scholarly journals Brain auditory evoked response test, the standard method for the diagnosis of the hereditary deafness in dogs

2021 ◽  
Vol 26 (2) ◽  
pp. 24-28
Author(s):  
Nicolae Coldea
Keyword(s):  
Hearing Loss ◽  
Standard Method ◽  
Short Communication ◽  
Evoked Response ◽  
Hearing Impairments ◽  
Hereditary Deafness ◽  
Response Test ◽  
Auditory Evoked Response ◽  
Life Threatening

Hearing deficiency is one of the most common hearing impairments that affect humans and other mammalian alike. Hearing loss is not painful or a life-threatening change but can endanger the patient by taking into account a large number of breeds predisposed to hereditary deafness, this short communication aims to synthesize the steps and the method for BAER test. For the affected breeds, the BAER test is recommended starting at the age of two months

Unilateral Deafness Diagnosed using the Brainstem Auditory Evoked Response Test in a Shih-tzu Dog

2018 ◽  
Vol 35 (3) ◽  
pp. 103-106
Author(s):  
Na-Ni Ji ◽  
Joong-Hyun Song ◽  
Tae-Sung Hwang ◽  
Hee-Chun Lee ◽  
Do-Hyeon Yu ◽  
...  
Keyword(s):  
Evoked Response ◽  
Unilateral Deafness ◽  
Response Test ◽  
Auditory Evoked Response ◽  
Brainstem Auditory Evoked Response

Brain Stem Auditory Evoked Response

1979 ◽  
Vol 88 (4_suppl) ◽  
pp. 11-21 ◽  
Author(s):  
Kevin T. Kavanagh ◽  
James V. Beardsley
Keyword(s):  
Hearing Loss ◽  
Brain Stem ◽  
Normal Wave ◽  
Sound Intensity ◽  
Evoked Response ◽  
Sound Stimulation ◽  
Auditory Evoked Response ◽  
Occult Lesion ◽  
Intensity Functions ◽  
Wave Latency

This paper studies the effect of otologic disease on the brain stem auditory evoked response (BAER). Both conductive and neurosensory hearing losses are analyzed by plotting wave V latencies and amplitudes as a function of sound intensity. It was found that the BAER is elicited primarily by frequencies greater than 2000 Hz. Conductive hearing losses produce a latency intensity function which approaches the norm with high decibel stimulation. Neurosensory losses produce a variety of latency intensity functions. In determining the degree of hearing loss, wave threshold is found to be the best index. Wave latency at high decibels is found to have little correspondence to degree of neurosensory loss; wave amplitude is highly variable among subjects but still a useful indicator for detecting pathology. Between-ear comparisons of wave latencies elicited by high decibel sound stimulation suggest that unilateral nonrecruiting or partially recruiting hearing losses will result in a latency difference. This method can be used to detect unilateral acoustic neuromas, and the false positives found by this technique are probably caused by other unilateral nonrecruiting hearing losses. In evaluating neurological disease, and especially when testing for a second occult lesion in multiple sclerosis (MS) an audiogram should be obtained because the criterion of a normal wave latency with decreased amplitude for the diagnosis of MS can be mimicked by peripheral hearing loss.

scholarly journals Occupational Noise: Auditory and Non-Auditory Consequences

2020 ◽  
Vol 17 (23) ◽  
pp. 8963
Author(s):  
Adam Sheppard ◽  
Massimo Ralli ◽  
Antonio Gilardi ◽  
Richard Salvi
Keyword(s):  
Hearing Loss ◽  
Noise Exposure ◽  
Significant Risk ◽  
Significant Risk Factor ◽  
The United States ◽  
Occupational Noise ◽  
Hearing Impairments ◽  
True Value ◽  
Life Threatening ◽  
Limited Frequency

Occupational noise exposure accounts for approximately 16% of all disabling hearing losses, but the true value and societal costs may be grossly underestimated because current regulations only identify hearing impairments in the workplace if exposures result in audiometric threshold shifts within a limited frequency region. Research over the past several decades indicates that occupational noise exposures can cause other serious auditory deficits such as tinnitus, hyperacusis, extended high-frequency hearing loss, and poor speech perception in noise. Beyond the audiogram, there is growing awareness that hearing loss is a significant risk factor for other debilitating and potentially life-threatening disorders such as cardiovascular disease and dementia. This review discusses some of the shortcomings and limitations of current noise regulations in the United States and Europe.

The Onset–Offset N1–P2 Auditory Evoked Response in Individuals With High-Frequency Sensorineural Hearing Loss: Responses to Broadband Noise

2021 ◽  
pp. 1-10
Author(s):  
Jennifer E. Gonzalez ◽  
Frank E. Musiek
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Stimulus Duration ◽  
High Frequency ◽  
Repeated Measures ◽  
Normal Hearing ◽  
Broadband Noise ◽  
Evoked Response ◽  
Sensorineural Hearing ◽  
Auditory Evoked Response

