When parents and children disagree: Informant discrepancies in reports of depressive symptoms in clinical interviews

2020 ◽  
Vol 272 ◽  
pp. 223-230 ◽  
Author(s):  
Noemi Baumgartner ◽  
Isabelle Häberling ◽  
Sophie Emery ◽  
Michael Strumberger ◽  
Kristin Nalani ◽  
...  
2011 ◽  
Vol 198 (1) ◽  
pp. 17-23 ◽  
Author(s):  
Carol Joinson ◽  
Jon Heron ◽  
Glyn Lewis ◽  
Tim Croudace ◽  
Ricardo Araya

BackgroundA growing number of studies suggest a link between timing of menarche and risk of depressive symptoms in adolescence, but few have prospectively examined the emergence of depressive symptoms from late childhood into adolescence.AimsTo examine whether girls who experience earlier menarche than their peers have higher levels of depressive symptoms in adolescence.MethodThe study sample comprised 2184 girls from the Avon Longitudinal Study of Parents and Children. The association between timing of menarche and depressive symptoms at 10.5, 13 and 14 years was examined within a structural equation model.ResultsGirls with early menarche (<11.5 years) had the highest level of depressive symptoms at 13 (P = 0.007) and 14 years (P<0.001) compared with those with normative and late timing of menarche.ConclusionsEarly maturing girls are at increased risk of depressive symptoms in adolescence and could be targeted by programmes aimed at early intervention and prevention.


BJPsych Open ◽  
2021 ◽  
Vol 7 (5) ◽  
Author(s):  
Priya Rajyaguru ◽  
Alex S. F. Kwong ◽  
Elizabeth Braithwaite ◽  
Rebecca M. Pearson

Background The relationships between offspring depression profiles across adolescence and different timings of parental depression during the perinatal period remain unknown. Aims To explore different timings of maternal and paternal perinatal depression in relation to patterns of change in offspring depressive mood over a 14 year period. Method Data were obtained from the Avon Longitudinal Study of Parents and Children (ALSPAC). Parental antenatal depression (ANTD) was assessed at 18 weeks gestation, and postnatal depression (PNTD) at 8 weeks postpartum. Population-averaged trajectories of offspring depressive symptoms were estimated using the Short Mood and Feelings Questionnaire (SMFQ) on nine occasions between 10 and 24 years of age. Results Full data were available for 5029 individuals. Offspring exposed to both timings of maternal depression had higher depressive symptoms across adolescence compared with offspring not exposed to ANTD or PNTD, characterised by higher depressive symptoms at age 16 (7.07 SMFQ points (95% CI = 6.19, 7.95; P < 0.001)) and a greater rate of linear change (0.698 SMFQ points (95% CI = 0.47, 0.93; P = 0.002)). Isolated maternal ANTD and to a lesser extent PNTD were also both associated with higher depressive symptoms at age 16, yet isolated maternal PNTD showed greater evidence for an increased rate of linear change across adolescence. A similar pattern was observed for paternal ANTD and PNTD, although effect sizes were attenuated. Conclusions This study adds to the literature demonstrating that exposure to two timings of maternal depression (ANTD and PNTD) is strongly associated with greater offspring trajectories of depressive symptoms.


2007 ◽  
Vol 191 (1) ◽  
pp. 84-85 ◽  
Author(s):  
Jonathan Evans ◽  
Jon Heron ◽  
Roshni R. Patel ◽  
Nicola Wiles

SummaryThere is conflicting evidence regarding the effect of depression during pregnancy on birth weight. We used data from the Avon Longitudinal Study of Parents and Children to investigate whether depressive symptoms during pregnancy in 10 967 women led to low birth weight at term in their offspring. Those with a high depressive symptom score during pregnancy were more likely to have babies of low birth weight (95% CI 1.16–2.40, P < 0.01), but this attenuated after adjustment for confounders (OR = 1.29, 95% CI 0.87–1.91, P = 0.210). Hence there is little evidence of an independent association between depressive symptoms during pregnancy and birth weight.


