Corn (Zea mays L.) response to sublethal rates of paraquat and fomesafen at vegetative growth stages

2019 ◽  
Vol 33 (04) ◽  
pp. 595-600
Author(s):  
Benjamin P. Sperry ◽  
Benjamin H. Lawrence ◽  
Jason A. Bond ◽  
Daniel B. Reynolds ◽  
Bobby R. Golden ◽  
...  

AbstractResearch was conducted from 2013 to 2015 across three sites in Mississippi to evaluate corn response to sublethal paraquat or fomesafen (105 and 35 g ai ha−1, respectively) applied PRE, or to corn at the V1, V3, V5, V7, or V9 growth stages. Fomesafen injury to corn at three d after treatment (DAT) ranged from 0% to 38%, and declined over time. Compared with the nontreated control (NTC), corn height 14 DAT was reduced approximately 15% due to fomesafen exposure at V5 or V7. Exposure at V1 or V7 resulted in 1,220 and 1,110 kg ha−1 yield losses, respectively, compared with the NTC, but yield losses were not observed at any other growth stage. Fomesafen exposure at any growth stage did not affect corn ear length or number of kernel rows relative to the NTC. Paraquat injury to corn ranged from 26% to 65%, depending on growth stage and evaluation interval. Corn exposure to paraquat at V3 or V5 consistently caused greater injury across evaluation intervals, compared with other growth stages. POST timings of paraquat exposure resulted in corn height reductions of 13% to 50%, except at V7, which was most likely due to rapid internode elongation at that stage. Likewise, yield loss occurred after all exposure times of paraquat except PRE, compared with the NTC. Corn yield was reduced 1,740 to 5,120 kg ha−1 compared with the NTC, generally worsening as exposure time was delayed. Paraquat exposure did not reduce corn ear length, compared with the NTC, at any growth stage. However, paraquat exposure at V3 or V5 was associated with reduction of kernel rows by 1.1 and 1.7, respectively, relative to the NTC. Paraquat and fomesafen applications near corn should be avoided if conditions are conducive for off-target movement, because significant injury and yield loss can result.

Weed Science ◽  
2016 ◽  
Vol 64 (3) ◽  
pp. 495-500 ◽  
Author(s):  
Jill Alms ◽  
Sharon A. Clay ◽  
David Vos ◽  
Michael Moechnig

The widespread adoption of glyphosate-resistant corn and soybean in cropping rotations often results in volunteer plants from the previous season becoming problem weeds that require alternative herbicides for control. Corn yield losses due to season-long volunteer soybean competition at several densities in two growing seasons were used to define a hyperbolic yield loss function. The maximum corn yield loss observed at high volunteer soybean densities was about 56%, whereas, the incremental yield loss (I) at low densities was 3.2%. Corn yield loss at low volunteer soybean densities was similar to losses reported for low densities of velvetleaf and redroot pigweed, with 10% yield loss estimated to occur at 3 to 4 volunteer soybean plants m−2. Several herbicides, including dicamba with or without diflufenzopyr applied at the V2 growth stage of volunteer soybean, provided > 90% control, demonstrating several economical options to control volunteer glyphosate-resistant soybean in glyphosate-resistant corn. Reevaluation of control recommendations may be needed with commercialization of other genetically modified herbicide-resistant soybean varieties.


Plant Disease ◽  
2010 ◽  
Vol 94 (7) ◽  
pp. 820-826 ◽  
Author(s):  
Christian D. Cruz ◽  
Dennis Mills ◽  
Pierce A. Paul ◽  
Anne E. Dorrance

