Left Ventricular Myxoma Producing Cardiac Failure

2013 ◽  
Vol 16 (1) ◽  
pp. 57 ◽  
Author(s):  
Erdal Simsek ◽  
Serkan Durdu ◽  
Bledar Hodo ◽  
Levent Yazicioglu ◽  
Adnan Uysalel
Keyword(s):  
Heart Failure ◽  
Left Ventricle ◽  
Rare Complication ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Chronic Obstructive ◽  
Cardiac Tumors ◽  
Obstructive Pulmonary Disease ◽  
Mobile Mass ◽  
Transaortic Approach

<p><b>Introduction:</b> Seventy-five percent of primary cardiac tumors are benign, and most are myxomas. Seventy-five percent of myxomas originate from the left atrium, and 2.5% arise from the left ventricle. Heart failure is a rare complication of myxoma.</p><p><b>Case:</b> A 54-year-old male patient with chronic obstructive pulmonary disease was admitted to the pulmonology department with a diagnosis of pneumonia and congestive heart failure during hospitalization. An echocardiography evaluation revealed a mobile mass (3.3 cm X 1.2 cm) in the left ventricle. The measured ejection fraction was 22%. Transthoracic and transesophageal echocardiography and magnetic resonance imaging examinations confirmed the presence of a myxoma in the left ventricle. The myxoma was a hanging mass with a stalk on the interventricular septum near the anterior mitral valve annulus. We visualized the gelatinous fragile mass on the septum; we then extracted the myxoma via a transaortic approach with the patient on cardiopulmonary bypass. The patient was discharged 10 days after surgery.</p><p><b>Discussion:</b> Myxoma is treated by early surgical resection because of the potential for serious complications. Left ventricular myxomas have been reported to lead to a silent heart failure. This case is important because of its location and the patient's resultant heart failure.</p>

scholarly journals A Tale of Two Specialties: Differences between Heart Failure Patients Admitted to Internal Medicine and Cardiology

2016 ◽  
Vol 8 (1) ◽  
Author(s):  
Vanja Petrovic MD ◽  
Sirar Ibrahim MD ◽  
Valerie A. Palda MD MSc
Keyword(s):  
Internal Medicine ◽  
Heart Failure ◽  
Complex Disease ◽  
Systolic Function ◽  
Left Ventricular Systolic Function ◽  
Left Ventricular ◽  
Chronic Obstructive ◽  
Obstructive Pulmonary Disease ◽  
Risk Of Mortality ◽  
Clinical Profiles

The authors described and compared the clinical profiles of patients hospitalized with heart failure (HF) to internal medicine (IM) and cardiology services. Data on age, gender, length of stay, and left ventricular systolic function, as well as specific co-morbidities and clinical parameters, were recorded retrospectively to allow the assessment of provider-related differences in HF populations.IM patients were significantly older, more likely to be female, and more likely to have preserved left ventricular function. IM patients also suffered a significantly greater number of co-morbidities, in particular chronic obstructive pulmonary disease, pneumonia, other lung diseases, thromboembolic disease, anemia, and arthritis. Finally, significantly more IM patients had a high risk of mortality at the time of hospital admission compared with cardiology patients. In summary, IM patients were older, “sicker,” and more likely to die within the next year. Complex disease and advanced age may affect outcomes, therapeutic strategies, and impact on diagnostic accuracy.  

scholarly journals Different Impact of Beta-Blockers on Long-Term Mortality in Heart Failure Patients with and without Chronic Obstructive Pulmonary Disease

2021 ◽  
Vol 10 (19) ◽  
pp. 4378
Author(s):  
Satoshi Higuchi ◽  
Takashi Kohno ◽  
Shun Kohsaka ◽  
Yasuyuki Shiraishi ◽  
Makoto Takei ◽  
...  
Keyword(s):  
Heart Failure ◽  
Chronic Obstructive Pulmonary Disease ◽  
Pulmonary Disease ◽  
Beta Blockers ◽  
Left Ventricular ◽  
Left Ventricular Ejection ◽  
Chronic Obstructive ◽  
Cardiac Mortality ◽  
Obstructive Pulmonary Disease ◽  
All Cause Mortality