2013 ◽  
Vol 43 (12) ◽  
pp. 2615-2626 ◽  
Author(s):  
I. Culpin ◽  
J. Heron ◽  
R. Araya ◽  
R. Melotti ◽  
C. Joinson

BackgroundPrevious studies suggest a link between parental separation or divorce and risk of depression in adolescence. There are, however, few studies that have prospectively examined the effects of timing of biological father absence on risk for depressive symptoms in adolescence while controlling for a range of confounding factors.MethodWe examine the association between father absence occurring in early (the first 5 years) and middle childhood (5–10 years) and adolescent depressive symptoms in a sample comprising 5631 children from the UK-based Avon Longitudinal Study of Parents and Children (ALSPAC). Self-reported depressive symptoms at 14 years were assessed using the Short Mood and Feelings Questionnaire (SMFQ). Father absence was assessed from maternal questionnaires completed at regular intervals from the birth of the study child up to 10 years.ResultsThere was evidence for an association between father absence in early childhood and increased odds of depressive symptoms at 14 years. This association was stronger in girls than in boys and remained after adjusting for a range of socio-economic, maternal and familial confounders assessed prior to the father's departure. Conversely, there was no evidence for an association between father absence in middle childhood and depressive symptoms at 14 years.ConclusionsFather absence in early childhood increases risk for adolescent depressive symptoms, particularly in girls. Future research should be aimed at identifying possible biological and psychosocial mechanisms linking father absence to depressive symptomatology to enable the development of family-based early prevention and intervention programmes targeting young children at risk.


2020 ◽  
Author(s):  
Tim Cadman ◽  
Alex Siu Fung Kwong ◽  
Paul Moran ◽  
Heather O'Mahen ◽  
Iryna Culpin ◽  
...  

Background: Parental personality may influence the course of offspring depression but this is unclear. It is also unknown whether the impact of parental personality on offspring depression is moderated by socioeconomic position (SEP). Our aims were to describe trajectories of depressive symptoms across adolescence for offspring of parents with and without maladaptive personality traits and to test for effect modification by SEP. Methods: A longitudinal study in the Avon Longitudinal Study of Parents and Children birth cohort (ALSPAC; ns= 3054 to 7046). Exposures were binary measures of maladaptive parental personality traits and the outcome was depressive symptoms ages 11 to 24 (SMFQ; range 0 to 26). Results: Offspring of mothers with high maladaptive traits showed higher levels of depressive symptoms at all ages (SMFQ difference at age 10 = 0.66, CI 0.25, 1.28, p = 0.02; age 22 = 1.00, CI 0.51, 1.50, p < 0.001). There was weaker evidence of an association between paternal maladaptive personality traits and offspring depressive symptoms (SMFQ difference at age 10 = 0.21, CI -0.58, - 0.99, p = 0.60; age 22 = 0.02, CI -0.94, 0.90, p= 0.97). We found no consistent evidence of effect modification by SEP. Conclusions: Offspring of mothers with high levels of maladaptive personality traits show evidence of greater depressive symptoms throughout adolescence although the absolute increase in symptoms is small. Evidence for the effect of fathers personality was weaker. Socioeconomic position and maladaptive personality traits appear to be independent risk factors.


2021 ◽  
Vol 9 (1) ◽  
Author(s):  
Parisa Vidafar ◽  
Anastasia K. Yocum ◽  
Peisong Han ◽  
Melvin G. McInnis ◽  
Helen J. Burgess

Abstract Background There is increasing evidence that bipolar disorder is influenced by circadian timing, including the timing of sleep and waking activities. Previous studies in bipolar disorder have shown that people with later timed daily activities, also known as late chronotypes, are at higher risk for subsequent mood episodes over the following 12–18 months. However, these studies were limited to euthymic patients and smaller sample sizes. The aim of the current study was to further examine baseline chronotype as a potentially important predictor of mood-related outcomes in a larger sample of individuals with bipolar disorder and over the longest follow up period to date, of 5 years. Participants included 318 adults diagnosed with bipolar I and II (19–86 years) who were enrolled in the Prechter Longitudinal Study of Bipolar Disorder. Results Participants with a late chronotype were found to be more likely to have mild to more severe depressive symptoms (PHQ-9 ≥ 5) as captured with PHQ-9 assessments every 2 months over the 5 year follow up period. This higher risk for depressive symptoms remained even after adjusting for age, sex and mood at baseline. Additionally, late chronotypes reported fewer hypomania/mania episodes during the 5 year follow up, as derived from clinical interviews every two years. Conclusions These results highlight the potential clinical usefulness of a single self-report question, in identifying patients at risk for a more depressive mood course. The results also suggest that circadian phase advancing treatments, that can shift circadian timing earlier, should be explored as a means to reduce depressive symptoms in late chronotypes with bipolar disorder.


