Nutritional support in wound healing

2003 ◽  
Vol 17 (23) ◽  
pp. 55-58 ◽  
Author(s):  
Georgina Casey
Keyword(s):  
Wound Healing ◽  
Nutritional Support

Nutrition: A Critical Component of Wound Healing

1990 ◽  
Vol 1 (3) ◽  
pp. 585-592 ◽  
Author(s):  
Nancy A. Stotts ◽  
Donna F. Washington
Keyword(s):  
Health Care ◽  
Wound Healing ◽  
Critical Care ◽  
Nutritional Support ◽  
Nutritional Assessment ◽  
Current Knowledge ◽  
Health Care Team ◽  
Critical Component ◽  
Assessment Technique

Nutrients are essential to wound healing and patients in critical care are often at risk for lack of sufficient nutrients that may result in impaired healing. Understanding the role of the various nutrients in healing provides the basis for assessment and therapy. Use of a practical and consistent nutritional assessment technique is an important part of care for critically ill patients with wounds. The health care team must provide care based on current knowledge of the effects of nutrition on wound healing and work collaboratively in doing nutritional assessment and providing nutritional support to optimize wound healing outcomes

scholarly journals Nutritional Support and Wound Healing

2020 ◽  
Vol 12 (2) ◽  
pp. 21-25
Author(s):  
Chi-Min Park
Keyword(s):  
Wound Healing ◽  
Nutritional Support

Parenteral Nutrition in Patients with Head and Neck Cancer: Techniques and Results

1980 ◽  
Vol 88 (6) ◽  
pp. 707-713 ◽  
Author(s):  
John M. Daly ◽  
Stanley J. Dudrick ◽  
Edward M. Copeland
Keyword(s):  
Wound Healing ◽  
Radiation Therapy ◽  
Head And Neck Cancer ◽  
Weight Gain ◽  
Head And Neck ◽  
Nutritional Support ◽  
Dietary Habits ◽  
Perioperative Period ◽  
Protein Levels ◽  
Protein Calorie Malnutrition

Protein-calorie malnutrition occurs commonly in patients with head and neck cancers, secondary to poor dietary habits and oncologic treatments that compromise further a patient's ability to ingest adequate nutrients. Intravenous nutritional support of 70 patients with head and neck malignancies has allowed weight gain, improved wound healing, and increased serum protein levels during radiation therapy, chemotherapy, and in the perioperative period.

HUBUNGAN STATUS GIZI DENGAN KECEPATAN PENYEMBUHAN LUKA OPERASI PADA ANAK DI INSTALASI RAWAT INAP A BLU RSU PROF. Dr. R. D. KANDOU MANADO

e-NERS ◽  
2013 ◽  
Vol 1 (1) ◽  
Author(s):  
Rusmini . ◽  
Jeavery Bawotong ◽  
Gresty Masi
Keyword(s):  
Wound Healing ◽  
Nutritional Status ◽  
Nutritional Support ◽  
Statistical Tests ◽  
Surgical Patients ◽  
Cross Sectional ◽  
Weak Association ◽  
Healing Of Wounds ◽  
Resistance To Infection ◽  
Positive Pattern

Abstract: Good nutritional status is important in wound healing. Mal nutritionin surgical patients can affect the morbidity due to disruption of wound healing and decreased resistance to infection. The research objective was to identification of nutritional status relationship with the speed of wound healing in children Installing A BLU Hospital In patient Prof. DR. R.D. Kandou Manado. This study uses correlation, cross-sectional approach. The sample of 25 patients. The collection of data through observation and questionnaires. The results showeda no rmalnutritional status as many as 10 patients (40%) showed healing of wounds ≤5 days, while only 1 patient (4%) with sufficient nutrition having wound healing ≥5 days. Based on statistical tests there was no association between nutritional status at speeds wound healing in children (p=0.825) and the correlation coefficient or (r=0.047) indicating that indicates the level of a weak association with the positive pattern. Conclusions The clinical studyis very influential on the nutritional status of wound healing in which nutritional support showed a betterhealing process in surgical outcomes were good but the Stats test showed there was no correlation between the two variables. Keywords: Nutritional Status, Children, Wounds Operations, Wound healing.     Abstrak: Status gizi yang baik merupakan hal penting dalam penyembuhan luka operasi. Malnutrisi pada pasien bedah dapat mempengaruhi morbiditas karena terganggunya penyembuhan luka dan menurunnya daya tahan tubuh terhadap infeksi. Tujuan penelitian adalah untuk teridentifikasinya hubungan status gizi dengan kecepatan penyembuhan luka operasi pada anak di Instalasi Rawat Inap A BLU RSU Prof. DR. R. D. Kandou Manado. Metode penelitian korelasi dengan pendekatan cross sectional. Sampel 25 pasien. Pengumpulan data melalui pengamatan dan kuesioner. Hasil penelitian menunjukan status gizi yang normal sebanyak 10 pasien (40%) menunjukan penyembuhan luka ≤ 5 hari sedangkan hanya 1 pasien (4%) dengan gizi yang cukup  memiliki penyembuhan luka ≥ 5 hari. Berdasarkan uji statistik menunjukkan tidak ada hubungan yang bermakna antara status gizi dengan kecepatan penyembuhan luka operasi (p = 0,825) dan koefisien korelasi atau (r = 0,047) yang menunjukan tingkat hubungan yang lemah dengan pola positif. Kesimpulan dalam penelitian Secara klinis status nutrisi sangat berpengaruh pada penyembuhan luka dimana dukungan nutrisi menunjukan proses penyembuhan yang lebih baik pada hasil bedah yang baik tetapi pada uji statistik menunjukan bahwa tidak ada hubungan antara kedua variabel. Kata Kunci : Status Gizi, Anak, Luka Operasi, Penyembuhan luka.

