scholarly journals Low vegetable intake in pregnancy and associated maternal factors: A scoping review

2022 ◽  
Author(s):  
Kira Simmons ◽  
Nina Meloncelli ◽  
Lauren Kearney ◽  
Judith Maher
Keyword(s):  
Scoping Review ◽  
Vegetable Intake ◽  
Maternal Factors ◽  
In Pregnancy

scholarly journals Low vegetable intake in pregnancy and associated maternal factors: A scoping review

2021 ◽  
Author(s):  
Kira Simmons ◽  
Nina Meloncelli ◽  
Lauren Kearney ◽  
Judith Maher
Keyword(s):  
Pregnant Women ◽  
Scoping Review ◽  
Vegetable Intake ◽  
Economic Status ◽  
Positive Association ◽  
Inclusion Criteria ◽  
Scoping Reviews ◽  
Meta Analyses ◽  
The Relationship ◽  
In Pregnancy

Healthy eating is identified as a priority in pregnancy. Vegetables are low energy, nutrient dense foods that support health. Needs of populations differ by demographics, as such there is a need to investigate vegetable intake (VI) in pregnant women of lower socio-economic status (SES). The aim of this scoping review is 1) To describe VI during pregnancy in serves/gram and compare VI to recommendations; and 2) To explore the relationship between VI during pregnancy and maternal SES characteristics. Using Arksey and O’Malley’s framework and the Preferred Reporting Items for Systematic reviews and Meta-Analyses extension for Scoping Reviews (PRISMA-ScR), studies were identified in a search of electronic databases (MEDLINE, Web of Science, Global Health and Scopus) published up to July 2021. All observational studies assessing VI in pregnancy, written in English and conducted in an energy replete context worldwide, were included for review. Forty-seven publications met inclusion criteria. While VI of pregnant women varies across populations, VI falls below recommendations worldwide. Studies investigating older age (n=9), higher education (n=7), higher income (n=4) and VI, consistently found a positive association, whereas a negative association with food insecurity (n=4) was identified. Other variables explored that may influence VI is limited and too fragmented to generalise. Inconsistencies and possible inaccuracies in reporting VI may be related to the considerable variation in tools used for assessing VI. In conclusion, low VI in pregnancy needs to be addressed, with a particular focus on women of lower SES due to greater vulnerability to low VI.

Semi-quantitative cervical elastography in pregnancy and association with maternal factors

2013 ◽  
Vol 34 (S 01) ◽  
Author(s):  
GC Meyberg-Solomayer ◽  
C Gerlinger ◽  
A Hamza ◽  
F Schlaegel ◽  
Z Takacs ◽  
...  
Keyword(s):  
Maternal Factors ◽  
In Pregnancy

DESIGN AN “IN HOUSE” SOFTWARE FOR SCREENING PREECLAMPSIA BY MATERNAL FACTORS AND MEAN ARTERIAL PRESSURE AT 11 – 13 WEEKS IN COMMUNE HEALTH CENTERS.

2015 ◽  
pp. 115-126
Author(s):  
Viet Nhan Nguyen ◽  
Ngoc Thanh Cao ◽  
Thi Minh Thi Ha ◽  
Van Duc Vo ◽  
Quang Vinh Truong ◽  
...  
Keyword(s):  
Mean Arterial Pressure ◽  
Arterial Pressure ◽  
Gestational Hypertension ◽  
Area Under The Curve ◽  
Health Centers ◽  
Maternal Factors ◽  
Good Tool ◽  
Uterine Artery Doppler ◽  
Study Results ◽  
In Pregnancy

Objective: Design an “in house” software for screening preeclampsia by maternal factors and mean arterial pressure at 11 – 13 gestational weeks in commune health centers. Methods: Based on the algorithms for calculating the risk of preeclampsia (PE) by maternal factors and mean artirial pressure at 11 - 13 gestational weeks in the study results of the authors, an “in house” software was deigned in Excel. The results of prediction preeclampsia by The Fetal Medicine Foundation (FMF)(version 2.3) were compared with the results by “in house” software in 1110 singleton pregnant women. Results: The “in house” software met the requirements for calculating the risks of PE and save data. FMF risk for gestational hypertension disorder in pregnancy by maternal factors, mean arterial pressure,uterine artery Doppler and PAPP-A has an area under the curve of 0.68 (95%CI: 0.59 – 0.78). The “in house” software risk for gestational hypertension in pregnancy by maternal factors, mean arterial pressure has an area under the curve of 0.643 (0.55 – 0.73) There was no statistically significant different between two programs (p:0.52). The risk cut-off 1:50 in the prediction of gestational hypertension of the “in house” software was used to identify the group of high risk with detetion rate (DR) 28.6% (95%CI: 14.9-42.2) comparing to 40.5% (95%CI:25.6-55.3) of FMF. Conclusion: The FMF version 2.3 is better but in the absence of Doppler ultrasound and PAPP-A test in the commune health cares, the “in house” software for screening PE is a good tool for councelling, following up and early intervention for PE.