The administration of beta-blockers is challenging and their efficacy is unclear in heart failure (HF) patients with chronic obstructive pulmonary disease (COPD). This study aimed to investigate the association of beta-blockers with mortality in such patients. This multicenter observational cohort study included hospitalized HF patients with a left ventricular ejection fraction <50% and evaluated them retrospectively. COPD was diagnosed based on medical records and/or the clinical judgment of each investigator. The study endpoints were two-year all-cause, cardiac, and non-cardiac mortality. This study included 83 patients with COPD and 1760 patients without. Two-year all-cause, cardiac, and non-cardiac mortality were observed in 315 (17%), 149 (8%), and 166 (9%) patients, respectively. Beta-blockers were associated with lower all-cause mortality regardless of COPD (COPD: hazard ratio [HR] 0.39, 95% CI 0.16–0.98, p = 0.044; non-COPD: HR 0.62, 95% CI 0.46–0.83, p = 0.001). This association in HF patients with COPD persisted after multivariate analysis and inverse probability weighting and was due to lower non-cardiac mortality (HR 0.40, 95% CI 0.14–1.18. p = 0.098), not cardiac mortality (HR 0.37, 95% CI 0.07–2.01, p = 0.248). Beta-blockers were associated with lower all-cause mortality in HF patients with COPD due to lower non-cardiac mortality. This may reflect selection biases in beta-blocker prescription.

scholarly journals Congestive heart failure in a young cat with excessive moderator bands (false tendons) in the left ventricle

2021 ◽  
Vol 51 (10) ◽  
Author(s):  
Fernanda Genro Cony ◽  
Matheus Viezzer Bianchi ◽  
Fernando Froner Argenta ◽  
Carolina Rodrigues Oliveira ◽  
Carine Stefanello ◽  
...  
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Left Ventricle ◽  
Interventricular Septum ◽  
Single Layer ◽  
Left Ventricular ◽  
Pulmonary Atelectasis ◽  
History Of ◽  
Adult Cats ◽  
Well Differentiated

ABSTRACT: Left ventricular false tendons are fibrous or fibromuscular bands that transverse the ventricular cavity and have no attachment to the mitral valve in many species. In cats it is considered a congenital defect that is rarely related to clinical disease and death in adult cats. A 45 days-old mixed breed cat had a history of inappetence since birth. At the physical exam the patient was lethargic and presented restrictive dyspnoea. At necropsy, there were marked ascites, hydrothorax, hepatomegaly with enhanced lobular pattern (nutmeg liver), and the lungs were markedly diminished (compressive pulmonary atelectasis). The heart was enlarged due to marked dilation of the cardiac chambers. Moreover, multiple slightly whitish and irregular cord-like structures were connecting the posterior papillary muscle to the interventricular septum (excessive moderator bands /left ventricular false tendons) at the left ventricle. Microscopically, these structures were characterized by a marked proliferation of fibrous connective tissue intermixed with Purkinje cells and well-differentiated cardiomyocytes lined by a single layer of endocardium. This study described a case of excessive moderator bands (left ventricular false tendons) in a young cat associated with congestive heart failure and death.

scholarly journals Diagnostic and Prognostic Markers of Chronic Heart Failure in Patients with Occupational Chronic Obstructive Pulmonary Disease

Kardiologiia ◽  
2020 ◽  
Vol 60 (7) ◽  
pp. 44-52
Author(s):  
L. A. Shpagina ◽  
N. V. Kamneva ◽  
L. M. Kudelya ◽  
O. S. Kotova ◽  
I. S. Shpagin ◽  
...  
Keyword(s):  
Heart Failure ◽  
Chronic Obstructive Pulmonary Disease ◽  
Pulmonary Disease ◽  
Left Ventricular ◽  
Left Ventricular Ejection ◽  
Chronic Obstructive ◽  
Serum Concentrations ◽  
Obstructive Pulmonary Disease ◽  
Work Exposure ◽  
S 100