Brown spot, caused by Septoria glycines, is the most common foliar disease of soybean in Ohio, but its economic impact has not been assessed on modern cultivars. Therefore, the objectives of this study were to (i) evaluate the effect of S. glycines on soybean yield and (ii) evaluate the efficacy of strobilurin- and triazole-based fungicides on the control of brown spot. Yield loss associated with S. glycines was determined using weekly applications of chlorothalonil. The efficacy of azoxystrobin, pyraclostrobin, tebuconazole, and flutriafol alone and in combinations were also assessed using applications at the R3 and R5 growth stages at two locations over 3 years. Significantly different levels of brown spot developed following applications of chlorothalonil, with mean yield differences between treated and nontreated plots ranging from 196 to 293 kg/ha. Pyraclostrobin and azoxystrobin applied at the R3 growth stage significantly reduced final levels of brown spot; however, significant increases in yield occurred in only three of the six location-years. Triazoles, flutriafol and tebuconazole, applied at R3 or R5 did not significantly decrease levels of brown spot or impact yield. More data on the accurate timing of fungicides are still required to establish a long-term management program for this disease, and resistance to brown spot should be monitored in soybean cultivar development to prevent future yield losses.


2020 ◽  
pp. 1-5
Author(s):  
Hunter D. Bowman ◽  
Tom Barber ◽  
Jason K. Norsworthy ◽  
Trenton L. Roberts ◽  
Jason Kelley ◽  
...  

Abstract Previous research has shown that glufosinate and nicosulfuron at low rates can cause yield loss to grain sorghum. However, research has not been conducted to pinpoint the growth stage at which these herbicides are most injurious to grain sorghum. Therefore, field tests were conducted in 2016 and 2017 to determine the most sensitive growth stage for grain sorghum exposure to both glufosinate and nicosulfuron. Field test were designed with factor A being the herbicide applied (glufosinate or nicosulfuron). Factor B consisted of timing of herbicide application including V3, V8, flagleaf, heading, and soft dough stages. Factor C was glufosinate or nicosulfuron rate where a proportional rate of 656 g ai ha−1 of glufosinate and 35 g ai ha−1 of nicosulfuron was applied at 1/10×, 1/50×, and 1/250×. Visible injury, crop canopy heights (cm), and yield were reported as a percent of the nontreated. At the V3 growth stage visible injury of 32% from the 1/10× rate of glufosinate and 51% from the 1/10× rate of nicosulfuron was observed. This injury was reduced by 4 wk after application (WAA) and no yield loss occurred. Nicosulfuron was more injurious than glufosinate at a 1/10× and 1/50× rate when applied at the V8 and flagleaf growth stages resulting in death of the shoot, reduced heading, and yield. Yield losses from the 1/10× rate of nicosulfuron were observed from V8 through early heading and ranged from 41% to 96%. Yield losses from the 1/50× rate of nicosulfuron were 14% to 16% at the flagleaf and V8 growth stages respectively. The 1/10× rate of glufosinate caused 36% visible injury 2 WAA when applied at the flagleaf stage, which resulted in a 16% yield reduction. By 4 WAA visible injury from either herbicide at less than the 1/10× rate was not greater than 4%. Results indicate that injury can occur, but yield losses are more probable from low rates of nicosulfuron at V8 and flagleaf growth stages.


2019 ◽  
Vol 33 (04) ◽  
pp. 555-561 ◽  
Author(s):  
Alanna B. Scholtes ◽  
Benjamin P. Sperry ◽  
Daniel B. Reynolds ◽  
J. Trenton Irby ◽  
Thomas W. Eubank ◽  
...  

AbstractField experiments were conducted in 2012 and 2013 across four locations for a total of 6 site-years in the midsouthern United States to determine the effect of growth stage at exposure on soybean sensitivity to sublethal rates of dicamba (8.8 g ae ha−1) and 2,4-D (140 g ae ha−1). Regression analysis revealed that soybean was most susceptible to injury from 2,4-D when exposed between 413 and 1,391 accumulated growing degree days (GDD) from planting, approximately between V1 and R2 growth stages. In terms of terminal plant height, soybean was most susceptible to 2,4-D between 448 and 1,719 GDD, or from V1 to R4. However, maximum susceptibility to 2,4-D was only between 624 and 1,001 GDD or from V3 to V5 for yield loss. As expected, soybean was sensitive to dicamba for longer spans of time, ranging from 0 to 1,162 GDD for visible injury or from emergence to R2. Likewise, soybean height was most affected when dicamba exposure occurred between 847 and 1,276 GDD or from V4 to R2. Regarding grain yield, soybean was most susceptible to dicamba between 820 and 1,339 GDD or from V4 to R2. Consequently, these data indicate that soybean response to 2,4-D and dicamba can be variable within vegetative or reproductive growth stages; therefore, specific growth stage at the time of exposure should be considered when evaluating injury from off-target movement. In addition, application of dicamba near susceptible soybean within the V4 to R2 growth stages should be avoided because this is the time of maximum susceptibility. Research regarding soybean sensitivity to 2,4-D and dicamba should focus on multiple exposure times and also avoid generalizing growth stages to vegetative or reproductive.


