Allergy and Urinary Infections: Is There an Association?

PEDIATRICS ◽  
1971 ◽  
Vol 48 (1) ◽  
pp. 166-166
Author(s):  
Anne Harrison
Keyword(s):  
Abdominal Pain ◽  
Acute Pyelonephritis ◽  
Urine Culture ◽  
Hay Fever ◽  
Laboratory Work ◽  
Low Grade ◽  
Chronic Infections ◽  
Urinary Infections

Noting in my small general pediatric practide that the same families kept returning for urine checks following infections as well as for allergy shots, I decided to review all of the current charts to determine the incidence of both problems. "Allergy" includes hay fever, asthma, allergic cough, eczema, bee and other stings, and reaction to foods or drugs severe enough to warrant avoidance, medication, or allergic workup. "G-U infection" includes typical symptoms of bladder irritation with or without pyuria and bacteriuria, acute pyelonephritis diagnosed by the usual symptomatology and laboratory work, and subacute or chronic infections with the children presenting often with more subtle symptoms (tired, cranky, low-grade fever, and off-and-on abdominal pain being the most common complaints) and with a "clean-catch" urine culture of at least 20,000 colonies of one organism.

The effect of ideal urine culture time on urinary infections after flexible ureteroscopy: A match‐pair case‐control study

2021 ◽  
Author(s):  
Mehmet Çağlar Çakıcı ◽  
Özgür Kazan ◽  
Muhammet Çiçek ◽  
Ayberk İplikçi ◽  
Asıf Yıldırım ◽  
...  
Keyword(s):  
Case Control Study ◽  
Urine Culture ◽  
Case Control ◽  
Flexible Ureteroscopy ◽  
Culture Time ◽  
Match Pair ◽  
Urinary Infections ◽  
Control Study

scholarly journals P045 Nothing is too 'cagey' for the rheumatologist

Rheumatology ◽  
2021 ◽  
Vol 60 (Supplement_1) ◽  
Author(s):  
Muhammad F Kazmi
Keyword(s):  
Weight Loss ◽  
Inflammatory Markers ◽  
Low Grade ◽  
Careful Monitoring ◽  
Chronic Infections ◽  
Asian Origin ◽  
Travel History ◽  
Chest Clinic ◽  
History Of ◽  
Inflammatory Changes

Abstract Background/Aims  Rheumatological conditions can present with a number of non-specific features like arthralgia, fever, fatigue, weight loss along with raised inflammatory markers and positive antibodies. Due to this, when similar symptoms are referred for input it is very important to consider other ‘mimics’. We report a case of Pigeon fancier’s lung presenting with these symptoms which was referred as likely connective tissue disease. Methods  A 52-year-old lady of South Asian origin was referred by her GP with six month history of 3kg weight loss, arthralgia, fatigue, low grade fever and persistently raised inflammatory markers (ESR ranging from 50-64 mm/hr, CRP 10-14 mg/L, normal BMI). On further questioning there was history of mouth ulcers, non-specific rash, occasional cough but no Raynaud’s or joint swelling. Blood investigations showed weakly positive ANA and RF but negative ENA, DNA, antiCCP , CK, C3,C4. C-ANCA was positive but PR3 negative. CXR was clear and tests for chronic infections including TB were negative. Due to lack of objective CTD signs, plan was to take a careful monitoring approach to see if clinical features evolved. A month later due to worsening cough, a CT chest/abdomen arranged by GP showed ground-glass changes consistent with pneumonitis and hence her rheumatology appointment was expedited to see if there was an autoimmune unifying diagnosis. She was also referred by her GP to the chest clinic in view of CT report and mild shortness of breath. Results  On further review, again there were no objective CTD signs. On direct questioning there was history of travelling before worsening chest symptoms to South Asia. Also around a year before her symptoms started she was given an African grey parrot. Based on this, serology for Avian precipitin was checked which showed strongly positive IgG antibodies to avian antigens (Budgerigar droppings and feathers, Pigeon feathers IgG Abs) confirming the diagnosis of pigeon fanciers lung. She fulfilled the diagnostic criteria and was asked to avoid the trigger. Urgent respiratory input was arranged where diagnosis was agreed with and disease was deemed sub-acute in presentation. Due to PFTs showing low transfer factor of 38%, Prednisolone was started with significant improvement within few days. Review of CT chest only showed inflammatory changes and no established fibrosis predicting excellent prognosis as delay in treatment can cause irreversible pulmonary fibrosis. Conclusion  A number of conditions can mimic rheumatological conditions which usually turn out to be either infectious or malignant in origin. This case highlights the importance of considering other differentials and along with taking a travel history also asking for other possible triggers like pets. In similar scenarios the diagnosis may be ‘cagey’ but as rheumatologists we are expected to answers questions which others can’t. Disclosure  M.F. Kazmi: None.