Congenital Limb Deficiency: Maternal Factors in Pregnancy

1986 ◽  
Vol 26 (4) ◽  
pp. 272-275 ◽  
Author(s):  
Anne Kricker ◽  
Jane Elliott ◽  
Jill Forrest ◽  
Janet McCredie
Keyword(s):  
Maternal Factors ◽  
Congenital Limb ◽  
Limb Deficiency ◽  
Congenital Limb Deficiency ◽  
In Pregnancy

scholarly journals Mood Symptoms and Other Maternal Factors in Pregnancy and Adverse Neonate Outcomes

2020 ◽  
Author(s):  
Hua Li ◽  
Angela Bowen ◽  
Rudy Bowen ◽  
Nazeem Muhajarine ◽  
Lloyd Balbuena
Keyword(s):  
Social Support ◽  
Statistical Significance ◽  
Depression Scale ◽  
Neonatal Outcomes ◽  
Maternal Factors ◽  
Mood Instability ◽  
Mood Symptoms ◽  
Antenatal Depressive Symptoms ◽  
Adverse Neonatal Outcomes ◽  
In Pregnancy

Abstract Background Antenatal women experience a higher level of mood symptoms, which have negative effects on mothers’ mental and physical health and their newborns. The relation of maternal moods including depression and anxiety and other maternal factors in pregnancy and neonate outcomes are well-studied with inconsistent findings. Although antenatal women experience a higher level of mood instability (MI), the association between antenatal MI and neonatal outcomes has not been investigated. We aimed to address this gap and to contribute to the pregnancy-neonate outcomes literature by examining the relationship between antenatal mood symptoms and other maternal factors and neonatal outcomes.Methods A prospective cohort of women (n = 555) participated in this study at early pregnancy (T1, 17.4 ± 4.9 weeks) and late pregnancy (T2, 30.6 ± 2.7 weeks). The Edinburgh Postnatal Depression Scale (EPDS) was used to assess antenatal depressive symptoms, the EPDS anxiety subscale was utilized to measure anxiety symptoms, and mood instability was measured using a visual analogue scale. These mood states together with stress, social support, as well as healthy and unhealthy behaviours were also examined in relation to neonatal outcomes using chi-square tests and logistic regression models. Results Depression and MI were unrelated to adverse neonatal outcomes and anxiety was related to Apgar score with marginal statistical significance. Higher stress, lack of partner support, smoking, and primiparous status were associated with some adverse outcomes.Conclusions The current study identified no associations between antenatal mood symptoms and neonatal outcomes. Our findings further support and extend previous evidence on smoking abstinence or cessation, and the provision of resources for stress management and social support that could help prevent or alleviate adverse neonatal outcomes.

scholarly journals Maternal factors associated with hyperglycemia in pregnancy and perinatal outcomes: a Brazilian reference center cohort study

2020 ◽  
Vol 12 (1) ◽  
Author(s):  
Bianca F. Nicolosi ◽  
Joice M. Vernini ◽  
Roberto A. Costa ◽  
Claudia G. Magalhães ◽  
Marilza V. C. Rudge ◽  
...  
Keyword(s):  
Cohort Study ◽  
Perinatal Outcomes ◽  
Maternal Factors ◽  
Reference Center ◽  
Factors Associated ◽  
In Pregnancy

565: Rate of change of lipid levels in pregnancy associated with first trimester lipid levels and maternal factors

2011 ◽  
Vol 204 (1) ◽  
pp. S226
Author(s):  
Heather Byers ◽  
Jeffery C. Murray ◽  
Bruce Bedell ◽  
Kelli K. Ryckman ◽  
Kristi S. Borowski
Keyword(s):  
First Trimester ◽  
Rate Of Change ◽  
Lipid Levels ◽  
Maternal Factors ◽  
In Pregnancy

Pressor responsiveness in pseudopregnant and pregnant rats: role of maternal factors

1989 ◽  
Vol 257 (4) ◽  
pp. R866-R871 ◽  
Author(s):  
M. S. Paller ◽  
G. Gregorini ◽  
T. F. Ferris
Keyword(s):  
Vascular Reactivity ◽  
Pressor Response ◽  
Ang Ii ◽  
Maternal Factors ◽  
Synthesis Inhibitor ◽  
Pregnant Rats ◽  
Plasma Progesterone ◽  
Early Increase ◽  
In Pregnancy

During pregnancy the pressor response to vasoconstrictor substances such as angiotensin II (ANG II) is diminished, and renal, uterine, and vascular prostaglandin (PG) production may increase. However, little is known about the factors that alter vascular reactivity or stimulate PG synthesis during pregnancy. To ascertain whether these factors are of maternal or fetal-placental origin, we studied vascular reactivity and urinary PGE excretion in pseudopregnant rats. Pseudopregnant rats had plasma progesterone and weight gain similar to that observed in pregnant rats. Urinary PG excretion in nonpregnant rats was approximately 70 ng/24 h and remained constant during a 12-day observation. In contrast, urinary PG excretion in both pregnant and in pseudopregnant rats rose to levels approximately twice control within 4-6 days. The pressor response to ANG II was diminished in pseudopregnant rats compared with nonpregnant rats. When the PG synthesis inhibitor meclofenamate was given there was no change in the pressor response to ANG II in nonpregnant animals, but in pseudopregnant animals meclofenamate produced a significant increase in the pressor response to ANG II. The pressor response to norepinephrine and arginine vasopressin (AVP) was not diminished in pseudopregnant animals, and meclofenamate did not increase the pressor response to these agents. Therefore, a developing fetus and placenta is not necessary for the decrease in pressor response to ANG II nor for the early increase in urinary PGE excretion. Like in pregnancy, the pressor response to ANG II was increased after meclofenamate in pseudopregnancy. Increased PG production may, therefore, be partly responsible for the decrease in pressor responsiveness to ANG II. However, pseudopregnancy, unlike pregnancy, did not affect pressor responsiveness to norepinephrine or AVP. Both maternal and fetal-placental factors seem required for the reduction in responsiveness to norepinephrine and AVP in pregnancy.

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