ultrastructural process of intestinal wound healing

1995 ◽  
Vol 53 ◽  
pp. 1024-1025
Author(s):  
Rick L. Vaughn ◽  
Shailendra K. Saxena ◽  
John G. Sharp
Keyword(s):  
Wound Healing ◽  
Epithelial Cells ◽  
Smooth Muscles ◽  
Microscopic Level ◽  
Light Microscopic Level ◽  
Ileal Mucosa ◽  
Wound Model ◽  
Serosal Surface ◽  
Complex Process ◽  
Orderly Fashion

We have developed an intestinal wound model that includes surgical construction of an ileo-cecal patch to study the complex process of intestinal wound healing. This allows approximation of ileal mucosa to the cecal serosa and facilitates regeneration of ileal mucosa onto the serosal surface of the cecum. The regeneration of ileal mucosa can then be evaluated at different times. The wound model also allows us to determine the rate of intestinal regeneration for a known size of intestinal wound and can be compared in different situations (e.g. with and without EGF and Peyer’s patches).At the light microscopic level it appeared that epithelial cells involved in regeneration of ileal mucosa originated from the enlarged crypts adjacent to the intestinal wound and migrated in an orderly fashion onto the serosal surface of the cecum. The migrating epithelial cells later formed crypts and villi by the process of invagination and evagination respectively. There were also signs of proliferation of smooth muscles underneath the migratory epithelial cells.

Healing gone wrong: convergence of hemostatic pathways and liver fibrosis?

2020 ◽  
Vol 134 (16) ◽  
pp. 2189-2201
Author(s):  
Jessica P.E. Davis ◽  
Stephen H. Caldwell
Keyword(s):  
Wound Healing ◽  
Liver Disease ◽  
Hepatic Fibrosis ◽  
Cell Activation ◽  
Stellate Cell ◽  
Factor Xa ◽  
Clinical Trial Data ◽  
Chronic Liver ◽  
Healing Response ◽  
Wound Healing Response

Abstract Fibrosis results from a disordered wound healing response within the liver with activated hepatic stellate cells laying down dense, collagen-rich extracellular matrix that eventually restricts liver hepatic synthetic function and causes increased sinusoidal resistance. The end result of progressive fibrosis, cirrhosis, is associated with significant morbidity and mortality as well as tremendous economic burden. Fibrosis can be conceptualized as an aberrant wound healing response analogous to a chronic ankle sprain that is driven by chronic liver injury commonly over decades. Two unique aspects of hepatic fibrosis – the chronic nature of insult required and the liver’s unique ability to regenerate – give an opportunity for pharmacologic intervention to stop or slow the pace of fibrosis in patients early in the course of their liver disease. Two potential biologic mechanisms link together hemostasis and fibrosis: focal parenchymal extinction and direct stellate cell activation by thrombin and Factor Xa. Available translational research further supports the role of thrombosis in fibrosis. In this review, we will summarize what is known about the convergence of hemostatic changes and hepatic fibrosis in chronic liver disease and present current preclinical and clinical data exploring the relationship between the two. We will also present clinical trial data that underscores the potential use of anticoagulant therapy as an antifibrotic factor in liver disease.

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