scholarly journals Validity of coronary heart diseases and heart failure based on hospital discharge and mortality data in the Netherlands using the cardiovascular registry Maastricht cohort study

2009 ◽  
Vol 24 (5) ◽  
pp. 237-247 ◽  
Author(s):  
Audrey H. H. Merry ◽  
Jolanda M. A. Boer ◽  
Leo J. Schouten ◽  
Edith J. M. Feskens ◽  
W. M. Monique Verschuren ◽  
...  
2019 ◽  
Vol 35 (5) ◽  
Author(s):  
Ana Luiza Bierrenbach ◽  
Gizelton Pereira Alencar ◽  
Cátia Martinez ◽  
Maria de Fátima Marinho de Souza ◽  
Gabriela Moreira Policena ◽  
...  

Heart failure is considered a garbage code when assigned as the underlying cause of death. Reassigning garbage codes to plausible causes reduces bias and increases comparability of mortality data. Two redistribution methods were applied to Brazilian data, from 2008 to 2012, for decedents aged 55 years and older. In the multiple causes of death method, heart failure deaths were redistributed based on the proportion of underlying causes found in matched deaths that had heart failure listed as an intermediate cause. In the hospitalization data method, heart failure deaths were redistributed based on data from the decedents’ corresponding hospitalization record. There were 123,269 (3.7%) heart failure deaths. The method with multiple causes of death redistributed 25.3% to hypertensive heart and kidney diseases, 22.6% to coronary heart diseases and 9.6% to diabetes. The total of 41,324 heart failure deaths were linked to hospitalization records. Heart failure was listed as the principal diagnosis in 45.8% of the corresponding hospitalization records. For those, no redistribution occurred. For the remaining ones, the hospitalization data method redistributed 21.2% to a group with other (non-cardiac) diseases, 6.5% to lower respiratory infections and 9.3% to other garbage codes. Heart failure is a frequently used garbage code in Brazil. We used two redistribution methods, which were straightforwardly applied but led to different results. These methods need to be validated, which can be done in the wake of a recent national study that will investigate a big sample of hospital deaths with garbage codes listed as underlying causes.


2015 ◽  
Vol 21 (10) ◽  
pp. S148
Author(s):  
Yohei Shiotani ◽  
Masakazu Saitoh ◽  
Junko Sakamoto ◽  
Hidetoshi Suzuki ◽  
Mariko Nakazawa ◽  
...  

2014 ◽  
Vol 4 (1) ◽  
pp. 63-72
Author(s):  
Antonio Totaro ◽  
Michele Correale ◽  
Matteo Biase ◽  
Natale Brunetti

2000 ◽  
Vol 2 ◽  
pp. 78-78
Author(s):  
A. Campeanu ◽  
R.O. Radu Olinescu ◽  
M.V. Mihaela Vasile ◽  
O.Z. Ondin Zaharia ◽  
N.C. Nicoara Campeanu

2021 ◽  
Vol 28 (Supplement_1) ◽  
Author(s):  
BMA Van Bakel ◽  
EA Bakker ◽  
F De Vries ◽  
DHJ Thijssen ◽  
TMH Eijsvogels

