scholarly journals Report on Pigeonpea Sterility Mosaic Virus (PSMV) Disease Incidence in CO (Rg) 8 in Tamil Nadu

2020 ◽  
Vol 9 (11) ◽  
pp. 1112-1115
Author(s):  
K. Kalaichelvi
Keyword(s):  
Mosaic Virus ◽  
Tamil Nadu ◽  
Disease Incidence ◽  
Pigeonpea Sterility Mosaic Virus

Pigeonpea sterility mosaic virus. [Distribution map].

2007 ◽  
Author(s):  
Keyword(s):  
Mosaic Virus ◽  
Tamil Nadu ◽  
Andhra Pradesh ◽  
West Bengal ◽  
Uttar Pradesh ◽  
Pigeon Pea ◽  
Distribution Map ◽  
Pigeonpea Sterility Mosaic Virus ◽  
Virus Distribution ◽  
New Distribution

Abstract A new distribution map is provided for Pigeonpea sterility mosaic virus. Unassigned virus. Hosts: pigeon pea (Cajanus cajan). Information is given on the geographical distribution in Asia (Bangladesh, India (Andhra Pradesh, Bihar, Chhattisgarh, Gujarat, Karnataka, Maharashtra, Punjab, Tamil Nadu, Uttar Pradesh, West Bengal), Myanmar, Nepal, Sri Lanka). It is vectored by Aceria cajani (Acari: Eriophyidae).

Inheritance Pattern of Mungbean Yellow Mosaic Virus (MYMV) Disease Resistance in Blackgram [Vigna mungo (L.) Hepper]

2021 ◽  
Author(s):  
K. Vadivel ◽  
N. Manivannan ◽  
A. Mahalingam ◽  
V.K. Satya ◽  
S. Ragul
Keyword(s):  
Disease Resistance ◽  
Mosaic Virus ◽  
Tamil Nadu ◽  
Gene Action ◽  
Disease Incidence ◽  
Yellow Mosaic Virus ◽  
Inheritance Pattern ◽  
Complementary Gene ◽  
Mungbean Yellow Mosaic Virus ◽  
Complementary Gene Action

Background: Mungbean yellow mosaic virus (MYMV) disease is the most destructive disease in blackgram. Development of MYMV resistant varieties is one of the best possible solutions to avoid the yield reduction in blackgram. There are conflicting reports on the genetics of resistance to MYMV disease claiming that it is controlled by both dominant and recessive genes. Hence the present study was aimed to understand the inheritance pattern of the MYMV disease resistance in eight crosses of blackgram.Methods: Parents, F1 and F2 generation of eight cross combinations were raised during July - Sep, 2018 at National Pulses Research Centre, Tamil Nadu Agricultural University, Vamban, Tamil Nadu. An infector row of CO 5 was raised to intensify the MYMV disease pressure after every eight rows. Based on disease incidence on 60th day after sowing, two phenotypic classes were formed among F2 plants with the scales of (1 to 3) as resistant phenotype and (4 to 9) as susceptible phenotype. The goodness of fit to Mendelian segregation ratio for MYMV disease resistance in the segregating population was tested by Chi square test (Stansfield, 1991).Result: The MYMV disease incidence was tri-genically controlled with inhibitory gene action in four crosses viz., MDU 1 x Mash 114, CO5 x Mash 114, MDU 1 x VBN 6 and CO 5 x VBN 6. Complementary gene action with two genes was observed in four crosses viz., MDU 1 x Mash 1008, CO 5 x Mash 1008, MDU 1 x VBN 8 and CO 5 x VBN 8. Differences in number of genes were observed due to the presence of recessive inhibitory gene in both male and female parents of the crosses which had complementary gene action for MYMV disease. The putative gene symbols assigned for the six genotypes viz., S1S1S2S2ii (MDU 1 and CO 5), s1s1s2s2II (Mash 114 and VBN 6) and s1s1s2s2ii (Mash 1008 and VBN 8), respectively. 

