scholarly journals Effects of Triazole Fungicides on Soil Microbiota and on the Activities of Enzymes Found in Soil: A Review

Agriculture ◽  
2021 ◽  
Vol 11 (9) ◽  
pp. 893
Author(s):  
Diana Larisa Roman ◽  
Denisa Ioana Voiculescu ◽  
Madalina Filip ◽  
Vasile Ostafe ◽  
Adriana Isvoran

Triazole fungicides can manifest toxicity to a wide range of non-target organisms. Within this study we present a systematic review of the effects produced on the soil microbiota and activity of soil enzymes by the following triazole fungicides: cyproconazole, difenoconazole, epoxiconazole, flutriafol, hexaconazole, metconazole, myclobutanil, paclobutrazole, propiconazole, tebuconazole, tetraconazole, triadimenol, triadimefon, and triticonazole. Known effects of the triazole fungicides on the soil activity are dose dependent. High doses of triazole fungicides strongly affects the structure of the microbial communities in soil and usually decrease the soil microbial population and the activities of enzymes found in soil.

1981 ◽  
Vol 61 (2) ◽  
pp. 445-450 ◽  
Author(s):  
S. P. MATHUR ◽  
C. M. PRESTON

As a part of attempts at examining the suitability of using moderate amounts of Cu for mitigating the undesirably fast decomposition and subsidence of some organic soils, this investigation revealed that both total and DTPA-TEA-extractable Cu contents of 14 samples of cultivated organic soils correlated negatively with the total microbial population counted on soil extract-acitidione-gelatin agar plates and positively with the percentage of that population capable of extracellular proteolysis. On the other hand, the concentrations of both total and extractable Cu did not correlate with either the total number of proteolytic organisms counted, or the capacities of the soils for ammonification and nitrification. The results are, therefore, in accord with the belief that Cu curtails the decomposition of organic soils primarily through inactivation of accumulated soil enzymes, but not through direct toxic effects on the general soil microbial population.


2004 ◽  
Vol 17 (2) ◽  
pp. 221-229 ◽  
Author(s):  
Merav Kedmi ◽  
Arthur L. Beaudet ◽  
Avi Orr-Urtreger

Nicotine, the main addictive component of tobacco, evokes a wide range of dose-dependent behaviors in rodents, and when administrated in high doses, it can induce clonic-tonic seizures. Nicotine acts through the nicotinic acetylcholine receptors (nAChRs). Mutations in the human α4- and the β2-nAChR subunit genes cause autosomal dominant nocturnal frontal lobe epilepsy. Using transgenic mice with mutations in nAChR subunits, it was demonstrated previously that the α4-, α5-, and α7-subunits are involved in nicotine-induced seizures. To examine the possibility that the β4-subunit is also involved in this phenotype, we tested mice with homozygous β4-subunit deficiency. The β4 null mice were remarkably resistant to nicotine-induced seizures compared with wild-type and α5 null mice. We also generated mice with double deficiency of both α5- and β4-nAChR subunits and demonstrated that they were more resistant to nicotine’s convulsant effect than either the α5 or the β4 single mutant mice. In addition, the single α5 mutants and the double α5β4-deficient mice exhibited a significantly shorter latency time to seizure than that of the wild-type mice. Our results thus show that β4-containing nAChRs have a crucial role in the pathogenesis of nicotine-induced seizures. Furthermore, by comparing multiple mutant mice with single and double subunit deficiency, we suggest that nicotinic receptors containing either α5- or β4-subunits are involved in nicotine-induced seizures and that receptors containing both subunits are likely to contribute to this phenomena as well. However, the α5-subunit, but not the β4-subunit, regulates the rate of response to high doses of nicotine.


Author(s):  
Wahyu Purbalisa ◽  
Ina Zulaehah ◽  
Dolty Melyga W. Paputri ◽  
Sri Wahyuni

Carbon and microbes in the soil fluctuated from time to time due to various things. This study aims to determine the dynamics of carbon and microbes in the soil in the treatment of biochar-compost. In addition to the use of biochar-compost, this research also uses nano biochar and enrichment with microbial consortia. The study was conducted at the screen house using a complete randomized design with three replications with following treatments: control / without organic fertilizer (P0), compost (P1), biochar-compost 1: 4 (P2), nano-biochar-compost 1: 4 (P3 ), biochar-compost + microbial consortia (P4), compost + microbial consortia (P5) and biochar-compost + microbial consortia (P6) with a dose of 2.5 tons/ha respectively. Biochar comes from corncobs. Compost biochar plus application was made before planting.  Parameters observed were soil carbon (C-organic), soil acidity (pH) at 7 DAA, 37 DAA and after harvest, and the total soil microbial population at 2 DAA and after harvest. Soil carbon was measured using Walkey and Black method measured by spectrophotometer, soil pH using a soil: water ratio = 1: 5 and measured by a pH meter, the total microbial population using Total Plate Counting (TPC) method. The results showed carbon and soil microbial populations decreased over time, except for microbial communities in a single compost treatment.


1990 ◽  
Vol 329 (1255) ◽  
pp. 369-373 ◽  

We tried to develop deterministic models for kinetics of 2,4-D breakdown in the soil based on the following considerations: (i) at low concentrations degradation results from maintenance consumption by a large fraction of the soil microbial population; (ii) at high concentration in addition to the maintenance consumption there is a growth-associated carbon incorporation by a small specific microbial population. Values for the biokinetic parameters are consistent with those commonly found in the literature. Comparison between observed and simulated curves suggests that a non-negligible part of the pesticidal carbon exists as microbial by-products.


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