scholarly journals The relation of small head circumference and thinness at birth to death from cardiovascular disease in adult life.

BMJ ◽  
1993 ◽  
Vol 306 (6875) ◽  
pp. 422-426 ◽  
Author(s):  
D J Barker ◽  
C Osmond ◽  
S J Simmonds ◽  
G A Wield
Keyword(s):  
Cardiovascular Disease ◽  
Head Circumference ◽  
Adult Life ◽  
Small Head

HEAD CIRCUMFERENCE, MENTAL RETARDATION, AND GROWTH FAILURE

PEDIATRICS ◽  
1965 ◽  
Vol 36 (1) ◽  
pp. 62-66
Author(s):  
Edward J. O'Connell ◽  
Robert H. Feldt ◽  
Gunnar B. Stickler
Keyword(s):  
Mental Retardation ◽  
Head Circumference ◽  
Growth Failure ◽  
Clinical Impression ◽  
Severe Mental Retardation ◽  
Small Head ◽  
The Mean ◽  
Standard Deviations ◽  
Mental Subnormality ◽  
Age And Sex

The purpose of this study was to re-affirm our clinical impression that non-institutionalized children whose head circumference was below minus 2 standard deviations were mentally subnormal and frequently had growth failure. A group of 134 children with a head circumference below minus 2 standard deviations from the mean were studied, and all but one were mentally subnormal. The most severe mental retardation was noted in the group of children with a head circumference of minus 4 standard deviations or below. We found, as have others, that children with mental retardation have height and weights below the expected norm and that children with a head circumference below minus 2 standard deviations have even lower mean heights and weights. The head circumference of 31 children with growth failure and normal intelligence was normal for age and sex, therefore disproving the concept that the abnormally small child has a proportionally small head. In the child with growth failure, should the head be proportionally small (below minus 2 standard deviations), mental subnormality should be suspected. We feel that the head circumference measurement has taken on new clinical significance in that our data support its use in suspecting the association of mental subnormality in children with growth failure and a head circumference of below minus 2 standard deviations from the mean for age and sex.

scholarly journals Effect of maternal anaemia on birth weight of term babies

2018 ◽  
Vol 5 (3) ◽  
pp. 1019
Author(s):  
Mohandas Nair ◽  
Gireesh S. ◽  
Rubeena Yakoob ◽  
Cheriyan N. C.
Keyword(s):  
Birth Weight ◽  
Low Birth Weight ◽  
Head Circumference ◽  
Adult Life ◽  
Anthropometric Measures ◽  
Maternal Anaemia ◽  
Medical College ◽  
Anemia During Pregnancy ◽  
The Difference ◽  
Long Term Impact

Background: Low birth weight is the major determinant of mortality, morbidity and disability in infancy and childhood and has a long-term impact on health outcome in adult life. The objectives of this study were to study the relationship between maternal anemia and birth weight of babies and to study anthropometric measures of babies born to anemic and non-anemic mothers and to correlate the timing of anemia with birth weight of babies.Methods: Term babies born in Institute of Maternal and Child Health, Government Medical College, Kozhikode from November 2014 to October 2016 fulfilling the criteria were divided into 2 groups, cases (term babies with birth weight <2500g) and controls (term babies with weight >2500g) and studied and their maternal hemoglobin values were compared.Results: Maternal anaemia in all three trimesters was found to be more in cases compared to controls. Mean 1st trimester hemoglobin of cases was 10.68 g/dl which was significantly lower when compared to controls. Mean 2nd trimester hemoglobin of cases was 10.36 g/dl compared to 11.47 g/dl in controls. Mean 3rd trimester hemoglobin of cases was 10.42 g/dl which was also significantly lower compared to 11.32 g/dl in controls. SGA babies were also found to be more in cases, 89%, compared to 18% in controls. The difference in head circumference between two groups was not statistically significant. Mean length of babies were higher in controls compared to cases. Mothers with anemia at any time during pregnancy was found to have 4.3 times higher risk of giving birth to low birth weight babies compared to non-anemic mothersConclusion: Anemia during pregnancy is a risk factor for low birth weight and SGA, independent of the trimester. Length of babies born to anaemic mothers is also low. But it does not have a significant effect on head circumference of babies.

scholarly journals Is isolated small head circumference at 20 weeks predictive of FGR or SGA at delivery?

2022 ◽  
Vol 226 (1) ◽  
pp. S204-S205
Author(s):  
Brad Bosse ◽  
Madeline Wetterhahn ◽  
Erin Bailey ◽  
Janine Rhoades ◽  
J. Igor Iruretagoyena ◽  
...  
Keyword(s):  
Head Circumference ◽  
Small Head

Head Circumference in ‘Preschizophrenic’ and Control Neonates

1993 ◽  
Vol 162 (4) ◽  
pp. 517-523 ◽  
Author(s):  
Thomas F. McNeil ◽  
Elizabeth Cantor-Graae ◽  
Lars G. Nordström ◽  
Thomas Rosenlund
Keyword(s):  
Body Length ◽  
Head Circumference ◽  
Medical Records ◽  
Genetic Factor ◽  
Cerebral Development ◽  
Small Head ◽  
Schizophrenic Patients ◽  
History Of ◽  
Preterm Babies ◽  
And Control

Head circumference, body weight, body length, and shoulder circumference at birth were studied in 70 RDC schizophrenic patients and 70 demographically matched controls from the same delivery series, using information recorded in the medical records at the time of birth. With preterm babies removed from both samples, only head circumference among preschizophrenic infants was significantly smaller than that of controls. Preschizophrenic infants also had a disproportionately smaller head circumference in relation to body length than did controls. Among the former, a small head circumference was systematically related to an absence of family history of psychosis, but was not related to season of birth or recorded pregnancy complications. The findings were strongest for females. The results suggest there is an unidentified non-genetic factor in schizophrenia that disturbs prenatal cerebral development.