scholarly journals Development of high yielding with durable resistance against Mungbean yellow mosaic virus genotypes in Blackgram

2019 ◽  
pp. 187-193
Author(s):  
Pandiyan M ◽  
Senthil N ◽  
Krishnaveni A ◽  
Sivakumar C ◽  
Singh BB ◽  
...  
Keyword(s):  
Powdery Mildew ◽  
Mosaic Virus ◽  
Tamil Nadu ◽  
Durable Resistance ◽  
Yellow Mosaic Virus ◽  
Yield Potential ◽  
Average Height ◽  
Crude Fibre ◽  
Mungbean Yellow Mosaic Virus ◽  
Virus Genotypes

The Blackgram culture VBG04-008 is a cross derivative of blackgram Vamban 3 x Vigna mungo var. silvestris 8 is released as TNAU blackgram VBN (Bg) 7 maturing in 65-70 days with an average height of 17 cm and suited for cultivation under both under rainfed and irrigated conditions. It has a yield potential of 981 Kg per hectare. This culture is resistant to Yellow Mosaic Virus, Powdery mildew and Leaf Curl Virus and less damage of pod borer. It possesses desirable characters like high protein content (21.05%), crude fibre (5.90g/100g) and iron (3.76 mg/100g). Grains are medium sized with black in colour. It is recommended for cultivation in Tamil Nadu, Andhra Pradesh, Karnataka and Orissa. Keywords: VBG04-008; Blackgram; VBN 7 Mung Bean Yellow Mosaic Virus; Powdery mildew-Rainfed; Irrigated

scholarly journals KETAHANAN BEBERAPA KULTIVAR CABAI TERHADAP CUCUMBER MOSAIC VIRUS DAN CHILLI VEINAL MOTTLE VIRUS

2012 ◽  
Vol 7 (2) ◽  
pp. 130-139
Author(s):  
Muhammad Taufik ◽  
Sri Hendrastuti Hidayat ◽  
Sriani Sujiprihati ◽  
Gede Suastika ◽  
Sientje Mandang Sumaraw
Keyword(s):  
Virus Infection ◽  
Cucumber Mosaic Virus ◽  
Mosaic Virus ◽  
Yield Loss ◽  
Disease Incidence ◽  
Growth And Yield ◽  
Mottle Virus ◽  
Tolerance Response ◽  
Chilli Veinal Mottle Virus ◽  
Das Elisa

Resistance Evaluation of Chillipepper Cultivars for Cucumber Mosaic Virus and Chilli Veinal Mottle Virus.  The use of resistance culivars is an important strategy for management of virus infection in chillipepper. A research was undergone to study the effect of single and mix infection of CMV and ChiVMV on the disease incidence and on the growth and yield of nine chillipepper cultivars, i.e. Cilibangi 4, Cilibangi 5, Cilibangi 6, Helem, Jatilaba, Tit Bulat, Tit Segitiga, Tit Super and Tampar. Mechanical inoculation was conducted to transmit the virus. Infection of the virus was then confirmed with DAS-ELISA.  In general, inoculated chillipepper cultivars developed similar symptoms, i.e. mosaic type for CMV and mottle type for ChiVMV.  More severe symptom was not always observed from mix infection of CMV and ChiVMV. Disease incidence occurred in the range of 16.67 – 86.0% and this caused 18.3 – 98.6% yield loss.  Based on symptom expression, ELISA result, and reduction on yield, it can be concluded that all chillipepper cultivars used in this study could not hold up the virus infection. However, several cultivars showed tolerance response :  Jatilaba, Tit Super, and Tampar for CMV; Cilibangi 4 for ChiVMV; Tit Super for mix infection; and Cilibangi 5 for CMV, ChiVMV, and mix infection.  Further evaluation and investigation involving different chillipepper cultivars should be conducted.

scholarly journals Evaluation and Characterization of High Yielding Cassava Mosaic Resistant Variety YTP2

