scholarly journals Noise Pollution and Its Correlations with Occupational Noise-Induced Hearing Loss in Cement Plants in Vietnam

2021 ◽  
Vol 18 (8) ◽  
pp. 4229
Author(s):  
Tinh Thai ◽  
Petr Kučera ◽  
Ales Bernatik
Keyword(s):  
Hearing Loss ◽  
Noise Level ◽  
Noise Exposure ◽  
Noise Pollution ◽  
Noise Induced Hearing Loss ◽  
Exposure Limit ◽  
Global Issue ◽  
Exposed Workers ◽  
Almost All ◽  
Cement Grinding

Noise-Induced Hearing Loss (NIHL) is a global issue that is caused by many factors. The purpose of this study was to survey noise level to identify NIHL and its relationship with other factors in cement plants in Vietnam. Noise level was measured at one cement plant and three cement grinding stations located in the South of Vietnam. The audiometric data of exposed workers were surveyed to determine NIHL. Finally, the relationship between NIHL and noise level in cement plants was determined. The results show that noise level in almost all processes exceeded the permissible exposure limit (PEL). In this study, 42 cases (10% of exposed workers) with occupational NIHL were found with mean age (SD) of 49 (9.0) years. All NIHL cases were found in the departments in which the noise level exceeded the PEL, which included quarry (n = 16), maintenance (n = 12), production (n = 10), co-waste processing (n = 3) and quality assurance (n = 1). There was a positive and significant correlation between the NIHL and the excessive noise exposure in the cement plants (r = 0.89, p = 0.04).

scholarly journals PROFIL GANGGUAN PENDENGARAN PADA PEMUSIK DI KOTA MANADO

2014 ◽  
Vol 1 (2) ◽  
Author(s):  
Riscky B. Lauw
Keyword(s):  
Hearing Loss ◽  
Noise Level ◽  
Noise Exposure ◽  
Noise Induced Hearing Loss ◽  
Noise Induce Hearing Loss ◽  
Cross Sectional ◽  
Musical Sound ◽  
Keywords Hearing Loss ◽  
Analytical Research ◽  
The City

Abstract: Hearing loss due to noise (Noise Induced Hearing Loss - NIHL) is hearing impairment, either part or all, loss, irreversible, occur in one or both ears, can be mild, moderate or severe, occurs because of exposure to the constant noise from the environment. The musician is one of the communities that are susceptible to noise induced hearing loss. Musicians had considerable exposure to noise. Most musicians have a hearing loss in varying degrees. The research objective is to determine the profile of hearing loss in the musicians in Manado, researching threshold musical sound that can cause hearing loss, and looking for a relationship with duration of music exposure to hearing loss. Research is explanatory research, analytical research methods and cross-sectional approach. Research conducted on hearing music group in the City of Manado. The study involved 30 people with the details of 15 respondents guitar players and 15 drummers. The result is: The degree of noise in the gym most of the 110-130 dB, experienced by 46.7% of respondents, mostly studied band of men which is about 90%. Hearing loss is the most followed by drummer and then gitarist, 46.7% and 36.6%, respectively; based upon long exposure to music,> 5 years 46.6%, based on length of exercise per day, more in the 3-4 hours it is 40%. Based on the result of the study, there is a relationship between noise exposure and hearing loss in musicians in the city of Manado. The noise level, exposure time per day, duration of exposure and vulnerability of respondents, closely related to hearing loss disorder the musicians in this study. Keywords: Hearing loss, the noise level, duration of exposure, musicians.Abstrak: Noise induce hearing loss (NIHL) atau gangguan pendengaran akibat kebisingan adalah gangguan pendengaran baik sebagian atau seluruh pendengaran, bersifat menetap, terjadi pada satu atau dua telinga, dapat bersifat ringan, sedang atau berat, terjadi karena paparan bising yang terus-menerus dari lingkungan. Pemusik adalah salah satu komunitas yang rentan terhadap gangguan pendengaran akibat bising. Pemusik mengalami paparan suara yang begitu besar. Kebanyakan pemusik mengalami gangguan pendengaran dalam berbagai derajat. Tujuan penelitian adalah untuk mengetahui profil gangguan pendengaran pemusik di kota manado, meneliti ambang batas suara musik yang dapat menyebabkan gangguan pendengaran serta mencari hubungan lama paparan musik dengan gangguan pendengaran. Penelitian bersifat Explanatory Research, dengan metode penelitian analitik dan pendekatan cross sectional. Penelitian gangguan pendengaran dilakukan pada kelompok musik di Kota Manado. Penelitian ini melibatkan 30 orang responden dengan rincian 15 pemain gitar dan 15 pemain drum. Hasil yang diperoleh adalah: Derajat kebisingan pada tempat latihan paling banyak antara 110-130 dB, dialami oleh 46,7 % responden, personil band yang diteliti kebanyakan laki-laki yaitu sekitar 90 %. Gangguan pendengaran terbanyak adalah pemain drum diikuti oleh gitaris 46,7 % and 36,6 %. Berdasarkan lama paparan musik, > 5 tahun 46,6 %; berdasarkan lama latihan per hari, lebih banyak pada 3-4 jam yaitu 40 %. Berdasarkan hasil penelitian kesimpulannya, terdapat hubungan antara paparan kebisingan dan gangguan pendengaran pada pemusik di Kota Manado. Tingkat kebisingan, waktu paparan perhari, lama terpapar serta kerentanan responden, berhubungan erat dengan pendengaran pada pemusik pada penelitian ini. Kata kunci: Gangguan pendengaran, tingkat kebisingan, lama paparan, pemusik.