Aim      Heart failure (HF) and chronic obstructive pulmonary disease (COPD) are a common comorbidity. Professional chronic obstructive pulmonary disease (PCOPD) is a specific phenotype, which suggests peculiarities in the development of HF. Difficulties of HF diagnosis in such patients determine the relevance of searching for additional markers. The aim of the study was identifying HF markers in patients with PCOPD.Material and methods  This single-site, cohort, prospective, observational study included 345 patients. The main group consisted of PCOPD patients; the comparison group consisted of patients with COPD induced by tobacco smoking; and the control group included conventionally healthy individuals. The groups were matched by the index of coincidence; pairs were matched at 1:1 by the “nearest neighbor index”; covariates for matching included COPD duration, sex, and age. Each group included 115 patients. The major professional adverse factors were silica-containing dust and organic solvents. COPD was diagnosed according to GOLD criteria; HF was diagnosed in accordance with Russian clinical guidelines. The markers were determined by multifactorial logistic regression. Likelihood of events with allowance for the time to the event was analyzed by the Kaplan-Meier method.Results HF in PCOPD patients was characterized by biventricular damage, preserved left ventricular ejection fraction, and frequent hospitalizations for decompensation (17.5 % vs. 9.5 % for COPD in smokers). HF markers in patients with PCOPD included the length of work of more than 20 years, pulmonary artery systolic pressure (PASP) higher than 35 mm Hg according to data of Doppler echocardiography, diffusing capacity of lungs for carbon monoxide (DLCO) less than 50 %, increased serum concentrations of CC-chemokine ligand 18 (CCL18), S-100‑beta protein, and N-terminal pro-brain natriuretic peptide (NT-pro-BNP). Diagnostic sensitivity of the multifactorial model was 84 % and specificity was 81 %. Two models were proposed for purposes of screening, which included the following parameters: length of work, exposure to aromatic hydrocarbons, decreased distance in 6-min walk test by more than 60 m per year and length of work, exposure to inorganic dust, and decreased forced expiratory volume during the first second by more than 55 ml per year.Conclusion      The markers for development of HF in PCOPD patients are length of work >20 years, PASP >35 mm Hg, DLCO <50 %, and increased serum concentrations of CCL18, S-100‑beta protein, and NT-pro-BNP.

scholarly journals Surgical Repair of a Giant Aorto–Left Ventricular Fistula

2019 ◽  
Vol 46 (2) ◽  
pp. 133-135
Author(s):  
Charles Mve Mvondo ◽  
Marta Pugliese ◽  
Ellen M. Dailor
Keyword(s):  
Heart Failure ◽  
Surgical Treatment ◽  
Left Ventricle ◽  
Symptom Severity ◽  
Aortic Wall ◽  
Surgical Repair ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Unusual Condition ◽  
The Right

Aortoventricular fistula, a rare congenital or acquired defect of the aortic wall, is characterized by an abnormal connection between the aorta and one of the ventricles. Symptom severity correlates with the diameter of the fistula and with the acute or chronic timing of presentation. The diagnosis is usually made by using echocardiography, and surgical treatment is necessary to avoid progression to heart failure. We describe the case of a 27-year-old woman who underwent successful surgical repair of an aortoventricular fistula that originated from the right coronary sinus and extended into the left ventricle through the interventricular septum. In addition to the patient's case, we briefly discuss this unusual condition

scholarly journals A Case Report of Delayed Diagnosed Chronic Aortocaval Fistula: A Rare Complication of Penetrating Trauma to the Abdomen