2021 ◽  
pp. 198-208
Author(s):  
L. Pugalendhi ◽  
M. Velmurugan ◽  
P. S. Kavitha ◽  
M. K. Kalarani ◽  
N. Senthil ◽  
...  
Keyword(s):  
Tamil Nadu ◽  
Starch Content ◽  
Disease Incidence ◽  
Mosaic Disease ◽  
Dna Fingerprint ◽  
Cassava Mosaic Disease ◽  
Resistant Variety ◽  
Research Station ◽  
Internodal Length ◽  
New Variety

The cassava variety YTP2 (Me 681) has been developed through selection from Thondamuthur type at Tapioca and Castor Research Station, TNAU, Yethapur. The performance of YTP2 in the Adaptive Research Trial (ART) and On Farm Trial (OFT) in the farmer’s field inferred that this new variety is well adapted to cassava growing districts of Tamil Nadu. In addition to the above, YTP2 was found to be resistant to cassava mosaic disease incidence (CMD). Plants are erect, medium growing and non-branching type and suitable for growing under irrigated and rainfed conditions. The internodal length is shorter and the leaf size is medium with sufficient canopy. The leaves of the plants droop down to reduce the transpiration loss which is more advantageous to overcome or escape from drought and heat stress during summer season. It is a dual purpose variety wherein the tubers contain high starch content which is much favourable for the manufacture of starch, sago and also suited for table purpose. The overall performance of this variety showed higher tuber yield (42.20 t ha-1) and starch content (28.40%) which is 15.94% and 18.20% increase over the check varieties YTP1 and H226 respectively. The results of DNA fingerprint data involving SSR markers (SSRY235, NS169 and NS928) showed that it is genetically distinct from the existing commercial varieties viz., YTP1, H226 and Sree Athulya.

scholarly journals First Report of Cucumber mosaic virus Subgroup II Infecting Lycopersicon esculentum in India

Plant Disease ◽  
2006 ◽  
Vol 90 (11) ◽  
pp. 1457-1457 ◽  
Author(s):  
N. Sudhakar ◽  
D. Nagendra-Prasad ◽  
N. Mohan ◽  
K. Murugesan
Keyword(s):  
Coat Protein ◽  
Cucumber Mosaic Virus ◽  
Mosaic Virus ◽  
Tamil Nadu ◽  
Indian Subcontinent ◽  
Enzyme Linked Immunosorbent Assay ◽  
Infected Leaf ◽  
Polymorphism Analysis ◽  
First Report ◽  
Subgroup Ii

During a survey in January 2006 near Salem in Tamil Nadu (south India), Cucumber mosaic virus was observed infecting tomatoes with an incidence of more than 70%. Plants exhibiting severe mosaic, leaf puckering, and stunted growth were collected, and the virus was identified using diagnostic hosts, evaluation of physical properties of the virus, compound enzyme-linked immunosorbent assay (ELISA) (ELISA Lab, Washington State University, Prosser), reverse-transcription polymerase chain reaction (RT-PCR), and restriction fragment length polymorphism analysis (DSMZ, S. Winter, Germany). To determine the specific CMV subgroup, total RNA was extracted from 50 infected leaf samples using the RNeasy plant RNA isolation kit (Qiagen, Hilden, Germany) and tested for the presence of the complete CMV coat protein gene using specific primers as described by Rizos et al. (1). A fragment of the coat protein was amplified and subsequently digested with MspI to reveal a pattern of two fragments (336 and 538 bp), indicating CMV subgroup II. No evidence of mixed infection with CMV subgroup I was obtained when CMV isolates representing subgroups I (PV-0419) and II (PV-0420), available at the DSMZ Plant Virus Collection, were used as controls. Only CMV subgroup I has been found to predominantly infect tomato in the Indian subcontinent, although Verma et al. (2) identified CMV subgroup II infecting Pelargonium spp., an ornamental plant. To our knowledge, this is the first report of CMV subgroup II infecting tomato crops in India. References: (1) H. Rizos et al. J. Gen. Virol. 73:2099, 1992. (2) N. Verma et al. J. Biol. Sci. 31:47, 2006.