scholarly journals Noise Exposure

2021 ◽  
Author(s):  
Alberto Behar
Keyword(s):  
Hearing Loss ◽  
Human Activity ◽  
Basic Concept ◽  
Noise Exposure ◽  
Noise Induced Hearing Loss ◽  
Loud Noise ◽  
Frequency Content ◽  
Noise Levels ◽  
Content Change ◽  
Almost All

Noise exposure is a basic concept used to assess the risk of noise induced hearing loss in the workplace. It is very important, since loud noise is omnipresent in almost all human activity, especially in industry, construction, mining and transportation. The question to answer is how to determine the risk of a person performing in an environment where the noise levels, duration and frequency content change with time. The answer is obtained by measuring his noise exposure. Although the measurement itself is not complex or difficult, a proper knowledge of what exactly is the noise exposure and how to deal with the measurement result in fundamental to avoid getting wrong conclusions.

scholarly journals Noise-Induced Hearing Loss and Hypertension: A Preliminary Analysis of Baseline Data of Occupational Noise-Exposed Workers

2021 ◽  
Author(s):  
Long Miao ◽  
Lihong Yin ◽  
Yuepu Pu
Keyword(s):  
Blood Pressure ◽  
Hearing Loss ◽  
Exposure Time ◽  
Noise Exposure ◽  
Normal Hearing ◽  
Baseline Data ◽  
Noise Induced Hearing Loss ◽  
Occupational Noise ◽  
Exposed Workers ◽  
The Mean

Abstract The present study aimed to determine the current levels of systolic blood pressure (SBP), diastolic blood pressure (DBP) and binaural high frequency (3, 4, and 6 kHz) threshold on average (BHFTA), to assess the prevalence of noise-induced hearing loss (NIHL) and hypertension and determine the factors influencing the risk of both, and to evaluate the association between NIHL and hypertension among occupational noise-exposed workers. Questionnaire and occupational health checkup were performed to collect the personal information and physical examination data. Finally, baseline data from 42,588 occupational noise-exposed workers were analyzed. The mean levels of SBP, DBP, and BHFTA were 126.85 ± 15.94 mm Hg, 79.94 ± 11.61 mm Hg and 23.09 ± 11.32 dB, respectively. Of the 42,588 subjects, the prevalence of NIHL and hypertension were 24.38% (n = 10,383) and 25.40% (n = 10,816). The results suggested that higher risk of NIHL and hypertension were more likely to be the groups of male gender, aged > 35 years, exposure time to noise > 5 years, and smoking. The SBP, DBP and BHFTA levels increased with age and noise exposure time (Ptrend < 0.001). Besides, similar trends were also observed in the prevalence of NIHL and hypertension. Furthermore, there was a significant association of NIHL with hypertension. We found that 32.25% (n = 3,348) workers with NIHL had hypertension. The mean levels of SBP and DBP in NIHL workers were significantly higher than those with normal hearing (P < 0.001). In addition, NIHL workers had a significantly higher risk of developing hypertension than normal hearing workers (adjusted OR = 1.08, 95%CI = 1.03–1.14). Our current findings suggest that the prevalence of NIHL and hypertension are high in our study workers and occupational noise exposure is an important factor. Therefore, it is urgent to reduce noise exposure in the workplaces and to strengthen industrial noise monitoring.