2015 ◽  
Vol 5 ◽  
pp. 62 ◽  
Author(s):  
Osman Beton ◽  
Hatice Kaplanoğlu ◽  
Öcal Berkan ◽  
Mehmet Birhan Yılmaz
Keyword(s):  
Heart Failure ◽  
Pulmonary Hypertension ◽  
Rare Complication ◽  
Penetrating Trauma ◽  
Severe Pneumonia ◽  
Right Heart Failure ◽  
Chronic Obstructive ◽  
Aortocaval Fistula ◽  
Right Heart ◽  
Obstructive Pulmonary Disease

Chronic aortocaval fistula (ACP) is a rare complication of penetrating trauma to the abdomen. We report a case of traumatic ACP presenting with pulmonary hypertension and right heart failure symptoms 15 years after the initial penetrating injury. Although symptoms of pulmonary hypertension started 5 years ago, it was wrongly diagnosed and treated as chronic obstructive pulmonary disease. The presence of a continuous abdominal bruit and history of penetrating abdominal trauma gave rise to suspicion of a fistula, which was confirmed by computed tomography and angiography. Percutaneous closure of ACP was planned, but the patient died of severe pneumonia. The clinical presentation of chronic ACP can vary from being asymptomatic to symptoms related to pulmonary hypertension, right heart failure, and pulmonary embolism; thus, definitive diagnosis can be challenging.

scholarly journals THE ASSOCIATION BETWEEN INTRACARDIAC HEMODYNAMICS AND LUNG FUNCTION IN PATIENTS WITH CHRONIC OBSTRUCTIVE PULMONARY DISEASE

2019 ◽  
Vol 9 (5) ◽  
pp. 373-381
Author(s):  
N. V. Vysotskaya ◽  
V. V. Lee ◽  
N. Yu. Timofeeva ◽  
V. S. Zadionchenko ◽  
T. V. Adasheva
Keyword(s):  
Cardiovascular Disease ◽  
Chronic Obstructive Pulmonary Disease ◽  
Left Ventricle ◽  
Airway Obstruction ◽  
Pulmonary Disease ◽  
Left Ventricular ◽  
Diastolic Filling ◽  
Chronic Obstructive ◽  
Inspiratory Capacity ◽  
Obstructive Pulmonary Disease

Study objective. To assess the association between intracardiac hemodynamics and airway obstruction with pulmonary hyperinflation in patients with chronic obstructive pulmonary disease.Materials and methods. Ninety-six patients with chronic obstructive pulmonary disease, aged 40 to 75 years, without concomitant cardiovascular disease, were examined and divided into 4 groups according to the severity of the disease. The patients underwent general clinical examination, spirometry, 24-hour pulse oximetry and echocardiography with assessment of linear and volumetric parameters, as well as diastolic function of left and right ventricles.Results. Linear and volumetric parameters of the left ventricle, LV myocardial mass and geometry in the examined patients with chronic obstructive pulmonary disease matched threshold values. The progression of the severity of chronic obstructive pulmonary disease was accompanied by decrease of the end-diastolic size of the left ventricle, ratio of peak early to late diastolic filling velocity for the left ventricle (E/A) without significant changes in the left ventricle isovolumetric relaxation time (IVRT). Moderate correlations of the inspiratory capacity with the end-diastolic size of the left ventricle (r=0.612; p=0.001) and the left ventricle E/A (r=0.464; p=0.001); forced expiratory volume in 1 second (FEV1) with the left ventricle E/A (r=0.600; p=0.011) were established. As a result of the logistic regression performed, the predictor value of the inspiratory capacity was confirmed (Wald χ2 — 5.795; р=0.024). Impairment of left ventricular diastolic function of grade I was revealed in 12 (31.6 %) patients in group 2, in 7 (24.1 %) patients in group 3, and in 9 (56.2 %) patients in group 4.Conclusion. Airway obstruction severity and pulmonary hyperinflation progression in patients with chronic obstructive pulmonary disease and without concomitant cardiovascular disease is associated with a decrease of left ventricular size and diastolic filling, contributes to the development of the left ventricular diastolic dysfunction, predominantly due to the decrease in filling velocity parameters.

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