scholarly journals The Effect of Bathing Length Time to Axillary Temperature and Dry Heat Loss on Newborns; A Comparative Analysis

2021 ◽  
Vol 14 (05) ◽  
pp. 969-974
2014 ◽  
Vol 3 (3) ◽  
Author(s):  
Hotma Sauhur Hutagaol ◽  
Eryati Darwin ◽  
Eny Yantri

AbstrakHipotermia merupakan penyebab utama kesakitan dan kematian bayi baru lahir di negara berkembang. Salah satu asuhan untuk mencegah hipotermi adalah dengan melaksanakan Inisiasi Menyusu Dini (IMD). Tujuan penelitian ini adalah untuk mengetahui pengaruh inisiasi menyusu dini terhadap suhu aksila dan kehilangan panas kering pada bayi baru lahir. Ini merupakan studi cross-sectional comparative yang melakukan observasi bayi yang lahir dengan persalinan normal yang dilaksanakan IMD atau tidak, kemudian dilakukan pengukuran suhu aksila dan kehilangan panas kering pada kedua kelompok. Data dianalisa menggunakan uji t-test, dan nilai p<0.05 dianggap bermakna secara statistik. Rerata suhu aksila kelompok IMD sebesar 37,1 ± 0,20C dan rerata suhu aksila pada kelompok non IMD sebesar 36,8 ± 0,40C. Rerata total kehilangan panas kering pada kelompok IMD sebesar 30,1 ± 3,4 J dan pada kelompok non IMD sebesar 31,2 ± 3,9 J. Hasil penelitian menyimpulkan bahwa IMD berpengaruh terhadap peningkatan suhu aksila. Kehilangan panas kering lebih rendah pada kelompok IMD walau tidak bermakna secara statistikKata kunci: IMD, suhu aksila, kehilangan panas keringAbstractHypothermia is a major cause of morbidity and mortality in neonatal period. One of essential care for newborn to prevent hypothermia is early initiation of breastfeeding. The objective of this study was to see the effects of early initiation of breastfeeding to increase axillary temperature and decrease dry heat loss in newborn. The design of this study is observational study with cross-sectional comparative design. The subjects were normal newborn with early initiation of breastfeeding and without early initiation of breastfeeding. Axillary mean temperature after early initiation of breastfeeding is 37,1 ± 0,20C and axillary mean temperature on non early initiation of breastfeeding group is 36,8 ± 0,40C. Total dry heat loss mean on early initiation of breastfeeding group is 30,1 ± 3,4 J and on non early initiation of breastfeeding group is 31,2 ± 3,9 J. This study concluded that there is the effect of early initiation of breastfeeding to axillary temperature. Total dry heat loss is lower on early initiation of breastfeeding group but not significant statistically.Keywords: early initiation of breastfeeding, axillary temperature, dry heat loss


2017 ◽  
Vol 235 (2) ◽  
pp. 111-122 ◽  
Author(s):  
Riccardo Dore ◽  
Luka Levata ◽  
Sogol Gachkar ◽  
Olaf Jöhren ◽  
Jens Mittag ◽  
...  

Nesfatin-1 is a bioactive polypeptide expressed both in the brain and peripheral tissues and involved in the control of energy balance by reducing food intake. Central administration of nesfatin-1 significantly increases energy expenditure, as demonstrated by a higher dry heat loss; yet, the mechanisms underlying the thermogenic effect of central nesfatin-1 remain unknown. Therefore, in this study, we sought to investigate whether the increase in energy expenditure induced by nesfatin-1 is mediated by the central melanocortin pathway, which was previously reported to mediate central nesfatin-1´s effects on feeding and numerous other physiological functions. With the application of direct calorimetry, we found that intracerebroventricular nesfatin-1 (25 pmol) treatment increased dry heat loss and that this effect was fully blocked by simultaneous administration of an equimolar dose of the melanocortin 3/4 receptor antagonist, SHU9119. Interestingly, the nesfatin-1-induced increase in dry heat loss was positively correlated with body weight loss. In addition, as assessed with thermal imaging, intracerebroventricular nesfatin-1 (100 pmol) increased interscapular brown adipose tissue (iBAT) as well as tail temperature, suggesting increased heat production in the iBAT and heat dissipation over the tail surface. Finally, nesfatin-1 upregulated pro-opiomelanocortin and melanocortin 3 receptor mRNA expression in the hypothalamus, accompanied by a significant increase in iodothyronine deiodinase 2 and by a nonsignificant increase in uncoupling protein 1 and peroxisome proliferator-activated receptor gamma coactivator-1 alpha mRNA in the iBAT. Overall, we clearly demonstrate that nesfatin-1 requires the activation of the central melanocortin system to increase iBAT thermogenesis and, in turn, overall energy expenditure.


