scholarly journals BINGE EATING AMONG OLDER WOMEN: PREVALENCE RATES AND HEALTH CORRELATES ACROSS THREE UNIQUE SAMPLES

2019 ◽  
Vol 3 (Supplement_1) ◽  
pp. S927-S927
Author(s):  
Lisa S Kilpela ◽  
Carolyn B Becker ◽  
Francesca Gomez ◽  
Keesha Middlemass ◽  
Sara E Espinoza ◽  
...  

Abstract Emerging research indicates that older women struggle with disordered eating more frequently than once thought. Among older women, binge eating (BE; consuming unusually large amounts of food in one siting while feeling a loss of control) appears to be the most common form of disordered eating. Notably, BE is associated with significant medical morbidity, including metabolic dysfunction and chronic pain. Aging-related experiences, such as sleep disruptions following menopause, mood changes and psychosocial stressors, may increase risk for BE. This study comprises a 3-sample comparison of BE prevalence and health correlates among older women. We gathered self-reported frequencies of BE in three separate samples of older women, using three different methods and validated measures. Sample 1: N = 185 women aged 60-83; collected online via snowball sampling and Amazon MTurk; 86% White. Sample 2: N = 100 women aged 55-79; collected online via snowball sampling; 72% White; 50% Masters/Doctoral Degree; 72% married. Sample 3: N = 64 women aged 66+ living with food insecurity, collected in person at local food pantries; 65% Hispanic, 16% African American; 39% disabled status; 48% < high school/GED; 47% household income < $10,000/year. Per DSM-5 frequency criterion of BE at least weekly, we found prevalence rates ranging from 19%-26.5% across the samples; correlates included elevated negative mood, worry, and BMI, and less nutritious food consumption. Across three very different samples in terms of race/ethnicity, education, and food security status, we found consistent rates of self-reported BE at least weekly (roughly 1/5). Implications will be discussed.

2021 ◽  
Author(s):  
Salome Adelia Wilfred ◽  
Carolyn Black Becker ◽  
Kathryn E Kanzler ◽  
Nicolas Musi ◽  
Sara E Espinoza ◽  
...  

Abstract BackgroundEmerging research indicates that older women struggle with binge eating (BE; consuming unusually large amounts of food in one siting while feeling a loss of control) more frequently than once thought. Yet, health correlates of BE in older adult populations are poorly understood. The original goal of the study was to investigate BE prevalence, frequency, and health correlates in a sample of older adult women. Following surprising findings in this first study, we then aimed to replicate findings in two additional samples of older adult women. MethodUsing self-reported frequencies of BE from three separate samples of older women with very different demographics, we compared BE prevalence, frequency, and health correlates among older women. Study 1 (N = 185) includes data collected online (86% White; 59% overweight/obese status). Study 2 (N = 64) was conducted in person at a local food pantry (65% Hispanic; 47% household income < $10,000/year). Study 3 (N = 100) comprises data collected online (72% White; 50% Masters/Doctoral Degree). ResultsPer DSM-5 frequency criterion of BE at least weekly, we found prevalence rates ranging from 19%-26% across the three samples. Correlates of BE frequency included elevated negative mood, worry, BMI, and less nutritious food consumption. ConclusionsAcross three very different samples in terms of race/ethnicity, education, food security status, measurements, and sampling methodology, we found fairly consistent rates of self-reported BE at least weekly (19-26%). BE is related to negative outcomes among older women, supporting the need for more research in this population.


2021 ◽  
Vol 9 (1) ◽  
Author(s):  
Salomé Adelia Wilfred ◽  
Carolyn Black Becker ◽  
Kathryn E. Kanzler ◽  
Nicolas Musi ◽  
Sara E. Espinoza ◽  
...  