Cucumber mosaic virus. [Distribution map].

2002 ◽  
Author(s):  
Keyword(s):  
South Carolina ◽  
Cucumber Mosaic Virus ◽  
Mosaic Virus ◽  
Tamil Nadu ◽  
Russian Far East ◽  
Far East ◽  
South Australia ◽  
French Polynesia ◽  
Peninsular Malaysia ◽  
Distribution Map

Abstract A new distribution map is provided for Cucumber mosaic virus Viruses: Bromoviridae: Cucumovirus Hosts: mainly Cucurbitaceae, Solanaceae and Araceae. Information is given on the geographical distribution in EUROPE, Austria, Belarus, Belgium, Bosnia-Herzegovina, Bulgaria, Croatia, Cyprus, Czech Republic, Denmark, Estonia, Finland, France, Germany, Greece, Hungary, Ireland, Italy, Latvia, Lithuania, Macedonia, Malta, Moldova, Netherlands, Poland, Portugal, Romania, Central Russia Russian Far East, Northern Russia, Southern Russia, Slovakia, Slovenia, Spain, Sweden, Switzerland, UK, Ukraine, Yugoslavia (Fed. Rep.), ASIA, Afghanistan, Bangladesh, China, Anhui, Chongqing, Fujian, Gansu, Guangdong, Guangxi, Guizhou, Hebei, Heilongjiang, Henan, Hong Kong, Hubei, Hunan, Jiangsu, Jiangxi, Jilin, Liaoning, Nei, Menggu, Qinghai, Shaanxi, Shandong, Shanxi, Sichuan, Xinjiang, Yunnan, Zhejiang, Republic of Georgia, India, Andhra Pradesh, Bihar, Delhi, Gujarat, Haryana, Himachal Pradesh, Karnataka, Kerala, Madhya Pradesh, Maharashtra, Orissa, Punjab, Rajasthan, Tamil Nadu, Uttar Pradesh, West Bengal, Indonesia, Java, Iran, Iraq, Israel, Japan, Hokkaido, Honshu, Kyushu, Ryukyu Archipelago, Shikoku, Jordan, Kazakhstan, Korea Republic, Kuwait, Kyrgyzstan, Lebanon, Malaysia, Peninsular Malaysia, Sabah, Sarawak, Nepal, Oman, Pakistan, Philippines, Saudi Arabia, Singapore, Sri Lanka, Syria, Taiwan, Tajikistan, Thailand, Turkey, Uzbekistan, Vietnam, Yemen, AFRICA, Algeria, Cameroon, Cote d'Ivoire, Egypt, Ethiopia, Ghana, Kenya, Mauritius, Morocco, Nigeria, Reunion, Sierra Leone, South Africa, Sudan, Tanzania, Togo, Tunisia, Zambia, Zimbabwe, NORTH AMERICA, Canada, British Columbia, Ontario, Quebec, Mexico, USA, Alabama, Arizona, Arkansas, California, Connecticut, Delaware, Florida, Georgia, Hawaii, Idaho, Illinois, Indiana, Iowa, Kansas, Kentucky, Louisiana, Maine, Maryland, Massachusetts, Michigan, Minnesota, Mississippi, Missouri, Montana, Nebraska, New Hampshire, New Jersey, New Mexico, New York, North Carolina, Ohio, Oklahoma, Oregon, Pennsylvania, South Carolina, Tennessee, Texas, Utah, Vermont, Virginia, Washington, Wisconsin, Antigua and Barbuda, Barbados, Bermuda, Costa Rica, Cuba, Dominica, Dominican Republic, El Salvador, Guadeloupe, Haiti, Honduras, Jamaica, Martinique, Montserrat, Puerto Rico, St Vincent and Grenadines, Trinidad and Tobago, SOUTH AMERICA, Argentina, Brazil, Ceara, Espirito, Santo, Goias, Maranhao, Minas Gerais, Para, Parana, Piaui, Sao Paulo, Chile, Colombia, French, Guiana, Guyana, Suriname, Venezuela, OCEANIA, Australia, New South Wales, Queensland, South Australia, Tasmania, Victoria, Western, Australia, Cook Islands, Fed. States of Micronesia, Fiji, French, Polynesia, Kiribati, NFW Zealand, Niue, Samoa, Solomon Islands, Tonga, Vanuatu.

