environmental controls on growth should be applicable in other systems where environmental data vary over relatively short time scales compared with animal observations.

scholarly journals The nature of the growth pattern and of the metabolic response to fasting in the rat are dependent upon the dietary protein and folic acid intakes of their pregnant dams and post-weaning fat consumption

2008 ◽  
Vol 99 (3) ◽  
pp. 540-549 ◽  
Author(s):  
Graham C. Burdge ◽  
Karen A. Lillycrop ◽  
Alan A. Jackson ◽  
Peter D. Gluckman ◽  
Mark A. Hanson
Keyword(s):  
Folic Acid ◽  
Dietary Protein ◽  
Metabolic Response ◽  
Somatic Growth ◽  
Metabolic Phenotype ◽  
Folic Acid Intake ◽  
Pregnant Rats ◽  
Acid Intake ◽  
Fat Consumption ◽  
Opposing Effects

The nutritional cues which induce different phenotypes from a single genotype in developing offspring are poorly understood. How well prenatal nutrient availability before birth predicts that after birth may also determine the offspring's response to later metabolic challenge. We investigated the effect of feeding pregnant rats diets containing protein at 180 g/kg (Control) or 90 g/kg (protein-restricted, PR) and either 1 or 5 mg folic acid/kg on growth and metabolic response to fasting in their offspring, and also the effect of diets with different fat contents (40 g/kg (Fat4) or 100 g/kg (Fat10)) after weaning. Offspring of dams fed the PR diet with 5 mg/kg folic acid were significantly lighter than other offspring. The PR offspring fed the Fat4diet had lower plasma TAG than the Control offspring, but this relationship was reversed when offspring were fed Fat10. Increasing the folic acid content of the Control or PR maternal diets induced opposing effects on plasma TAG, NEFA, β-hydroxybutyrate and glucose concentrations in offspring fed Fat4. The effect was accentuated in offspring fed the Fat10diet such that these metabolites were increased in the Control offspring, but reduced in the PR offspring. These data show for the first time that maternal dietary folic acid intake alters offspring phenotype depending upon dietary protein intake, and that this effect is modified by fat intake after weaning. Prevention by increased folic acid intake of an altered metabolic phenotype by maternal protein-restriction may be at the expense of somatic growth.

Feeding frequency and protein metabolism

1963 ◽  
Vol 205 (1) ◽  
pp. 71-78 ◽  
Author(s):  
C. Cohn ◽  
D. Joseph ◽  
L. Bell ◽  
A. Oler
Keyword(s):  
Young Adult ◽  
Protein Content ◽  
Dietary Protein ◽  
Protein Metabolism ◽  
Feeding Habits ◽  
Male Rats ◽  
Feeding Frequency ◽  
Ad Libitum ◽  
Total Body ◽  
Urinary Nitrogen

Normal young adult male rats were allowed to eat a diet of a specified protein content ad libitum, or were pair-force-fed the same diet twice a day. At the end of 14 days of controlled feeding, the animals were killed and analyzed for total body lipids. These analyses showed that the fat content of the force-fed animals, relative to those that ate ad libitum, became progressively greater as the dietary protein was increased in quantity from 0 to 67% of the diet. Furthermore, it was observed that force-fed animals excreted about 37% more urea nitrogen over a 7-day period than pair-fed animals eating ad libitum. Abruptly reversing the feeding habits of the rats immediately reversed the relative amounts of urinary nitrogen they excreted. The results of both types of experiments suggest that the intermediary metabolism of protein is related to feeding frequency (or load of absorbed nutrients to be metabolized per unit time). With fewer feedings but with total 24-hr intake constant, less dietary protein appears to participate in protein anabolic reactions since larger amounts of nitrogen are lost in the urine.

Embryonic steroids control developmental programming of energy balance

Endocrinology ◽  
2021 ◽  
Author(s):  
Meng-Chun Monica Shih ◽  
Chen-Che Jeff Huang ◽  
Hsueh-Ping Chu ◽  
Nai-Chi Hsu ◽  
Bon-chu Chung
Keyword(s):  
Energy Source ◽  
Blood Glucose ◽  
Energy Homeostasis ◽  
De Novo ◽  
Glycogen Synthesis ◽  
Glucose Production ◽  
Liver Glycogen ◽  
Glycogen Storage ◽  
Expression Of Genes ◽  
Major Energy Source

Abstract Glucose is a major energy source for growth. At birth, neonates must change their energy source from maternal supply to its own glucose production. The mechanism of this transition has not been clearly elucidated. To evaluate the possible roles of steroids in this transition, here we examine the defects associated with energy production of a mouse line that cannot synthesize steroids de novo due to the disruption of its Cyp11a1 gene. The Cyp11a1 null embryos had insufficient blood insulin, and failed to store glycogen in the liver since embryonic day 16.5. Their blood glucose dropped soon after maternal deprivation, and the expression of hepatic gluconeogenic and glycogenic genes were reduced. Insulin was synthesized in the mutant fetal pancreas, but failed to be secreted. Maternal glucocorticoid supply rescued the amounts of blood glucose, insulin, and liver glycogen in the fetus, but did not restore expression of genes for glycogen synthesis, indicating the requirement of de novo glucocorticoid synthesis for glycogen storage. Thus, our investigation of Cyp11a1 null embryos reveals that the energy homeostasis is established before birth, and fetal steroids are required for the regulation of glycogen synthesis, hepatic gluconeogenesis and insulin secretion at the fetal stage.

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