Time Course Analysis of the Reverse-Stroop Effect in Japanese Kanji

1998 ◽  
Vol 87 (1) ◽  
pp. 163-174 ◽  
Author(s):  
Kayu Moriguchi ◽  
Yasuo Morikawa

A reverse of the Stroop effect was obtained with Japanese kanji (logographic script) but not with Japanese kana (syllabic scripts) by Morikawa in 1981. In the present study, the normal effect on reaction times by word and color was altered by presenting the words before or after the color. The reverse Stroop effect was observed with kanji but not with kana even when the color was presented prior to the word. It was shown that the difference between kanji and kana in the reverse-Stroop effect could not be explained by the relative speed of processing of word and color and that the reading process of kanji is different from that of kana.

Author(s):  
P. Maupin-Szamier ◽  
T. D. Pollard

We have studied the destruction of rabbit muscle actin filaments by osmium tetroxide (OSO4) to develop methods which will preserve the structure of actin filaments during preparation for transmission electron microscopy.Negatively stained F-actin, which appears as smooth, gently curved filaments in control samples (Fig. 1a), acquire an angular, distorted profile and break into progressively shorter pieces after exposure to OSO4 (Fig. 1b,c). We followed the time course of the reaction with viscometry since it is a simple, quantitative method to assess filament integrity. The difference in rates of decay in viscosity of polymerized actin solutions after the addition of four concentrations of OSO4 is illustrated in Fig. 2. Viscometry indicated that the rate of actin filament destruction is also dependent upon temperature, buffer type, buffer concentration, and pH, and requires the continued presence of OSO4. The conditions most favorable to filament preservation are fixation in a low concentration of OSO4 for a short time at 0°C in 100mM sodium phosphate buffer, pH 6.0.


Author(s):  
Li Hsieh

Bilingual speakers rely on attentional and executive control to continuously inhibit or activate linguistic representations of competing languages, which leads to an increased efficiency known as “bilingual advantage”. Both monolingual and bilingual speakers were asked to perform multiple tasks of talking on a cell phone while simultaneously attending to simulated driving events. This study examined the effect of bilingualism on participants' performance during a dual-task experiment based on 20 monolingual and 13 bilingual healthy adults. The within-subject and between-subject comparisons were conducted on reaction times of a visual event detection task for (a) only driving and (b) driving while simultaneously engaged in a phone conversation. Results of this study showed that bilingual speakers performed significantly faster than monolingual speakers during the multitasking condition, but not during the driving only condition. Further, bilingual speakers consistently showed a bilingual advantage in reaction times during the multitasking condition, despite varying degrees on a bilingual dominance scale. Overall, experiences in more than one language yield bilingual advantage in better performance than monolingual speakers during a multitasking condition, but not during a single task condition. Regardless of the difference in bilingual proficiency level, such language experience reveals a positive impact on bilingual speakers for multitasking.


1990 ◽  
Vol 68 (5) ◽  
pp. 2100-2106 ◽  
Author(s):  
T. Chonan ◽  
M. B. Mulholland ◽  
J. Leitner ◽  
M. D. Altose ◽  
N. S. Cherniack

To determine whether the intensity of dyspnea at a given level of respiratory motor output depends on the nature of the stimulus to ventilation, we compared the sensation of difficulty in breathing during progressive hypercapnia (HC) induced by rebreathing, during incremental exercise (E) on a cycle ergometer, and during isocapnic voluntary hyperventilation (IVH) in 16 normal subjects. The sensation of difficulty in breathing was rated at 30-s intervals by use of a visual analog scale. There were no differences in the level of ventilation or the base-line intensity of dyspnea before any of the interventions. The intensity of dyspnea grew linearly with increases in ventilation during HC [r = 0.98 +/- 0.02 (SD)], E (0.95 +/- 0.03), and IVH (0.95 +/- 0.06). The change in intensity of dyspnea produced by a given change in ventilation was significantly greater during HC [0.27 +/- 0.04 (SE)] than during E (0.12 +/- 0.02, P less than 0.01) and during HC (0.30 +/- 0.04) than during IVH (0.16 +/- 0.03, P less than 0.01). The difference in intensity of dyspnea between HC and E or HC and IVH increased as the difference in end-tidal PCO2 widened, even though the time course of the increase in ventilation was similar. No significant differences were measured in the intensity of dyspnea that occurred with changes in ventilation between E and IVH. These results indicate that under nearisocapnic conditions the sensation of dyspnea produced by a given level of ventilation seems not to depend on the method used to produce that level of ventilation.(ABSTRACT TRUNCATED AT 250 WORDS)


2016 ◽  
Vol 81 (6) ◽  
pp. 697-705 ◽  
Author(s):  
Xuepeng Yang ◽  
Yan Ji ◽  
Fangfang Wang ◽  
Jia Xu ◽  
Xiangzhen Liu ◽  
...  