2015 ◽  
Vol 42 (2) ◽  
pp. 109-120 ◽  
Author(s):  
B.H. Blanchett ◽  
T.L. Grey ◽  
E.P. Prostko ◽  
T.M. Webster

ABSTRACT The development of dicamba-resistant cotton and soybean cultivars has created great concern about the potential off-target movement of dicamba onto sensitive species, including broadleaf crops. Peanut is often grown in close proximity to cotton and soybean. Therefore, field studies were conducted during 2012 and 2013 at Plains, Ty Ty, and Attapulgus, GA to evaluate peanut response to rates of dicamba (35, 70, 140, 280, and 560 g ae ha−1) applied at preemergence (PRE), 10, 20, or 30 d after planting (DAP) corresponding to PRE, V2, V3, and V5 peanut growth stages, respectively. Nontreated controls were included for comparison. As dicamba rate increased, both peanut injury and peanut yield loss increased. Peanut response to dicamba was fit to log-logistic regression models for injury and linear regression models for yield loss. Peanut injury increased with rate of dicamba, but was variable among the locations. A general trend was that peanut plants became more sensitive to dicamba injury as plants approached reproductive stage, as evidenced through a declining linear relationship between I50 values (i.e. rate of dicamba that elicits a 50% crop response) and timing of application. PRE applications of dicamba had I50 values that ranged from 125 to 323 g ha−1 of dicamba, while I50 values were 44 to 48 g ha−1 of dicamba at the V5 peanut growth stage. There was a linear relationship between peanut yield and dicamba rate, with 560 g ha−1 causing maximum yield losses ranging from 0 to 86% when applied PRE, 24 to 82% when applied at V2 growth stage, 30 to 95% when applied at V3 growth stage, and 45 to 88% when applied at V5 growth stage. Across all treatments and locations, there was also a negative linear relationship between peanut yield and peanut crop injury, with a decline of 8.5% yield for every 10% increase in crop injury. Growers and their consultants/extension agents can use this peanut injury data to predict potential peanut yield loss from sprayer contamination or off-target movement of dicamba.


Weed Science ◽  
2014 ◽  
Vol 62 (4) ◽  
pp. 647-656 ◽  
Author(s):  
Elbé Hugo ◽  
Liesl Morey ◽  
Anna E. J. Saayman-Du Toit ◽  
Carl F. Reinhardt

Difficulties in chemically controlling large crabgrass in corn in South Africa have recently been attributed to the occurrence of naked crabgrass. In contrast to large crabgrass, naked crabgrass is not easily controlled with acetanilide herbicides. Critical periods of weed control (CPWC) for naked crabgrass in corn was determined in field studies during the 2009/2010 and 2010/2011 growing seasons at two separate localities for an early and late planting date of corn. Weed-free and weed–crop interference treatments of increasing duration were maintained at various crop growth stages in the presence of naked crabgrass. Biomass of naked crabgrass was determined as dry weight per square meter quadrant, which yielded 428 g m−2at Potchefstroom and 594 g m−2at Wesselsbron. An exponential regression model was used to determine the CPWC expressed as growing degree days after crop emergence, on the basis of an estimated 10% relative yield loss in corn. The onset and ending, as well as the duration of the CPWC, differed between seasons and localities. At 10% relative yield loss, the onset of the CPWC ranged between the two-leaf (V2) and six-leaf (V6) stages, and the ending between the 12-leaf (V12) stage and 2 wk after tasseling (T + 2). The duration of the CPWC ranged between 22 and 80 d for the respective planting dates, years, and localities. Yield losses ranged from 28 to 82% in the season-long weedy plots. The shifting of planting dates alone did not reduce yield losses since the effect of late infestations of naked crabgrass is significant. Naked crabgrass control from crop emergence is essential, followed by POST herbicide application during the critical period of weed control to lower the risk of corn yield losses.