Abstract Background Emerging research indicates that binge eating (BE; consuming unusually large amounts of food in one siting while feeling a loss of control) is prevalent among older women. Yet, health correlates of BE in older adult populations are poorly understood. The original study aimed to investigate BE prevalence, frequency, and health correlates in a sample of older adult women. Based on results from this first study, we then sought to replicate findings in two additional samples of older adult women from separate studies. Method Using self-reported frequencies of BE from three separate samples of older women with very different demographics, we compared BE prevalence, frequency, and health correlates among older women. Study 1 (N = 185) includes data collected online (86% White; 59% overweight/obese status). Study 2 (N = 64) was conducted in person at a local food pantry (65% Hispanic; 47% household income < $10,000/year). Study 3 (N = 100) comprises data collected online (72% White; 50% Masters/Doctoral Degree). Results Per DSM-5 frequency criterion of BE at least weekly, we found prevalence rates ranging from 19 to 26% across the three samples. Correlates of BE frequency included elevated negative mood, worry, BMI, and less nutritious food consumption. Conclusions Across three very different samples in terms of race/ethnicity, education, food security status, measurements, and sampling methodology, we found fairly consistent rates of self-reported BE at least weekly (19–26%). Results suggest that BE is related to negative health indices among older women and support the need for more research in this population.


Author(s):  
Tracey D. Wade

The current chapter reviews our progress in understanding how genes influence eating and eating disorders (EDs) by addressing the following areas: (1) how recognition of genetic influences on eating and EDs emerged; (2) the complex nature of genetic action; (3) what twin studies can tell us about genetic influences; and (4) the current state of linkage and association studies. It is concluded that genes are an important part of the explanatory framework for the etiology of EDs, with an important contribution of the shared environment to the development of cognition and attitudes that may initiate disordered eating practices, and a critical contribution of the environment in providing a context within which genetic risk is more likely to be expressed. We currently have a limited understanding of the specific genes that are implicated, and the ways in which genes and the environment work together to increase risk for disordered eating.


2020 ◽  
pp. 003329412097175
Author(s):  
Johanna Levallius ◽  
Elin Monell ◽  
Andreas Birgegård ◽  
David Clinton ◽  
Emma Forsén Mantilla

Introduction Binge eating is a common behaviour that is strongly linked to both obesity and eating disorder. There is evidence that binge eating commonly co-occurs with other problematic and addictive-like behaviours; however, this has not been explored systematically. The present study aimed to examine the relationship between binge eating, body weight, disordered eating behaviours and associated addictive-like behaviours, with particular attention paid to gender differences. Method A community sample ( N = 500; 75% female, Mage = 32.5 years) reported disordered eating behaviours (i.e. binge eating, purging, restriction of eating, compulsive exercise), body mass index (BMI), food addiction, starvation addiction, exercise dependence, tobacco use and alcohol consumption. Results 42% of females and 21% of males reported binge eating during the past four weeks. Binge eating was significantly associated with all investigated behaviours in females, and with purging, compulsive exercise and overweight/obesity in males. Controlling for BMI, self-starvation predicted binge eating in males (OR = 1.07), while food addiction (OR = 1.73) and alcohol dependence (OR = 1.11) predicted binge eating in females. Conclusions The multiple associations between binge eating and addictive-like behaviors supports broad screening and generalized prevention efforts. Prevention efforts should reflect gender differences.


2021 ◽  
pp. 135910532098688
Author(s):  
Marta de Lourdes ◽  
Ana Pinto-Bastos ◽  
Paulo PP Machado ◽  
Eva Conceição

This study explored the associations between individuals presenting different problematic eating behaviors (Objective/Subjective binge-eating—OBE/SBE—and Compulsive/Non-compulsive grazing—C_Grazing/NC_Grazing) and eating disorder related symptoms. About 163 pre- and 131 post-bariatric patients were assessed. Assessment included: Face-to-face clinical interview to assess binge-eating and grazing episodes, and self-report measures to assess eating disorder symptomatology, psychological distress, and negative urgency. OBE and NC_Grazing were the problematic eating behaviors most and least associated with psychopathology, respectively. OBE and C_Grazing uniquely accounted for the significant variance in the most disordered eating variables. Our findings emphasize the need for the conceptualization of grazing behavior in the spectrum of disordered eating.