scholarly journals Effect of Using Hearing Protection and Earphone on Noise Induced Hearing Loss and Tinnitus in Workshop’s Workers

2018 ◽  
Vol 9 (1) ◽  
pp. 21
Author(s):  
Putri Berliana Syah
Keyword(s):  
Hearing Loss ◽  
Random Sampling ◽  
Noise Level ◽  
Noise Exposure ◽  
Health Effect ◽  
Simple Random Sampling ◽  
Hearing Protection ◽  
Noise Induced Hearing Loss ◽  
Cross Sectional ◽  
Workplace Noise

Noise exposure can leads to health effect such as Noise Induced Hearing Loss (NIHL) and tinnitus. Besides workplace, noise can be obtained from using earphones. The aim of this study was to analyze the effect of using hearing protection and earphone on NIHL and tinnitus in workers at PT DPS machine workshop. This was a cross sectional with observational analytic study design. This study was conducted in 37 workers at PT DPS machine workshop exposed to noise by using simple random sampling. Results showed that noise level in workplace was 88,57 dBA (8 hours TWA) which exceed TLV 85 dBA. Prevalence of NIHL and tinnitus in workplace was respectively 21.6% and 54%. NIHL affected by using hearing protection and earphone (MLR, p < 0.01; p < 0.05 respectively) and tinnitus affected by using earphone (MLR, p < 0.01) after controlled by working period. It can be concluded that prevalence of NIHL and tinitus are relatively high, NIHL affected by using hearing protection and earphone while tinnitus affected by using earphone. It is recommended for workers to use hearing protection in noise workplace and to reduce using earphone while PT DPS must provide hearing protection and do briefi ng to increase workers’ awareness about using hearing protection.

MicroRNAs in Noise-Induced Hearing Loss and their Regulation by Oxidative Stress and Inflammation

2020 ◽  
Vol 21 (12) ◽  
pp. 1216-1224
Author(s):  
Fatemeh Forouzanfar ◽  
Samira Asgharzade
Keyword(s):  
Oxidative Stress ◽  
Hearing Loss ◽  
Hair Cell ◽  
Noise Exposure ◽  
Cell Damage ◽  
Sensory Cells ◽  
Noise Induced Hearing Loss ◽  
Irreversible Damage ◽  
Open Access Journals

Noise exposure (NE) has been recognized as one of the causes of sensorineural hearing loss (SNHL), which can bring about irreversible damage to sensory hair cells in the cochlea, through the launch of oxidative stress pathways and inflammation. Accordingly, determining the molecular mechanism involved in regulating hair cell apoptosis via NE is essential to prevent hair cell damage. However, the role of microRNAs (miRNAs) in the degeneration of sensory cells of the cochlea during NE has not been so far uncovered. Thus, the main purpose of this study was to demonstrate the regulatory role of miRNAs in the oxidative stress pathway and inflammation induced by NE. In this respect, articles related to noise-induced hearing loss (NIHL), oxidative stress, inflammation, and miRNA from various databases of Directory of Open Access Journals (DOAJ), Google Scholar, PubMed; Library, Information Science & Technology Abstracts (LISTA), and Web of Science were searched and retrieved. The findings revealed that several studies had suggested that up-regulation of miR-1229-5p, miR-451a, 185-5p, 186 and down-regulation of miRNA-96/182/183 and miR-30b were involved in oxidative stress and inflammation which could be used as biomarkers for NIHL. There was also a close relationship between NIHL and miRNAs, but further research is required to prove a causal association between miRNA alterations and NE, and also to determine miRNAs as biomarkers indicating responses to NE.