1980 ◽  
Vol 238 (5) ◽  
pp. R400-R405 ◽  
Author(s):  
M. L. Laudenslager ◽  
C. W. Wilkinson ◽  
H. J. Carlisle ◽  
H. T. Hammel

The effect of estrogen replacement on several parameters of energy balance was investigated in ovariectomized rats tested during the dark phase of their diurnal cycle. Estrogen replacement, either as 17 beta-estradiol or beta-estradiol-3-benzoate via subcutaneous Silastic capsules, was associated with elevated rates of heat production and dry heat loss relative to untreated ovariectomized controls. Estrogen treatment reduced body mass and retarded fur growth. The effects of estrogen replacement on heat production and dry heat loss could not be attributed to these differences in body mass and fur growth or locomotor activity. Estrogen replacement had no effect on rate of evaporative heat loss. If estrogen replacement was delayed 75 days following ovariectomy, the increase in heat production and dry heat loss was not observed. There was no effect of the hormone treatment on rectal temperature. It was concluded that either heat production was elevated, with dry heat loss increased to compensate for the additional thermal load, or dry heat loss was accelerated with heat production elevated in compensation.


1988 ◽  
Vol 24 (2) ◽  
pp. 272-272
Author(s):  
Georg Simbruner ◽  
Margit-Andrea Glatzl-Hawlik
Keyword(s):  

2019 ◽  
Vol 10 (1) ◽  
pp. 32
Author(s):  
Fenny Fernando ◽  
Ayu Gustia Ningsih ◽  
Melia Pebrina ◽  
Honesty Diana Morika

ABSTRAKBBLR masih terus menjadi masalah kesehatan masyarakat yang signifikan secara global mencapai 15% bayi di seluruh dunia (lebih dari 20 juta jiwa). Sangat penting Terapi pendamping menggunakan metode kangguru yang bisa digunakan untuk mencegah terjadinya hipotermi karena tubuh ibu dapat memberikan kehangatan kepada bayinya secara terus menerus dengan cara kontak antara kulit ibu dengan kulit bayi.Tujuan penelitian ini mempelajari pengaruh metode kangguru terhadap peningkatan suhu pada bayi BBLR. Jenis penelitian ini quasi eksperimen ,dengan rancangan studi rancangan yang berupaya untuk mengungkapkan hubungan sebab akibat dengan melibatkan kelompok kontrol Bayi BBLR yang melakukan perawatan dengan inkubator dan kelompok eksperimen  Bayi  BBLR yang melakukan perawatan dengan metode kanguru dengan rancangan post test only. Penelitian dilakukan pada Bulan Juni sampai dengan November 2018. Penelitian dilaksanakan di RSUD D.Rasidin dan RS TK III Dr.Reksdiwiryo Populasi adalah Seluruh ibu yang melahirkan bayi BBLR   terdata di rekam medis RSUD D.Rasidin dan RS Tk III.Reksodiwiryo  berjumlah 26 Bayi. Sampel berjumlah 26 BBLR yang diambil dengan teknik consecutive sampling. Data dianalisis  menggunakan uji Wilcoxon, dan nilai p < 0,05 dianggap bermakna secara statistik. Rerata suhu aksila kelompok metode kanguru 36,8 ±0,3  dan rerata suhu aksila pada kelompok inkubator 36,4±0,1. Rerata total kehilangan panas kering pada kelompok metode kanguru dsan inkubator sebesar 29,66 ± 0,53 J dan pada kelompok inkubator 34,28 ± 0,77 J. Hasil penelitian menyimpulkan bahwa ada pengaruh metode kangguru terhadap suhu aksila pada bayi BBLR. Disarankan hasil penelitian ini dapat dijadikan bahan perbandingan apabila dilakukan penelitian lebih lanjut mengenai perawatan yang tepat pada BBLR.Kata Kunci : BBLR, Metode Kangguru dan Inkubator THE EFFECTIVENESS OF KANGAROO METHOD ON TEMPERATURE AND HEAT LOSS IN LOW BIRTH WEIGHT BABIES (LBW)                ABSTRACTLow Birth Weight still continues to be a significant public health problem globally that reached until 15% of babies worldwide (more than 20 million people). The therapy of using the Kangaroo method can be used to prevent hypothermia because the mother's body can provide warmth to the baby continuously by contact the mother's skin with the baby's skin. The purpose of this study was to study the effect of kangaroo method on increasing temperature in Low Birth Weight. The type of research was quasi-experimental, with a design study to reveal a causal relationship between control group of Low Birth Weight infants who cared for an incubator and an experimental group for LBW infants who were treated with Kangaroo methods. This research used a post test design only. The study have done in June untill November 2018. The study was carried out in Dr.Rasidin Hospital and Dr.Reksodiwiryo Hospital. The population was all mothers who gave birth to low bith weight were recorded in the medical records of Dr.Rasidin Hospital and Reksodiwiryo Hospital. The sample amounted to 26 low birth weight taken by consecutive sampling technique. Data were analyzed using Wilcoxon Test, and p <0.05 was considered statistically significant. The mean axillary temperature of the kangaroo method group was 36.8 ± 0.3 and the mean axillary temperature in the incubator group was 36.4 ± 0.1. The average total dry heat loss in the kangaroo method was 29.66 ± 0.53 J and in the incubator group 34.28 ± 0.77 J. The results concluded that there the kangaroo method on axillary temperature in low birth weight. It is suggested that the results of this study can be used as a comparison for the next research. Keyword: Low Birth Weight, Kangaroo methode,incubator methodeE