Abstract Funding Acknowledgements Type of funding sources: Foundation. Main funding source(s): Dutch Heart Foundation - senior E-Dekker grant Physical activity and sedentary behaviour in cardiovascular disease patients during the COVID-19 lockdown in the Netherlands; a longitudinal cohort study Background Previous studies showed that the COVID-19 lockdown caused a more inactive lifestyle, but it is unknown whether these acute effects persist over time. We prospectively evaluated changes in physical activity and sedentary behaviour among chronic cardiovascular disease (CVD) patients during the first-wave COVID-19 lockdown and aimed to identify factors associated with physical inactivity. Methods 1,565 CVD patients were included and baseline physical activity and sedentary behaviour were assessed using validated questionnaires at 5 weeks after the initiation of the Dutch lockdown (March 2020). Follow-up measures were collected every subsequent 4 weeks until July 2020. Multivariate mixed model analyses were performed to identify whether age, gender, CVD subtype, lockdown adherence and mental health factors impacted changes in physical (in)activity. Results Patients were 67 (interquartile range [60, 73]) years, mostly male (73%) and primarily diagnosed with myocardial infarction (48%) or angina pectoris (18%). Daily time spent in moderate-to-vigorous physical activity was 143 minutes (95% confidence interval (CI) 137; 148) at baseline, with almost no changes during follow-up on a group level (Δ+8.0 (95%CI -1.1; 17.0); Δ+11.2 (95%CI 1.9; 20.5) and Δ+8.0 (95%CI -1.5; 17.5) min/day after 4, 8 and 12 weeks, respectively).Female gender (Δ-40.7 (95%CI -48.5; -33.0) min/day); heart failure (Δ-23.0 (95%CI -36.5; -9.5) min/day); fear of a COVID-19 infection (Δ-6.6 (95%CI -9.4; -3.8) min/day) and limited possibilities for physical activity (Δ-7.4 (95%CI -10.1; -4.7) min/day) were independently associated with a decrease in physical activity. Sedentary time was 567 (95%CI 555; 578) min/day at baseline which did not change after 4 weeks (Δ+12.1 (95%CI -6.0; 30.2) min/day) and after 8 weeks (Δ+15.2 (95%CI -3.3; 33.8) min/day), but significantly increased after 12 weeks of follow-up (Δ+19.0 (95%CI 0.1; 37.8) min/day). Lack of social contact (Δ+8.4 (95%CI 2.3; 14.5); limited possibilities for physical activity (Δ+14.7 (95%CI 8.8; 20.5) and younger age (Δ+2.1 (95%CI 1.3; 2.8) min/day were independently associated with an increase in sedentary time. Conclusions A time-dependent increase in daily sedentary time was observed among chronic CVD patients during the COVID-19 lockdown, whereas physical activity levels did not substantially change. Our findings highlight the need to develop and implement novel solutions to increase physical activity and reduce sedentary time during (and beyond) the COVID-19 pandemic, especially in CVD patients who are female, younger, diagnosed with heart failure, have a lack of social contact, fear of COVID-19 infection and experience limited physical activity possibilities during the lockdown.


BMJ ◽  
2019 ◽  
pp. l223 ◽  
Author(s):  
Clare J Taylor ◽  
José M Ordóñez-Mena ◽  
Andrea K Roalfe ◽  
Sarah Lay-Flurrie ◽  
Nicholas R Jones ◽  
...  

Abstract Objectives To report reliable estimates of short term and long term survival rates for people with a diagnosis of heart failure and to assess trends over time by year of diagnosis, hospital admission, and socioeconomic group. Design Population based cohort study. Setting Primary care, United Kingdom. Participants Primary care data for 55 959 patients aged 45 and over with a new diagnosis of heart failure and 278 679 age and sex matched controls in the Clinical Practice Research Datalink from 1 January 2000 to 31 December 2017 and linked to inpatient Hospital Episode Statistics and Office for National Statistics mortality data. Main outcome measures Survival rates at one, five, and 10 years and cause of death for people with and without heart failure; and temporal trends in survival by year of diagnosis, hospital admission, and socioeconomic group. Results Overall, one, five, and 10 year survival rates increased by 6.6% (from 74.2% in 2000 to 80.8% in 2016), 7.2% (from 41.0% in 2000 to 48.2% in 2012), and 6.4% (from 19.8% in 2000 to 26.2% in 2007), respectively. There were 30 906 deaths in the heart failure group over the study period. Heart failure was listed on the death certificate in 13 093 (42.4%) of these patients, and in 2237 (7.2%) it was the primary cause of death. Improvement in survival was greater for patients not requiring admission to hospital around the time of diagnosis (median difference 2.4 years; 5.3 v 2.9 years, P<0.001). There was a deprivation gap in median survival of 0.5 years between people who were least deprived and those who were most deprived (4.6 v 4.1 years, P<0.001). Conclusions Survival after a diagnosis of heart failure has shown only modest improvement in the 21st century and lags behind other serious conditions, such as cancer. New strategies to achieve timely diagnosis and treatment initiation in primary care for all socioeconomic groups should be a priority for future research and policy.