Noise Exposure and Hearing Disorders

2017 ◽  
Author(s):  
David C. Byrne ◽  
Thais C. Morata
Keyword(s):  
Hearing Loss ◽  
Noise Exposure ◽  
Hearing Protection ◽  
Noise Induced Hearing Loss ◽  
Hearing Disorders ◽  
Industrial Noise ◽  
Significant Impairment ◽  
Protection Devices ◽  
The Impact ◽  
Occupational Hearing Loss

Exposure to industrial noise and the resulting effect of occupational hearing loss is a common problem in nearly all industries. This chapter describes industrial noise exposure, its assessment, and hearing disorders that result from overexposure to noise. Beginning with the properties of sound, noise-induced hearing loss and other effects of noise exposure are discussed. The impact of hearing disorders and the influence of other factors on hearing loss are described. Typically, noise-induced hearing loss develops slowly, and usually goes unnoticed until a significant impairment has occurred. Fortunately, occupational hearing loss is nearly always preventable. Therefore, this chapter gives particular attention to recommendations for measures to prevent occupational hearing loss such as engineering noise controls and hearing protection devices.

scholarly journals Contributions and limitations of using machine learning to predict noise-induced hearing loss

2021 ◽  
Author(s):  
Feifan Chen ◽  
Zuwei Cao ◽  
Emad M. Grais ◽  
Fei Zhao
Keyword(s):  
Machine Learning ◽  
Hearing Loss ◽  
Roc Curve ◽  
Prediction Error ◽  
Noise Exposure ◽  
Model Construction ◽  
Support Vector ◽  
Exposure Level ◽  
Noise Induced Hearing Loss ◽  
Current Review

Abstract Purpose Noise-induced hearing loss (NIHL) is a global issue that impacts people’s life and health. The current review aims to clarify the contributions and limitations of applying machine learning (ML) to predict NIHL by analyzing the performance of different ML techniques and the procedure of model construction. Methods The authors searched PubMed, EMBASE and Scopus on November 26, 2020. Results Eight studies were recruited in the current review following defined inclusion and exclusion criteria. Sample size in the selected studies ranged between 150 and 10,567. The most popular models were artificial neural networks (n = 4), random forests (n = 3) and support vector machines (n = 3). Features mostly correlated with NIHL and used in the models were: age (n = 6), duration of noise exposure (n = 5) and noise exposure level (n = 4). Five included studies used either split-sample validation (n = 3) or ten-fold cross-validation (n = 2). Assessment of accuracy ranged in value from 75.3% to 99% with a low prediction error/root-mean-square error in 3 studies. Only 2 studies measured discrimination risk using the receiver operating characteristic (ROC) curve and/or the area under ROC curve. Conclusion In spite of high accuracy and low prediction error of machine learning models, some improvement can be expected from larger sample sizes, multiple algorithm use, completed reports of model construction and the sufficient evaluation of calibration and discrimination risk.

scholarly journals Effect of Phlorofucofuroeckol A and Dieckol Extracted from Ecklonia cava on Noise-induced Hearing Loss in a Mouse Model

Marine Drugs ◽  
2021 ◽  
Vol 19 (8) ◽  
pp. 443
Author(s):  
Hyunjun Woo ◽  
Min-Kyung Kim ◽  
Sohyeon Park ◽  
Seung-Hee Han ◽  
Hyeon-Cheol Shin ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Hair Cell ◽  
Noise Exposure ◽  
Radical Scavenging ◽  
Threshold Shift ◽  
Ecklonia Cava ◽  
Control Group ◽  
Noise Induced Hearing Loss ◽  
Antioxidant Agents ◽  
Brainstem Response

One of the well-known causes of hearing loss is noise. Approximately 31.1% of Americans between the ages of 20 and 69 years (61.1 million people) have high-frequency hearing loss associated with noise exposure. In addition, recurrent noise exposure can accelerate age-related hearing loss. Phlorofucofuroeckol A (PFF-A) and dieckol, polyphenols extracted from the brown alga Ecklonia cava, are potent antioxidant agents. In this study, we investigated the effect of PFF-A and dieckol on the consequences of noise exposure in mice. In 1,1-diphenyl-2-picrylhydrazyl assay, dieckol and PFF-A both showed significant radical-scavenging activity. The mice were exposed to 115 dB SPL of noise one single time for 2 h. Auditory brainstem response(ABR) threshold shifts 4 h after 4 kHz noise exposure in mice that received dieckol were significantly lower than those in the saline with noise group. The high-PFF-A group showed a lower threshold shift at click and 16 kHz 1 day after noise exposure than the control group. The high-PFF-A group also showed higher hair cell survival than in the control at 3 days after exposure in the apical turn. These results suggest that noise-induced hair cell damage in cochlear and the ABR threshold shift can be alleviated by dieckol and PFF-A in the mouse. Derivatives of these compounds may be applied to individuals who are inevitably exposed to noise, contributing to the prevention of noise-induced hearing loss with a low probability of adverse effects.