Vibrio fischeri bioluminescence inhibition has been widely used to test acute toxicities of metals and organics contaminants. However, the differences of metals and organics acute toxicities to V. fischeri have not been compared. Here, four heavy metals (Zn2+, Cu2+, Cd2+, Cr6+) and five organics (phenol, benzoic acid, p-hydroxy benzoic acid, nitro-benzene and benzene) acute toxicities to V. fischeri were investigated. Heavy metals toxicities to V. fischeri were increased along with the reaction time, while the organics toxicities kept the same level in different reaction times. In order to explain the difference, the relative cell death rate of V. fischeri was detected. In metals toxicities tests, the bioluminescence inhibition rate of V. fischeri was found to be significantly higher than the relative cell death rate (P<0.05), while for the organics toxicities tests, the cell death rate was similar to the bioluminescence inhibition rate. These results indicated that organics acute toxicities to V. fischeri could reflect the death of cell, but metals acute toxicities to V. fischeri may not lead to the death of cell, just represent the bioluminescence inhibition.


2001 ◽  
Vol 170 (3) ◽  
pp. 555-564 ◽  
Author(s):  
AR Green ◽  
EL Parrott ◽  
M Butterworth ◽  
PS Jones ◽  
P Greaves ◽  
...  

This study compares the actions of oestradiol, tamoxifen, toremifene and raloxifene on enzyme and gene expression in uterine tissues of ovariectomised rats over 72 h. The time-course for the induction of ornithine decarboxylase by the compounds showed a rapid biphasic response, while for creatine kinase brain type (BB) there was a continued increase over 72 h. The efficacy of induction showed that, with both markers, oestradiol gave the highest induction level, followed by tamoxifen or toremifene and then raloxifene. RT-PCR demonstrated that all compounds decreased oestrogen receptor (ER) alpha, ERbeta and ERbeta2 gene expression, 8-24 h after the first dose, suggesting that down-regulation of ER is not the primary cause of the difference in efficacy between these compounds. Using cDNA arrays, expression of 512 genes was examined in the uteri of oestradiol- or tamoxifen-treated rats. Both compounds resulted in the up-regulation of heat-shock protein 27, telomerase-associated protein 1 and secretin. However, most surprising was the marked down-regulation of Wilms' tumour and retinoblastoma genes. We speculate that this may result in a loss of regulation of the transition from the G1 to the S phase in the cell cycle and may make cells more vulnerable to the carcinogenic effects of tamoxifen in this tissue.


2021 ◽  
Vol 11 ◽  
Author(s):  
Wang Xiang

To investigate whether implicit detection occurs uniformly during change blindness with single or combination feature stimuli, and whether implicit detection is affected by exposure duration and delay, two one-shot change detection experiments are designed. The implicit detection effect is measured by comparing the reaction times (RTs) of baseline trials, in which stimulus exhibits no change and participants report “same,” and change blindness trials, in which the stimulus exhibits a change but participants report “same.” If the RTs of blindness trials are longer than those of baseline trials, implicit detection has occurred. The strength of the implicit detection effect was measured by the difference in RTs between the baseline and change blindness trials, where the larger the difference, the stronger the implicit detection effect. In both Experiments 1 and 2, the results showed that the RTs of change blindness trials were significantly longer than those of baseline trials. Whether under set size 4, 6, or 8, the RTs of the change blindness trials were significantly longer than those in the baseline trials. In Experiment 1, the difference between the baseline trials’ RTs and change blindness trials’ RTs of the single features was significantly larger than that of the combination features. However, in Experiment 2, the difference between the baseline trials’ RTs and the change blindness trials’ RTs of single features was significantly smaller than that of the combination features. In Experiment 1a, when the exposure duration was shorter, the difference between the baseline and change blindness trials’ RTs was smaller. In Experiment 2, when the delay was longer, the difference between the two trials’ RTs was larger. These results suggest that regardless of whether the change occurs in a single or a combination of features and whether there is a long exposure duration or delay, implicit detection occurs uniformly during the change blindness period. Moreover, longer exposure durations and delays strengthen the implicit detection effect. Set sizes had no significant impact on implicit detection.