2013 ◽  
Vol 2 (2) ◽  
Author(s):  
Novita Paliliewu ◽  
Reginald L. Lefrandt

Abstract: Cardiorenal syndrome can be generally defined as a pathophysiological disorder of the heart and kidneys, whereby acute or chronic dysfunction of one organ may induce acute or chronic dysfunction of the other ones. In the end stage renal disease, the prevalences of left ventricular hypertrophy and coronary heart diseases are high enough. On the other hand, patients with moderate congestive heart failure show low glomerular filtration rates. There is no consistent and effective strategy for the management of cardiorenal patients. Most approaches are empirical and include: recognizing the cardiorenal syndrome and anticipating the development of worsening renal function and/or diuretic resistance, optimizing heart failure therapy, evaluating renal structure and function, and optimizing diuretic dosaging and renal specific therapy. Investigational therapies such as vasopressin antagonist and adenosine antagonist are still being developed. Keywords: cardiorenal syndrome, organ dysfunction, therapy.   ABSTRAK: Sindrom kardiorenal secara umum dapat didefinisikan sebagai keadaan gangguan patofisiologi jantung dan ginjal, dimana terjadi disfungsi akut atau kronis salah satu organ yang mengakibatkan disfungsi akut atau kronis organ lainnya. Pada penyakit ginjal tahap akhir prevalensi hipertrofi ventrikel kiri dan penyakit jantung koroner cukup tinggi. Demikian pula halnya dengan pasien-pasien gagal jantung sedang memiliki gangguan laju filtrasi glomerulus (LFG). Sampai saat ini belum terdapat strategi yang konsisten dan efektif  dalam penanganan pasien sindrom kardiorenal. Umumnya dilakukan pendekatan secara empirik yaitu: deteksi sindrom kardiorenal dan mengantisipasi timbulnya perburukan fungsi ginjal dan atau resistensi diuretik, optimalisasi pengobatan gagal jantung, mengevaluasi struktur dan fungsi ginjal, optimalisasi dosis diuretik serta terapi khusus untuk ginjal. Penggunaan antagonis vasopresin dan antagonis adenosin untuk sindrom kardiorenal masih sedang dalam tahap penelitian. Kata kunci: sindrom kardiorenal, disfungsi organ, pengobatan.


2010 ◽  
Vol 104 (8) ◽  
pp. 1212-1221 ◽  
Author(s):  
Lina J. Leurs ◽  
Leo J. Schouten ◽  
R. Alexandra Goldbohm ◽  
Piet A. van den Brandt

Chronic mild dehydration has been associated with several diseases, including fatal IHD and stroke. It has been suggested that hydration through total fluid intake (or water) is inversely associated with IHD or stroke mortality. The objective of the present study was to evaluate the relationship between total fluid (and specific beverage) intake and IHD or stroke mortality in the Netherlands Cohort Study (NLCS). In 1986, 120 852 participants aged 55–69 years were enrolled into the NLCS. Mortality data were collected over a 10-year follow-up period. Analysis was done through a case–cohort approach, and it was based on the subjects without a history of heart disease, stroke or diabetes at baseline. A total of 1789 IHD mortality cases and 708 stroke mortality cases occurred during the follow-up. Higher total fluid consumption was not associated with either IHD mortality or stroke mortality in men or women. When analysing specific beverages, a positive association between coffee consumption (increment 270 ml/d) and IHD mortality was observed in men (hazard ratio (HR) 1·09, 95 % CI 1·00, 1·18), while an inverse relationship was observed in women (HR: 0·88, 95 % CI 0·78, 1·00). For tea consumption (increment of 253 ml/d), an inverse relationship with IHD mortality was observed in men (HR: 0·91, 95 % CI 0·83, 1·00). No association with water intake was observed. In the study population, fresh water consumption was very low. In conclusion, total fluid intake was not associated with IHD or stroke mortality in either men or women. Coffee consumption was inversely associated with IHD mortality in women only, while a higher tea intake was associated with lower IHD mortality in men only.


Sign in / Sign up

Export Citation Format

Share Document