2019 ◽  
Author(s):  
Joshua K. Robertson ◽  
Gabriela Mastromonaco ◽  
Gary Burness

AbstractChanges in body temperature following exposure to stressors have been documented for nearly two millennia, however, the functional value of this phenomenon is poorly understood. We tested two competing hypotheses to explain stress-induced changes in temperature, with respect to surface tissues. Under the first hypothesis, changes in surface temperature are a consequence of vasoconstriction that occurs to attenuate blood-loss in the event of injury and serves no functional purpose per se; defined as the Haemoprotective Hypothesis. Under the second hypothesis, changes in surface temperature reduce thermoregulatory burdens experienced during activation of a stress response, and thus hold a direct functional value; here, the Thermoprotective Hypothesis. To understand whether stress-induced changes in surface temperature have functional consequences, we tested predictions of the Haemoprotective and Thermoprotective hypotheses by exposing Black-capped Chickadees (n = 20) to rotating stressors across an ecologically relevant ambient temperature gradient, while non-invasively monitoring surface temperature (eye region temperature) using infrared thermography. Our results show that individuals exposed to rotating stressors reduce surface temperature and dry heat loss at low ambient temperature and increase surface temperature and dry heat loss at high ambient temperature, when compared to controls. These results support the Thermoprotective Hypothesis and suggest that changes in surface temperature following stress exposure have functional consequences and are consistent with an adaptation. Such findings emphasize the importance of the thermal environment in shaping physiological responses to stressors in vertebrates, and in doing so, raise questions about their suitability within the context of a changing climate.Summary StatementWe provide empirical evidence for a functional value to stress-induced changes in surface temperature that is consistent with an adaptation, using a temperate endotherm (Black-capped Chickadee) as a model species.


1982 ◽  
Vol 243 (1) ◽  
pp. R70-R76
Author(s):  
M. L. Laudenslager ◽  
H. J. Carlisle ◽  
S. E. Calvano

The role of the thyroid in the mediation of an estrogen-associated change in thermal balance was studied in thyroidectomized and in propylthiouracil-treated ovariectomized rats. Prior to propylthiouracil treatment, estrogen-treated ovariectomized rats and intact female rats had higher rates of heat production and dry heat loss at -5 degrees C than ovariectomized rats. Heat production of estrogen-treated and intact female rats was well below their rates of dry heat loss without an alteration in the absolute rate of heat loss in the hypothyroid condition. Heat production exceeded heat loss only in the hypothyroid ovariectomized group not receiving estrogen. Ovariectomized rats without estrogen maintained thermal balance, whereas rectal temperatures fell in both intact and estrogen-treated ovariectomized rats during cold exposure. Increased heat loss unbalanced by heat production was also observed in surgically thyroidectomized estrogen-treated ovariectomized rats tested at -5 degrees C. These results suggest that an estrogen-induced increase in heat loss, which is compensated by an increase in heat production in the euthyroid but not the hypothyroid condition, is one mechanism responsible for estrogen-associated changes in thermal balance during cold exposure.


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