1999 ◽  
Vol 112 (4) ◽  
pp. 447-454 ◽  
Author(s):  
D. Zicha ◽  
E. Genot ◽  
G.A. Dunn ◽  
I.M. Kramer

We have previously shown that addition of type 1 transforming growth factor-beta (TGFbeta1) to an exponentially growing population of mink lung CCl64 cells increases their average intermitotic time from 14.4 to 20.3 hours, predominantly by extending G1 from 7.5 to 13.5 hours. Here we have used the DRIMAPS system (digitally recorded interference microscopy with automatic phase-shifting) for obtaining data on cellular mass distribution, cell motility and morphology. We found no significant change in the cells' rate of mass increase following TGFbeta1 treatment, which implies that the treated cells attained a higher mass during their extended cell cycle and this was confirmed by direct measurement of cell size. However, the cells showed a dramatic motile response to treatment: TGFbeta1-treated cells had a significantly higher time-averaged speed of 36.2 microm hour-1 compared to 14.5 microm hour-1 for the control cells. The time course of the response was gradual, reaching a maximum mean speed of 52.6 microm hour-1 after 15 hours exposure. We found that the gradual onset of the response was probably not due to a slow accumulation of a secondary factor but because cells were dividing throughout the experiment and most of the response to TGFbeta1 occurred only after the first cell division in its presence. Thus, taking only those cells that had not yet divided, the time-averaged speed of treated cells (26.1 micrometer hour-1) was only moderately higher than that of untreated cells (14.9 micrometer hour-1) whereas, for those cells that had divided, the difference in speed between treated cells (45.1 micrometer hour-1) and untreated cells (14.1 microm hour-1) was much greater. Increased speed was a consequence of enhanced protrusion and retraction of the cell margin coupled with an increase in cell polarity. TGFbeta1 also increased the mean spreading of the cells, measured as area-to-mass ratio, from 3.2 to 4.4 micrometer2 pg-1, and the intracellular mass distribution became more asymmetric. The observations indicate that a G2 signal may be necessary to reach maximal motility in the presence of TGFbeta1.


1981 ◽  
Author(s):  
Lj Popović

Changes in reaction time of clot assays are usually expressed only in time units, which fails to indicate the extent of the increase or decrease of the reaction time of the tested specimens against that of the basic sample. Reaction time increases of, e.g. , 6 seconds in tested samples, compared to basic sample reaction times of 12 and 24 seconds respectively, signify an increase twice as large in the first as in the second instance.Changes in reaction time of clot assays can be expressed as the increment or decrement of the reaction time per time unit. This amount of increase or decrease (positive or negative alteration of reaction time, T a ) can be expressed as the quotient of the difference between the reaction times of the tested (T x ) and basic (To) samples and of the basic sample, e.g. in seconds per second, T a =T x -To/To. A test sample reaction time 6 seconds longer than basic sample reaction times of 12 and 2k seconds would mean an increase of 0.5 and 0.25 seconds per second, respectively.Reaction time changes of tested samples against that of the standard sample (T std ) can be calculated in a similar way, T a =T x -T std /T std .It can be assumed that this parameter reflects the intensity of the increase or decrease of reaction time per time unit. The quotient of the tested and basic samples can be considered as the coefficient of the increase or decrease of the total reaction time (CT=T x /To).


1985 ◽  
Vol 249 (1) ◽  
pp. C124-C128 ◽  
Author(s):  
P. K. Lauf ◽  
C. M. Perkins ◽  
N. C. Adragna

The effects of incubation in anisosmotic media and of metabolic depletion on ouabain-resistant (OR) Cl--dependent K+ influxes stimulated by N-ethylmaleimide (NEM) were studied in human red blood cells using Rb+ as K+ analogue. The NEM-stimulated but not the basal Rb+-Cl- influx measured in phosphate-buffered anisosmotic media was found to be cell volume dependent. When cellular ATP, [ATP]c, was lowered to less than 0.10 of its initial level by exposure to nonmetabolizable 2-deoxy-D-glucose, the NEM-stimulated but not the basal Cl--dependent Rb+ influxes were abolished. Metabolically depleted red blood cells subsequently repleted by incubation in glucose plus inosine regained the NEM-inducible Rb+ (K+) transport activity. The difference in the time course of ATP breakdown and Rb+ influx inhibition suggests that energization of the NEM-stimulated Rb+ flux by metabolism may involve factors additional to ATP.


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