Author(s):  
Myrna M. Weissman ◽  
John C. Markowitz ◽  
Gerald L. Klerman

This chapter provides an overview of the use of IPT for patients with eating disorders. The most common eating disorders are anorexia nervosa, bulimia nervosa, and binge eating disorder. The chapter discusses the adaptations of IPT that have been used for the treatment of eating disorders and evaluates their performance in research studies. The assumption for testing IPT with eating disorders is that they occur in response to distress at poor social and interpersonal functioning and consequent negative mood, to which the patient responds with maladaptive eating behaviors. For anorexia nervosa, few data provide evidence for the benefit of IPT. For bulimia and binge eating disorder, however, IPT is considered a viable option for treatment and is recommended in numerous guidelines. A case example of a woman with bulimia nervosa is provided.


Nutrients ◽  
2020 ◽  
Vol 12 (11) ◽  
pp. 3521
Author(s):  
David A. Wiss ◽  
Nicole Avena ◽  
Mark Gold

The role of stress, trauma, and adversity particularly early in life has been identified as a contributing factor in both drug and food addictions. While links between traumatic stress and substance use disorders are well documented, the pathways to food addiction and obesity are less established. This review focuses on psychosocial and neurobiological factors that may increase risk for addiction-like behaviors and ultimately increase BMI over the lifespan. Early childhood and adolescent adversity can induce long-lasting alterations in the glucocorticoid and dopamine systems that lead to increased addiction vulnerability later in life. Allostatic load, the hypothalamic-pituitary-adrenal axis, and emerging data on epigenetics in the context of biological embedding are highlighted. A conceptual model for food addiction is proposed, which integrates data on the biological embedding of adversity as well as upstream psychological, social, and environmental factors. Dietary restraint as a feature of disordered eating is discussed as an important contextual factor related to food addiction. Discussion of various public health and policy considerations are based on the concept that improved knowledge of biopsychosocial mechanisms contributing to food addiction may decrease stigma associated with obesity and disordered eating behavior.


2019 ◽  
Vol 115 ◽  
pp. 61-68 ◽  
Author(s):  
Jennifer Svaldi ◽  
Dustin Werle ◽  
Eva Naumann ◽  
Eva Eichler ◽  
Matthias Berking

2020 ◽  
Vol 9 (4) ◽  
pp. 1187 ◽  
Author(s):  
Mohamed Abdulkadir ◽  
Moritz Herle ◽  
Bianca L. De Stavola ◽  
Christopher Hübel ◽  
Diana L. Santos Ferreira ◽  
...  

Background: Disordered eating (DE) is common and is associated with body mass index (BMI). We investigated whether genetic variants for BMI were associated with DE. Methods: BMI polygenic scores (PGS) were calculated for participants of the Avon Longitudinal Study of Parents and Children (ALSPAC; N = 8654) and their association with DE tested. Data on DE behaviors (e.g., binge eating and compensatory behaviors) were collected at ages 14, 16, 18 years, and DE cognitions (e.g., body dissatisfaction) at 14 years. Mediation analyses determined whether BMI mediated the association between the BMI-PGS and DE. Results: The BMI-PGS was positively associated with fasting (OR = 1.42, 95% CI = 1.25, 1.61), binge eating (OR = 1.28, 95% CI = 1.12, 1.46), purging (OR = 1.20, 95% CI = 1.02, 1.42), body dissatisfaction (Beta = 0.99, 95% CI = 0.77, 1.22), restrained eating (Beta = 0.14, 95% CI = 0.10, 1.17), emotional eating (Beta = 0.21, 95% CI = 0.052, 0.38), and negatively associated with thin ideal internalization (Beta = −0.15, 95% CI = −0.23, −0.07) and external eating (Beta = −0.19, 95% CI = −0.30, −0.09). These associations were mainly mediated by BMI. Conclusions: Genetic variants associated with BMI are also associated with DE. This association was mediated through BMI suggesting that weight potentially sits on the pathway from genetic liability to DE.


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