Field Monitoring of Otoacoustic Emissions During Noise Exposure: Pilot Study in Controlled Environment

2017 ◽  
Vol 26 (3S) ◽  
pp. 352-368 ◽  
Author(s):  
Vincent Nadon ◽  
Annelies Bockstael ◽  
Dick Botteldooren ◽  
Jérémie Voix
Keyword(s):  
Hearing Loss ◽  
Pilot Study ◽  
Otoacoustic Emissions ◽  
Ambient Noise ◽  
Noise Exposure ◽  
High Temporal Resolution ◽  
Noise Induced Hearing Loss ◽  
Conservation Programs ◽  
Hearing Conservation ◽  
Hearing Status

Purpose In spite of all the efforts to implement workplace hearing conservation programs, noise-induced hearing loss remains the leading cause of disability for North American workers. Nonetheless, an individual's susceptibility to noise-induced hearing loss can be estimated by monitoring changes in hearing status in relation to the level of ambient noise exposure. The purpose of this study was to validate an approach that could improve workplace hearing conservation practices. The approach was developed using a portable and robust system designed for noisy environments and consisted of taking continuous measurements with high temporal resolution of the health status of the inner ear using otoacoustic emissions (OAEs). Method A pilot study was conducted in a laboratory, exposing human subjects to industrial noise recordings at realistic levels. In parallel, OAEs were measured periodically using the designed OAE system as well as with a commercially available OAE system, used as a reference. Results Variations in OAE levels were analyzed and discussed along with the limitations of the reference and designed systems. Conclusions This study demonstrates that the monitoring of an individual's OAEs could be useful in monitoring temporary changes in hearing status induced by exposure to ambient noise and could be considered as a new tool for effective hearing conservation programs in the workplace.

scholarly journals Primed to die: an investigation of the genetic mechanisms underlying noise-induced hearing loss and cochlear damage in homozygous Foxo3-knockout mice

2021 ◽  
Vol 12 (7) ◽  
Author(s):  
Holly J. Beaulac ◽  
Felicia Gilels ◽  
Jingyuan Zhang ◽  
Sarah Jeoung ◽  
Patricia M. White
Keyword(s):  
Oxidative Stress ◽  
Hearing Loss ◽  
Noise Exposure ◽  
Multiphoton Microscopy ◽  
Cell Loss ◽  
Outer Hair Cells ◽  
Noise Induced Hearing Loss ◽  
Knock Out ◽  
Cell Death Pathway ◽  
Cochlear Damage

AbstractThe prevalence of noise-induced hearing loss (NIHL) continues to increase, with limited therapies available for individuals with cochlear damage. We have previously established that the transcription factor FOXO3 is necessary to preserve outer hair cells (OHCs) and hearing thresholds up to two weeks following mild noise exposure in mice. The mechanisms by which FOXO3 preserves cochlear cells and function are unknown. In this study, we analyzed the immediate effects of mild noise exposure on wild-type, Foxo3 heterozygous (Foxo3+/−), and Foxo3 knock-out (Foxo3−/−) mice to better understand FOXO3’s role(s) in the mammalian cochlea. We used confocal and multiphoton microscopy to examine well-characterized components of noise-induced damage including calcium regulators, oxidative stress, necrosis, and caspase-dependent and caspase-independent apoptosis. Lower immunoreactivity of the calcium buffer Oncomodulin in Foxo3−/− OHCs correlated with cell loss beginning 4 h post-noise exposure. Using immunohistochemistry, we identified parthanatos as the cell death pathway for OHCs. Oxidative stress response pathways were not significantly altered in FOXO3’s absence. We used RNA sequencing to identify and RT-qPCR to confirm differentially expressed genes. We further investigated a gene downregulated in the unexposed Foxo3−/− mice that may contribute to OHC noise susceptibility. Glycerophosphodiester phosphodiesterase domain containing 3 (GDPD3), a possible endogenous source of lysophosphatidic acid (LPA), has not previously been described in the cochlea. As LPA reduces OHC loss after severe noise exposure, we treated noise-exposed Foxo3−/− mice with exogenous LPA. LPA treatment delayed immediate damage to OHCs but was insufficient to ultimately prevent their death or prevent hearing loss. These results suggest that FOXO3 acts prior to acoustic insult to maintain cochlear resilience, possibly through sustaining endogenous LPA levels.

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