RISK FACTORS FOR SCHOOL FAILURE OF STUDENTS IN SMALL-TOWN SCHOOLS

2021 ◽  
pp. 191-199
Author(s):  
Oksana M. Gornaeva
Acta Tropica ◽  
2017 ◽  
Vol 174 ◽  
pp. 158-164 ◽  
Author(s):  
Márcia B. Silva ◽  
Ana L.M. Amor ◽  
Leonardo N. Santos ◽  
Alana A. Galvão ◽  
Aida V. Oviedo Vera ◽  
...  

Author(s):  
Emily S. Fisher ◽  
Kelly S. Kennedy ◽  
Brianna Meshke McLay

This chapter reviews strategies for working with students who are at risk for school failure or dropout. Because each of the populations discussed in the earlier chapters may be at risk for dropout or failure, this chapter builds upon previous strategies by providing a framework for targeting motivation and academic enabling skills (e.g., note taking, study skills). It describes risk factors (both membership in certain groups and personal risk factors) that may contribute to school failure or dropout. It also addresses the need to distinguish between skills/acquisitions deficits and performance deficits in identifying the reasons a student is struggling academically, is chronically truant, or is at risk of dropping out. Counseling strategies to address motivation and academic enabling skills include motivational interviewing, solution-focused brief therapy, cognitive-behavioral therapy, and group counseling.


1980 ◽  
Vol 2 (1) ◽  
pp. 5-11
Author(s):  
Frank Oberklaid ◽  
Melvin D. Levine

School failure is pervasive. Its impact extends beyond the classroom and can contribute to emotional turmoil, social difficulties, delinquent behavior and lifelong maladjustment. School failure does not qualify as a disease, a syndrome, or a unique pathogenetic process. It is rather the product of constitutional predispositions that have interacted with environmental factors and life events in an ongoing process. Intrinsic handicaps may be subtle. Their outward manifestations are likely to vary from child to child and evolve with age, shaped by neurologic maturation, experience, and changing external expectations. Children with school problems form a heterogeneous group with diverse etiologies, symptom complexes, diagnostic findings and service needs. Nine common precursors of school failure are suggested in this review. Although presented individually, these precursors are not necessarily discrete, nor are they mutually exclusive; they may themselves stem from multiple factors. They are conceptualized as "risk factors" that may predispose to or foreshadow later failure. These, in turn, interact with a set of variables we have characterized as sources of "developmental buoyancy." The latter may neutralize or counteract early susceptibility to academic problems. Individual children may have one or multiple identifiable precursors of variable severity and differ in the extent to which they can minimize or overcome these potential predispositions to school failure.


2019 ◽  
Vol 133 (22) ◽  
pp. 2283-2299
Author(s):  
Apabrita Ayan Das ◽  
Devasmita Chakravarty ◽  
Debmalya Bhunia ◽  
Surajit Ghosh ◽  
Prakash C. Mandal ◽  
...  

Abstract The role of inflammation in all phases of atherosclerotic process is well established and soluble TREM-like transcript 1 (sTLT1) is reported to be associated with chronic inflammation. Yet, no information is available about the involvement of sTLT1 in atherosclerotic cardiovascular disease. Present study was undertaken to determine the pathophysiological significance of sTLT1 in atherosclerosis by employing an observational study on human subjects (n=117) followed by experiments in human macrophages and atherosclerotic apolipoprotein E (apoE)−/− mice. Plasma level of sTLT1 was found to be significantly (P<0.05) higher in clinical (2342 ± 184 pg/ml) and subclinical cases (1773 ± 118 pg/ml) than healthy controls (461 ± 57 pg/ml). Moreover, statistical analyses further indicated that sTLT1 was not only associated with common risk factors for Coronary Artery Disease (CAD) in both clinical and subclinical groups but also strongly correlated with disease severity. Ex vivo studies on macrophages showed that sTLT1 interacts with Fcɣ receptor I (FcɣRI) to activate spleen tyrosine kinase (SYK)-mediated downstream MAP kinase signalling cascade to activate nuclear factor-κ B (NF-kB). Activation of NF-kB induces secretion of tumour necrosis factor-α (TNF-α) from macrophage cells that plays pivotal role in governing the persistence of chronic inflammation. Atherosclerotic apoE−/− mice also showed high levels of sTLT1 and TNF-α in nearly occluded aortic stage indicating the contribution of sTLT1 in inflammation. Our results clearly demonstrate that sTLT1 is clinically related to the risk factors of CAD. We also showed that binding of sTLT1 with macrophage membrane receptor, FcɣR1 initiates inflammatory signals in macrophages suggesting its critical role in thrombus development and atherosclerosis.


2011 ◽  
Vol 21 (2) ◽  
pp. 59-62
Author(s):  
Joseph Donaher ◽  
Christina Deery ◽  
Sarah Vogel

Healthcare professionals require a thorough understanding of stuttering since they frequently play an important role in the identification and differential diagnosis of stuttering for preschool children. This paper introduces The Preschool Stuttering Screen for Healthcare Professionals (PSSHP) which highlights risk factors identified in the literature as being associated with persistent stuttering. By integrating the results of the checklist with a child’s developmental profile, healthcare professionals can make better-informed, evidence-based decisions for their patients.


2010 ◽  
Vol 20 (3) ◽  
pp. 76-83 ◽  
Author(s):  
Joseph Donaher ◽  
Tom Gurrister ◽  
Irving Wollman ◽  
Tim Mackesey ◽  
Michelle L. Burnett

Parents of children who stutter and adults who stutter frequently ask speech-language pathologists to predict whether or not therapy will work. Even though research has explored risk-factors related to persistent stuttering, there remains no way to determine how an individual will react to a specific therapy program. This paper presents various clinicians’answers to the question, “What do you tell parents or adults who stutter when they ask about cure rates, outcomes, and therapy efficacy?”


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