Genetic analysis of Okra Yellow Vein Mosaic Virus disease resistance in wild relative of okra Abelmoschus angulosus Wall. ex Wight & Arn

2019 ◽  
Vol 17 (04) ◽  
pp. 346-351
Author(s):  
SamanthiKumari Wasala ◽  
Sumudu I. Senevirathne ◽  
Jayantha Bandara Senanayake ◽  
Anuradini Navoditha
Keyword(s):  
Disease Resistance ◽  
Mosaic Virus ◽  
Disease Severity ◽  
Virus Disease ◽  
Disease Incidence ◽  
Severity Index ◽  
Yellow Vein ◽  
Wild Relative ◽  
Yellow Vein Mosaic Virus ◽  
Progress Curve

AbstractWild relative of okra, Abelmoschus angulosus Wall. ex Wight & Arn. was identified as a resistant germplasm for Okra Yellow Vein Mosaic Virus (OYVMV) which is the devastating disease for okra cultivation in Sri Lanka. The mode of resistance of OYVMV in A. angulosus was studied with the aim of tagging responsible genes for the disease resistance. Wide hybridization was performed between A. angulosus and highly virus susceptible A. esculentus variety, MI-7. Very poor seed setting was observed in F1 and F2 generations due to post zygotic abortion. Disease screening was carried out using F1 and F2:3 populations along with parents in the field under induced disease pressure. Disease severity index and area under disease progress curve were calculated to measure disease severity. Number of genes segregating for OYVMV disease resistance was calculated for the F2:3 generation. Modified Wright's formula was used to estimate the effective gene number and mode of inheritance by a quantitative method. A χ2 test was performed for qualitative analysis. Plants of A. angulosus were totally free of virus incidence while 100% disease incidence was observed in the variety MI-7. F2:3 population showed between and within progeny segregation for disease incidence. Results indicated that the disease susceptibility was dominant over resistance. χ2 analysis revealed that the segregation of disease severity was significantly fit to the gene model of 9:6:1 (χ2 = 0.1757 at P ≤ 0.05) suggesting the disease resistance in A. angulosus is governing by two recessive genes in an additive manner. Result was confirmed by the quantitative analysis.

scholarly journals Identification of Small Grains Genotypes Resistant to Wheat spindle streak mosaic virus

Plant Disease ◽  
2006 ◽  
Vol 90 (8) ◽  
pp. 1045-1050 ◽  
Author(s):  
L. Cadle-Davidson ◽  
M. E. Sorrells ◽  
S. M. Gray ◽  
G. C. Bergstrom
Keyword(s):  
Winter Wheat ◽  
Mosaic Virus ◽  
Control Strategy ◽  
Significant Interaction ◽  
Disease Incidence ◽  
Virus Titer ◽  
Incidence Data ◽  
Small Grains ◽  
Resistant Genotypes

Once Wheat spindle streak mosaic virus (WSSMV) becomes established in a field, the only available control strategy is the planting of resistant genotypes. In this study, we assessed 112 genotypes of winter wheat, rye, triticale, and barley for resistance to WSSMV in a 3-year trial in a field that had been used continuously for WSSMV evaluation for over 20 years. Because resistance to WSSMV reduces the percentage of plants that develop detectable virus titer and symptoms, we collected and analyzed disease incidence data. None of the genotypes was immune to infection. Sixty-two of the regionally adapted genotypes repeatedly expressed resistance to WSSMV, thus providing growers with a choice of cultivars resistant to WSSMV. Because of a significant interaction between genotypes and environment (year), genotypes should be assessed for incidence of symptomatic plants in multiple years, particularly when differentiating intermediate responses from highly susceptible and highly resistant responses.

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