Preterm Birth as a Risk Factor for Metabolic Syndrome and Cardiovascular Disease in Adult Life: A Systematic Review and Meta-Analysis

2019 ◽  
Vol 210 ◽  
pp. 69-80.e5 ◽  
Author(s):  
Panagiota Markopoulou ◽  
Eleni Papanikolaou ◽  
Antonis Analytis ◽  
Emmanouil Zoumakis ◽  
Tania Siahanidou
Keyword(s):  
Systematic Review ◽  
Cardiovascular Disease ◽  
Metabolic Syndrome ◽  
Risk Factor ◽  
Preterm Birth ◽  
Meta Analysis ◽  
Adult Life

The Diet of Girls and Young Women at the Beginning of the Century

1993 ◽  
Vol 9 (1) ◽  
pp. 15-23 ◽  
Author(s):  
J. Fellague Ariouat ◽  
D.J.P. Barker
Keyword(s):  
Cardiovascular Disease ◽  
Young Women ◽  
Cardiovascular Mortality ◽  
Maternal Nutrition ◽  
Adult Life ◽  
Cardiovascular Death ◽  
Early Growth ◽  
Fetal Life ◽  
Fertility Rates ◽  
Death Rates

Recent research has shown that retarded growth during fetal life and infancy is linked to the development of cardiovascular disease (coronary heart disease and stroke) in adult life. Maternal nutrition has an important effect on early growth and the diets of young women may therefore influence cardiovascular disease in the next generation. Samples of women aged 80 years and over were interviewed in six areas of England and Wales with different cardiovascular death rates. The women, 281 in total, were asked about their diets when they were aged 10 to 15 years. Those who grew up in areas which now have low cardiovascular mortality tended to eat four meals a day rather than three, to live in households which had gardens, kept hens or livestock, and to go into domestic service, where diets were generally good. Those who grew up in areas which now have high cardiovascular mortality tended to eat less red meat, to live in houses without gardens, to enter industrial occupations and have higher fertility rates.

Adult life events increase the risk of Cardiovascular Disease and Depressiveness among those with childhood adversities

2008 ◽  
Vol 23 ◽  
pp. S85
Author(s):  
K. Korkeila ◽  
J. Korkeila ◽  
J. Vahtera ◽  
L. Sillanmäki ◽  
M. Koskenvuo
Keyword(s):  
Cardiovascular Disease ◽  
Life Events ◽  
Adult Life ◽  
Childhood Adversities

scholarly journals Possible Health Implications and Low Vitamin D Status during Childhood and Adolescence: An Updated Mini Review

2010 ◽  
Vol 2010 ◽  
pp. 1-7 ◽  
Author(s):  
Dimitrios Papandreou ◽  
Pavlos Malindretos ◽  
Zacharoula Karabouta ◽  
Israel Rousso
Keyword(s):  
Cardiovascular Disease ◽  
Vitamin D ◽  
Developing Countries ◽  
Life Span ◽  
Vitamin D Deficiency ◽  
Chronic Diseases ◽  
Adult Life ◽  
Childhood And Adolescence ◽  
Vitamin D Status ◽  
Calcium Insufficiency

Vitamin D deficiency is common in the developing countries and exists in both childhood and adult life. The great importance of Vitamin D is the moderation of calcium (Ca) and phosphorus (P) homeostasis as well as the absorption of Ca. While insufficiency of vitamin D is a significant contributing factor to risk of rickets in childhood, it is possible that a more marginal deficiency of vitamin D during life span contribute to osteoporosis as well as potentially to the development and various other chronic diseases such as cardiovascular disease, cancer and diabetes. This paper reviews the metabolism, epidemiology, and treatment of vitamin D and calcium insufficiency as well as its relation to various diseases during childhood and adolescence.

Elastic Properties of the Human Umbilical Cord in Preeclampsia

2011 ◽  
Author(s):  
Reuben B. Dodson ◽  
Kendall S. Hunter ◽  
Virginia L. Ferguson
Keyword(s):  
Cardiovascular Disease ◽  
Coronary Heart Disease ◽  
High Fat Diet ◽  
Adult Life ◽  
Circulatory System ◽  
Human Umbilical Cord ◽  
High Fat ◽  
Epidemiological Evidence ◽  
Fetal Origins ◽  
Genetically Determined

Maternal diseases of pregnancy have been found to detrimentally affect the fetal circulatory system, with consequences lasting well into adulthood. In 1995, Barker introduced the idea that major disorders of adult life (including coronary heart disease, hypertension, stroke and diabetes) arise not only through an interaction between factors in our lifestyle, such as a high-fat diet, obesity and smoking, and a genetically determined susceptibility but also through development in utero [1]. Epidemiological evidence continues to support the notion that adult cardiovascular disease (CVD) has fetal origins [2–5].

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