Purpose Clinical use of electrophysiologic measures has been limited to use of brief stimuli to evoke responses. While brief stimuli elicit onset responses in individuals with normal hearing and normal central auditory nervous system (CANS) function, responses represent the integrity of a fraction of the mainly excitatory central auditory neurons. Longer stimuli could provide information regarding excitatory and inhibitory CANS function. Our goal was to measure the onset–offset N1–P2 auditory evoked response in subjects with normal hearing and subjects with moderate high-frequency sensorineural hearing loss (HFSNHL) to determine whether the response can be measured in individuals with moderate HFSNHL and, if so, whether waveform components differ between participant groups. Method Waveforms were obtained from 10 participants with normal hearing and seven participants with HFSNHL aged 40–67 years using 2,000-ms broadband noise stimuli with 40-ms rise–fall times presented at 50 dB SL referenced to stimulus threshold. Amplitudes and latencies were analyzed via repeated-measures analysis of variance (ANOVA). N1 and P2 onset latencies were compared to offset counterparts via repeated-measures ANOVA after subtracting 2,000 ms from the offset latencies to account for stimulus duration. Offset-to-onset trough-to-peak amplitude ratios between groups were compared using a one-way ANOVA. Results Responses were evoked from all participants. There were no differences between participant groups for the waveform components measured. Response × Participant Group interactions were not significant. Offset N1–P2 latencies were significantly shorter than onset counterparts after adjusting for stimulus duration (normal hearing: 43 ms shorter; HFSNHL: 47 ms shorter). Conclusions Onset–offset N1–P2 responses were resistant to moderate HFSNHL. It is likely that the onset was elicited by the presentation of a sound in silence and the offset by the change in stimulus envelope from plateau to fall, suggesting an excitatory onset response and an inhibitory-influenced offset response. Results indicated this protocol can be used to investigate CANS function in individuals with moderate HFSNHL. Supplemental Material https://doi.org/10.23641/asha.14669007

scholarly journals ASFIKSIA PERINATAL SEBAGAI FAKTOR RESIKO GANGGUAN PENDENGARAN PADA ANAK

2012 ◽  
Vol 36 (1) ◽  
pp. 1
Author(s):  
Sukri Rahman ◽  
Hanifatryevi Hanifatryevi
Keyword(s):  
Hearing Loss ◽  
Early Detection ◽  
Tissue Damage ◽  
Otoacoustic Emissions ◽  
Perinatal Asphyxia ◽  
Body Part ◽  
The Body ◽  
Evoked Response ◽  
Auditory Evoked Response ◽  
The Third

AbstrakAsfiksia adalah keadaan di mana tubuh atau bagian tubuh kekurangan oksigen. Jika kondisi ini terjadi pada bayi baru lahir disebut dengan asfiksia perinatal, yang dapat menimbulkan kerusakan jaringan secara permanen maupun bersifat sementara, salah satunya adalah gangguan pendengaran sensorineural.Makalah ini akan membahas tentang mekanisme gangguan pendengaran akibat asfiksia, deteksi dini gangguan pendengaran dengan pemeriksaan Brain Evoked Response Auditory (BERA) dan Otoacoustic Emissions (OAE).Gangguan pendengaran akibat asfiksia dapat terjadi akibat beberapa mekanisme. Kelainan ini terlihat pada gambaran BERA dan OAE, dimana terjadinya peningkatan masalaten dan interval gelombang yang bersifat sementara atau menetap, serta ditemukan gangguan pada fungsi koklea bayi dengan asfiksia perinatal terutama pada frekuensi 1-5 kHz yang terdeteksi pada hari ke tiga sampai hari kelima kelahiran, dan terdapat perbaikan pada usia 1 bulan.Kata Kunci : Asfiksia perinatal, gangguan pendengaran, BERA, OAEAbstractAsphyxia is a condition in which the body or body part to lack of oxygen. If this condition occurs in newborns with perinatal asphyxia is also called, which can cause tissue damage is permanent or temporary. One of that is a sensorineural hearing loss.This paper will discuss the mechanisms of hearing loss due to asphyxia, early detection of hearing loss with an examination Brain Auditory Evoked Response (BERA) and Otoacoustic Emissions (OAE).Hearing loss due to asphyxia can occur due to several mechanisms. These abnormalities are seen on the results of BERA and OAE, where an increase in latent period and the interval waves that are temporary or permanent, and found interference with the function of the cochlea infants with perinatal asphyxia, especially at frequency of 1-5 kHz were detected on the third day until the fifth day of birth, and there are improvements to the age of 1 month.Key word : Perinatal asphyxia, hearing loss, Brain Auditory Evoked Response, Otoacoustic Emissions

scholarly journals Bone-conducted brainstem auditory evoked response in a dog with total bilateral ear canal ablation: a case report

2010 ◽  
Vol 55 (No. 1) ◽  
pp. 39-41 ◽  
Author(s):  
A. Pomianowski ◽  
Z. Adamiak
Keyword(s):  
Case Report ◽  
Bone Conduction ◽  
Evoked Response ◽  
Ear Canal ◽  
Surgical Ablation ◽  
Response Test ◽  
Auditory Evoked Response ◽  
Hearing Function ◽  
Brainstem Auditory Evoked Response

The bone-conducted brainstem auditory evoked response test was performed in an eight-year-old dog with total bilateral ear canal ablation. It was found that 12 months after bilateral surgical ablation of the external acoustic meatus the dog still maintained its hearing function due to the mechanism of acoustic bone conduction.

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