Rheumatic Pneumonia: The Need for a New Approach

PEDIATRICS ◽  
1975 ◽  
Vol 56 (6) ◽  
pp. 1075-1078
Author(s):  
Steven P. Serlin ◽  
Mary Ellen Rimsza ◽  
John H. Gay
Keyword(s):  
Abdominal Pain ◽  
Case Report ◽  
Acute Appendicitis ◽  
Mexican American ◽  
Community Hospital ◽  
Good Health ◽  
Low Grade ◽  
New Approach ◽  
Rare Manifestation ◽  
Comprehensive Discussion

Rheumatic pneumonia is a well-described, poorly understood, rare manifestation of rheumatic fever that is generally fatal. Until 1958, when Brown and his colleagues presented their comprehensive discussion, pediatric journals provided only five references. Since then, only one article has appeared in the pediatric literature. As illustrated by the following case report, pediatricians need to be aware of rheumatic pnuemonia in order to determine optimal therapy and management. CASE REPORT A.M., a 13-year-old Mexican-American boy, was in apparent good health until he developed fleeting arthralgia, abdominal pain, and low-grade fever. The day following the onset of symptoms acute appendicitis was suspected, and a laparotomy was performed at a community hospital.

scholarly journals Incidence of Acute Pyelonephritis in Hospitalized Children under 5 Years Old with Positive Urine Culture

2018 ◽  
Vol 59 (4) ◽  
pp. 1-5
Author(s):  
Alexandra Castaño González ◽  
Juan Gabriel Ruiz Peláez
Keyword(s):  
Urinary Tract Infection ◽  
Urinary Tract ◽  
Tract Infection ◽  
Acute Pyelonephritis ◽  
Urine Culture ◽  
Renal Scintigraphy ◽  
Hospitalized Children ◽  
Positive Urine Culture ◽  
Positive Urine ◽  
Urinary Culture

Introduction: Urinary tract infection is a major cause of child morbidity. The diagnosis of acute pyelonephritis is important to decide the treatment. Methods: Retrospective observational study. We collected information of urinalysis, urine Gram and urine culture of hospitalized children between 3 months and 5 years old, with suspected urinary tract infection between January 2008 and December 2010. In patients with positive urine culture, the results of renal scintigraphy (Gamma scan) were evaluated to estimate the incidence of acute pyelonephritis. Results: We identified 1,463 medical records. Urinary culture was obtained in 237 patients, of whom 54.4% were positive. Renal scintigraphy was obtained in 93 of these patients and 59.1% were positive. Conclusions: The incidence of acute pyelonephritis in patients with confirmed urinary tract infection was 59.1%.

scholarly journals Urothelial Carcinoma in a 22-Year-Old Female with Angelman Syndrome

2017 ◽  
Vol 2017 ◽  
pp. 1-3
Author(s):  
Jessica Pugh ◽  
R. Keith Huffaker
Keyword(s):  
Abdominal Pain ◽  
Urothelial Carcinoma ◽  
Angelman Syndrome ◽  
White Female ◽  
Genetic Mutations ◽  
Low Grade ◽  
Gross Hematuria ◽  
Delay In Diagnosis ◽  
History Of

A 22-year-old nulligravid white female with Angelman syndrome was noted to have a 4-month history of premenstrual nausea, vomiting, and abdominal pain. She had an echogenic focus in her bladder noted on ultrasound. She was diagnosed with low grade urothelial carcinoma after cystoscopic evaluation with biopsy and was sent to urology for further treatment. Urothelial carcinoma is rare in individuals younger than age 40. Patients may present with gross hematuria. There is often a delay in diagnosis in younger individuals with different genetic mutations noted upon diagnosis.

scholarly journals Multiple Renal Abscesses due to ESBL Extended-Spectrum Beta-Lactamase-Producing Escherichia coli Causing Acute Pyelonephritis and Bacteremia: A Case Report with a Good Outcome (No Drainage Required)

2016 ◽  
Vol 2016 ◽  
pp. 1-4 ◽  
Author(s):  
Abdalla Khalil ◽  
Musaad Qurash ◽  
Asem Saleh ◽  
Rasha Ali ◽  
Mohamed Elwakil
Keyword(s):  
Ct Scan ◽  
Acute Pyelonephritis ◽  
Urine Culture ◽  
Flank Pain ◽  
Beta Lactamase ◽  
Extended Spectrum Beta Lactamase ◽  
Left Flank ◽  
Extended Spectrum ◽  
Abdomen Ct ◽  
Left Flank Pain

Extended-spectrum beta-lactamase-producing Enterobacteriaceae urinary tract infections are challenging infections with increased mortality, morbidity, and failure of therapy. A 44-year-old Saudi male diabetic patient was seen at the ER of IMC Hospital with features of acute pyelonephritis: fever, burning urine, and left flank pain for three days. He was treated for cystitis at the Endocrine Clinic two weeks prior to his ER visit with nitrofurantoin and levofloxacin orally according to urine culture and sensitivity result. The patient was admitted, received IV meropenem, and continued to be febrile for three days. His urine and blood culture at ER grew the same ESBL-producing E. coli as in his urine culture from the Endocrine Clinic. His abdomen CT scan showed two left renal abscesses at the upper and middle poles. His temperature resolved on the fourth day of IV therapy. Intravenous meropenem was continued for 4 weeks after inserting PICC line and the patient was followed up by home healthcare. He was feeling better with occasional left flank pain and repeated abdomen CT scan showed complete resolution of both renal abscesses.