2017 ◽  
Vol 44 (1) ◽  
pp. 53-59 ◽  
Author(s):  
B.H. Blanchett ◽  
T.L. Grey ◽  
E.P. Prostko ◽  
W.K. Vencill ◽  
T.M. Webster

ABSTRACT The development of 2,4-D-resistant cotton and soybean cultivars has created great concern about the potential off-target movement of 2,4-D onto sensitive broadleaf crops. Peanut is often grown in close proximity to cotton and soybean. Therefore, field studies were conducted during 2012 and 2013 at Plains, Ty Ty, and Attapulgus, GA to evaluate peanut response to 2,4-D at 67, 133, 266, 533, and 1066 g ae ha−1 applied at preemergence (PRE), 10, 20, or 30 d after planting (DAP), corresponding to PRE, V2, V3, and V5 peanut growth stages. Nontreated controls (NTC) were included for comparison. Treatment timing by rate interactions were significant (P < 0.0001). As 2,4-D rate increased peanut injury increased. There was variation in yield loss response dependent on peanut growth stage at application timing. Peanut that was treated preemergence and at the V2 growth stage did not have yield loss at any of the 2,4-D evaluated rates (67 to 1066 g ha−1) relative to the NTC. When peanut was treated at V3 and V5 growth stages with 2,4-D, injury estimates were 5 to 32% from the 67 to 1066 g ha−1 rates respectively, and peanut canopy diameter was stunted 5 to 35% at the same rates. The resulting peanut yield loss was 23 and 36% from 533 and 1066 g ha−1 of 2,4-D applied at V3 and V5 growth stages; in part due to reproductive growth being initiated during that time-frame and peanut had less time to recuperate before harvest. Linear regression models were used to evaluate peanut injury and peanut yield results. Significant correlations were established for V3 and V5 treatments between injury and yield, injury and canopy diameter, and canopy diameter and yield (P < 0.0001), with correlation coefficients of − 0.48, − 0.76, and 0.51, respectively. Growers and extension agents will be able to use these peanut injury estimates and canopy diameter data to make improved predictions of potential peanut yield loss where off-target movement of 2,4-D or sprayer contamination has occurred.


Plant Disease ◽  
2001 ◽  
Vol 85 (5) ◽  
pp. 517-520 ◽  
Author(s):  
O. Erincik ◽  
L. V. Madden ◽  
D. C. Ferree ◽  
M. A. Ellis

Intact ‘Seyval’ grape clusters in the greenhouse and ‘Catawba’ clusters in the field were inoculated with conidia of Phomopsis viticola at seven Eichorn-Lorenz growth stages between 12 (prebloom) and 35 (véraison) in 1998. Five pots (10 clusters) were used per inoculation, and the experiment was repeated three times. Also, 10 to 20 randomly selected Catawba clusters were inoculated in the field for each of three replications at each growth stage. Studies were repeated in 1999. In addition, Chambourcin clusters were also inoculated at four growth stages in the greenhouse in 1999. Results obtained in the greenhouse and field during both years and for all cultivars indicate that berry and rachis infections can occur at all growth stages between 12 and 35 with no evidence of decreasing susceptibility over time. Results disagree with some literature reports that indicate that berry infection occurs primarily during bloom and shortly after bloom, and susceptibility decreases as fruit matures.