2020 ◽  
pp. 1-11
Author(s):  
Anna B. Chaplin ◽  
Peter B. Jones ◽  
Golam M. Khandaker

Abstract Background Childhood infections are associated with adult psychosis and depression, but studies of psychotic experiences (PEs) and depressive symptoms in childhood, adolescence, and early-adulthood are scarce. Previous studies have typically examined severe infections, but studies of common infections are also scarce. Methods Using data from the Avon Longitudinal Study of Parents and Children (ALSPAC) birth cohort, we examined associations of the number of infections in childhood from age 1.5 to 7.5 years with depressive symptom scores at age 10, 13, 14, 17, 18, and 19 years, and with PEs at 12 and 18 years. We performed additional analysis using infection burden (‘low’ = 0–4 infections, ‘medium’ = 5–6, ‘high’ = 7–9, or ‘very high’ = 10–22 infections) as the exposure. Results The risk set comprised 11 786 individuals with childhood infection data. Number of childhood infections was associated with depressive symptoms from age 10 (adjusted beta = 0.14; standard error (s.e.) = 0.04; p = <0.01) to 17 years (adjusted beta = 0.17; s.e. = 0.08; p = 0.04), and with PEs at age 12 (suspected/definite PEs: adjusted odds ratio (OR) = 1.18; 95% confidence interval (CI) = 1.09–1.27). These effect sizes were larger when the exposure was defined as very high infection burden (depressive symptoms age 17: adjusted beta = 0.79; s.e. = 0.29; p = 0.01; suspected/definite PEs at age 12: adjusted OR = 1.60; 95% CI = 1.25–2.05). Childhood infections were not associated with depressive/psychotic outcomes at age 18 or 19. Conclusions Common early-childhood infections are associated with depressive symptoms up to mid-adolescence and with PEs subsequently in childhood, but not with these outcomes in early-adulthood. These findings require replication including larger samples with outcomes in adulthood.


2020 ◽  
Author(s):  
Tim Cadman ◽  
Amanda Hughes ◽  
Caroline Wright ◽  
José A López-López ◽  
Tim Morris ◽  
...  

ABSTRACTPrevious research on the relationship between children’s externalising and depressive symptoms, experience of school, and later academic attainment is inconclusive. The present study uses data from the Avon Longitudinal Study of Parents and Children (n=6,409) to investigate bidirectional associations between school experience (enjoyment and connectedness) and externalising and depressive symptoms at age 10-11 and 13-14. We also investigate the relationship between school experience and academic attainment at 16 and test whether school experience mediates associations of externalising and depressive symptoms with later attainment. A cross-lagged structural equation model was employed. Externalising and depressive symptoms at 10-11 were negatively associated with school connectedness at 13-14 (externalizing: standardised β=−0.13, CI: −0.17, −0.08; depressive: β=−0.06, CI: −0.11, 0.01), and with school enjoyment at 13-14 (externalising: β=−0.08, CI: −0.13, −0.03; depressive β=−0.04, −0.08, 0.03). School enjoyment at 13-14 was positively associated with attainment at 16 (β=0.10, CI: 0.04, 0.15), and partially mediated associations between externalising and depressive symptoms at 10-11 and attainment at 16 (externalising: proportion mediated; 4.7%, CI: 0.7, 10.1, depressive: proportion mediated 2.2%, I: −1.5, 5.9). School enjoyment is a potentially modifiable risk factor that may affect educational attainment of adolescents with depressive and externalising symptoms.


2017 ◽  
Vol 21 (5) ◽  
pp. 831-837 ◽  
Author(s):  
Kate Northstone ◽  
Carol Joinson ◽  
Pauline Emmett

AbstractObjectiveThere is evidence to suggest that individual components of dietary intake are associated with depressive symptoms. Studying the whole diet, through dietary patterns, has become popular as a way of overcoming intercorrelations between individual dietary components; however, there are conflicting results regarding associations between dietary patterns and depressive symptoms. We examined the associations between dietary patterns extracted using principal component analysis and depressive symptoms, taking account of potential temporal relationships.DesignDepressive symptoms in parents were assessed using the Edinburgh Postnatal Depression Scale (EPDS) when the study child was 3 and 5 years of age. Scores >12 were considered indicative of the presence of clinical depressive symptoms. Diet was assessed via FFQ when the study child was 4 years of age.SettingLongitudinal population-based birth cohort.SubjectsMothers and fathers taking part in the Avon Longitudinal Study of Parents and Children when their study child was 3–5 years old.ResultsUnadjusted results suggested that increased scores on the ‘processed’ and ‘vegetarian’ patterns in women and the ‘semi-vegetarian’ pattern in men were associated with having EPDS scores ≥13. However, after adjustment for confounders all results were attenuated. This was the case for all those with available data and when considering a sub-sample who were ‘disease free’ at baseline.ConclusionsWe found no association between dietary patterns and depressive symptoms after taking account of potential confounding factors and the potential temporal relationship between them. This suggests that previous studies reporting positive associations may have suffered from reverse causality and/or residual confounding.


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