Maternal Infections Associated with Bad Obstetric Outcome: Toxoplasmosis and Rubella.

2018 ◽  
Vol 1 (3) ◽  
pp. 5-11
Author(s):  
Abdulghani M. Alsamarai ◽  
Hala M. Hassan
Keyword(s):  
Protozoan Parasite ◽  
Low Grade ◽  
Chronic Infections ◽  
Mild Disease ◽  
Geographical Regions ◽  
The Face ◽  
Droplet Transmission ◽  
Infectious Encephalitis ◽  
Post Infectious ◽  
Intracellular Protozoan Parasite

Toxoplasmosis is caused by infection with the obligated intracellular protozoan parasite Toxoplasma gondii. It is one of the most prevalent chronic infections affecting one third of the world's human population. The prevalence of T. gondii infection varies among different geographical regions. The infection is characterized by non-specific signs with the consequent formation of cysts that may stay in latent form in many organs. Primary infection is usually subclinical but in some patient's cervical lymphoadenopathy or ocular disease can be present. Rubella is a mild disease caused by a togavirus. There may be a mild prodromal illness involving a low-grade fever, malaise, coryza and mild conjunctivitis. Lymphadenopathy involving post-auricular and sub-occipital glands may lead to rash. The rash is usually transitory, erythematous and mostly seen behind the ears and on the face and neck. Clinical diagnosis is unreliable as the rash may be fleeting and is not specific to rubella. Rubella is spread by droplet transmission. The incubation period is 14 to 21 days, with the majority of individuals developing a rash 14 to 17 days after exposure. Individuals with rubella are infectious from one week before symptoms appear to four days after the onset of the rash. Complications include thrombocytopaenia (the rate may be as high as one in 3000 infections) and post-infectious encephalitis (one in 6000 cases).

Hemorrhagic Fever in Schoenlein Henoch Purpura.

Blood ◽  
2007 ◽  
Vol 110 (11) ◽  
pp. 3941-3941
Author(s):  
Giorgio Corinaldesi ◽  
Christian Corinaldesi
Keyword(s):  
Abdominal Pain ◽  
Cell Damage ◽  
Leukocytoclastic Vasculitis ◽  
Parvovirus B19 ◽  
Hemorrhagic Fever ◽  
Antineutrophil Cytoplasmic Antibodies ◽  
Direct Immunofluorescence ◽  
Low Grade ◽  
Inflammatory Disorder ◽  
Streptococcus Group

Abstract Schoenlein Henoch Purpura (SHP) may show a cohort of symptoms such as: a palpable purpuric rash where lesions may be prominent mostly on lower extremities, abdominal pain, arthritis and nephritis. It is an inflammatory disorder characterized by an IgA-mediated vasculitis with immune complexes deposition in smaller veins, capillaries and arterioles which often occur as a response to infections (mycoplasma, streptococcus group A, Campylobacter enteritis, Helycobacter, Herpes virus, Parvovirus B19, Epstein-Barr virus), or to an exposure to allergens, drugs, some particular food, cold, or even insect bites. Symptoms are often dramatic at the onset, and the exitus occurs sadly between 30 – 50% of the patients, despite the therapeutic support, for this reason the diagnosis and the therapy should be precocious. Clinically we can observe multi organ failure, hypotension, flushing, capillary leak haemorrhage secondary to thrombocytopenia, platelet dysfunction, disseminated intravascular coagulation, shock. We have studied a young boy (19 years old) with an urticarial wheals, erythematous maculo-papules, purple spots, and larger petechiae, migrant arthritis and arthralgia, renal disease (low grade hematuria with proteinuria), weakness, necrotic and hemorrhagic plaques, with haematemesis, diarrhoea, abdominal pain, fever (38.5°/39.5° C), and increased blood level of human Parvovirus-B19 NS1-gene component. We performed a skin biopsy of SHP that showed: leukocytoclastic vasculitis, with angiocentric neutrophilic infiltrate, fibrin deposition and focal fibrinoid necrosis. We also evaluated a kidney biopsy which showed: mild increase of mesangial cellularity, with segmental capillary luminal leukocytes and scattered capillary wall fuchsinophilic deposit, while the diagnostic finding is constituted by granular mesangial IgA with C3. The management is based on support therapy, with plenty of haemotrasfusions, and immunosuppressive therapy (prednisone 40 mg os/die, and cyclophosphamide100–200 mg/daily/os for 30–60 days (may decrease inflammation, capillary permeability and suppressing granulocytes activity and migration. The prognosis is usually excellent when the syndrome is self-limited and resolves spontaneously often 4/8 weeks without recurrence. Sometimes the diagnostic investigation may show an elevated level of vascular IgA deposition (direct immunofluorescence), elevated plasma levels of C3 and C4, elevated levels of IgA, ANCA (antineutrophil cytoplasmic antibodies: pANCA anti-MPO, cANCA anti-PR3), CRP, fibrinogen, ICAM-1, and increase of TNF, IL-1, IL-6, TGF-beta that reflect endothelial cell damage and dysfunction.

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