2019 ◽  
Vol 99 (4) ◽  
pp. 437-443
Author(s):  
Nader Soltani ◽  
Robert E. Nurse ◽  
Amit J. Jhala ◽  
Peter H. Sikkema

A study consisting of 13 field experiments was conducted during 2014–2016 in southwestern Ontario and southcentral Nebraska (Clay Center) to determine the effect of late-emerging weeds on the yield of glyphosate-resistant soybean. Soybean was maintained weed-free with glyphosate (900 g ae ha−1) up to the VC (cotyledon), V1 (first trifoliate), V2 (second trifoliate), V3 (third trifoliate), V4 (fourth trifoliate), and R1 (beginning of flowering) growth stages, after which weeds were allowed to naturally infest the soybean plots. The total weed density was reduced to 24%, 63%, 67%, 72%, 76%, and 92% in Environment 1 (Exeter, Harrow, and Ridgetown) when soybean was maintained weed-free up to the VC, V1, V2, V3, V4, and R1 soybean growth stages, respectively. The total weed biomass was reduced by 33%, 82%, 95%, 97%, 97%, and 100% in Environment 1 (Exeter, Harrow, and Ridgetown) and 28%, 100%, 100%, 100%, 100%, and 100% in Environment 2 (Clay Center) when soybean was maintained weed-free up to the VC, V1, V2, V3, V4, and R1 stages, respectively. The critical weed-free periods for a 2.5%, 5%, and 10% yield loss in soybean were the V1–V2, VC–V1, and VC–V1 soybean stages in Environment 1 (Exeter, Harrow, and Ridgetown) and V2–V3, V2–V3, and V1–V2 soybean stages in Environment 2 (Clay Center), respectively. For the weed species evaluated, there was a minimal reduction in weed biomass (5% or less) when soybean was maintained weed-free beyond the V3 soybean growth stage. These results shows that soybean must be maintained weed-free up to the V3 growth stage to minimize yield loss due to weed interference.


2018 ◽  
Vol 33 (1) ◽  
pp. 87-94
Author(s):  
Daniel O. Stephenson ◽  
Todd A. Spivey ◽  
Michael A. Deliberto ◽  
David C. Blouin ◽  
Brandi C. Woolam ◽  
...  

AbstractAll herbicides will move off-target to sensitive crops when not applied correctly. Therefore, low-dose applications of flumioxazin and metribuzin were evaluated in soybean at the unifoliate, V2, and V4 growth stages. Rates evaluated were 12.5%, 25%, and 50% of the labeled use rates of 72 and 316 g ai ha−1 of flumioxazin and metribuzin, respectively. Flumioxazin injury was characterized by necrosis and visible height and width reduction. Injury increased with rate 3 d after treatment (DAT), with unifoliate, V2, and V4 soybean injured 15% to 30%, 18% to 27%, and 5% to 8%, respectively. Unifoliate and V4 soybean were injured more than V4 soybean 3 to 14 DAT, but injury decreased to <5% by 42 DAT. Soybean yields in the flumioxazin study were 92% to 96% of the nontreated, resulting in a yield loss of 196 to 393 kg ha−1 and a revenue loss of 71 to 141 US$ ha−1. Metribuzin injury was primarily chlorosis with necrosis and a visible reduction in soybean height and width. Soybean at the V2 growth stage was injured 14% more than V4 soybean 3 DAT, regardless of metribuzin rate. Injury to V2 and V4 soybean was similar 14 DAT, with injury of 21% to 40% across rates. Soybean injury when treated at the V2 and V4 growth stages was 6% to 29% 42 DAT compared to unifoliate soybean at 0 to 17%. Soybean yields in the metribuzin study yields were 96% to 98% of the nontreated. However, a 2% to 4% reduction equates to a loss of 90 to 180 kg ha−1 and a revenue loss of 32 to 65 US$ ha−1. Unifoliate and V2 soybean are more sensitive to a low dose of flumioxazin POST, and V2 and V4 soybean are more sensitive to a low dose of metribuzin POST. Injury and the impact on soybean growth could potentially cause economic loss for a soybean producer.


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