scholarly journals 1117 Associations Between Health Behaviors And Sleep Duration In Women With Depression

SLEEP ◽  
2020 ◽  
Vol 43 (Supplement_1) ◽  
pp. A425-A426
Author(s):  
R Peprah ◽  
D Jenkins ◽  
T Donley ◽  
A Sexias ◽  
G Jean-Louis
Keyword(s):  
Sleep Duration ◽  
Adult Women ◽  
Obese Women ◽  
Short Sleep ◽  
Representative Study ◽  
Falling Asleep ◽  
Significance Levels ◽  
Normal Sleep ◽  
Nationally Representative ◽  
Impaired Sleep

Abstract Introduction Sleep duration can have important effects on health. Long and short sleep has been associated with negative health outcomes in women. Depression may aggravate an already impaired sleep quality. This study explored associations between sleep duration and depression in pregnant women. Methods We analyzed data for adult women (n=9,372) from the 2017 and 2018 National Health Interview Survey (NHIS), which is a nationally representative study of the US civilian non-institutionalized population. Sleep was categorized by short (≤6 hrs), normal/healthy (7-8 hrs), and long (≥9 hrs) sleep. Using STATA 15.0 for Windows, we report weighted frequencies and Chi-and square tests. Alpha of 0.05 was used for all significance levels. Results Of the sample, 81.7% of the women were White, 10.6% were Black and 7.7% were other minorities. The mean age was 51.4±18.3. We found that the proportion of women who reported short sleep increased with age (p<0.000). Current drinkers (37%) had higher numbers of normal sleep than those who were former drinkers or abstainers (p<0.000). With respect to BMI, more obese women were short and sleepers (17% and 4% respectively), but women with normal BMI (19%) were normal sleepers (p<0.000). In short sleepers, more women had trouble falling asleep (13%) and staying asleep (17%), but reported not using medication and never feeling rested. Similar results were found for long sleepers. Higher proportions of normal sleepers reported not have trouble falling asleep (27%), staying asleep (26%), or using medication for sleep (40%) (p<0.000). However, of those who reported normal sleep, greater frequencies of working up feeling rested occurred only 3-6 times in the past week (p<0.000). Conclusion In this study, women with depression self-reported more normal sleep duration. This finding is inconsistent with previous research. Whether this association is causal and what pathways explain this association is unknown. Support This study was supported by funding from the NIH: R01MD007716, R01HL142066, K01HL135452, and K07AG052685.

Abstract P545: Insufficient Sleep and Nitric Oxide-Mediated Endothelium-Dependent Vasodilation in Adults With Elevated Blood Pressure

Circulation ◽  
2020 ◽  
Vol 141 (Suppl_1) ◽  
Author(s):  
Kelly A Stockelman ◽  
Anthony R Bain ◽  
Dana M Withrow ◽  
Tracey A Larson ◽  
Elizabeth M Boland ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Nitric Oxide ◽  
Blood Pressure ◽  
Sleep Duration ◽  
Elevated Blood Pressure ◽  
Elevated Blood ◽  
Short Sleep Duration ◽  
Insufficient Sleep ◽  
Short Sleep ◽  
Normal Sleep

Elevated blood pressure (BP ≥130/80 mmHg) is associated with increased risk for myocardial infarction, heart failure, stroke and vascular disease. Insufficient nightly sleep (<7 h/night) has been linked not only to the etiology of elevated blood pressure but is a prevalent, often ignored, comorbidity. Indeed, short sleep duration is now considered to be a plausible risk factor for elevated blood pressure and a harbinger of increased cardiovascular risk. A high prevalence of insufficient nightly sleep has been reported in adults with elevated blood pressure. The influence of insufficient sleep on endothelial vasodilator function in adults with elevated blood pressure is unknown. We tested the hypotheses that chronic insufficient sleep is associated with diminished nitric oxide (NO)-mediated endothelium-dependent vasodilation in adults with elevated blood pressure. Moreover, the insufficient sleep-related reduction in endothelial vasodilator function is due, at least in part to increased oxidative stress. Thirty-five middle-aged and older adults with elevated blood pressure were studied: 15 with normal nightly sleep duration (11M/4F; age: 58±2 yr; BP: 136/82±1/2 mmHg; sleep: 7.6±0.2 h/night) and 20 with short nightly sleep duration (14M/6F; 58±1 yr; BP: 138/84±1/1 mmHg; sleep: 6.0±0.1 h/night). Forearm blood flow (FBF) responses to intra-arterial infusion of acetylcholine (ACh), in the absence and presence of the endothelial NO synthase inhibitor N G -monomethyl-L-arginine (L-NMMA) and the antioxidant vitamin C were determined by venous occlusion plethysmography. The FBF response to ACh was significantly lower (~20%) in the short sleep (from 3.8±0.2 to 11.0±0.6 ml/100 ml tissue/min) compared with the normal sleep duration group (from 4.2±0.2 to 13.6±0.6 ml/100 ml tissue/min). L-NMMA significantly reduced (~25%) the FBF response to ACh in the normal sleep but not the short sleep group. Vitamin C markedly increased (~35%; P<0.05) the vasodilator response to ACh in short sleepers only. In summary, habitual short sleep duration worsens NO-mediated endothelium-dependent vasodilation in adults with elevated blood pressure. Furthermore, the sleep-related diminishment in endothelial vasodilator function is due, in part, to increased oxidative stress.

scholarly journals Associations of Urinary Phytoestrogen Concentrations with Sleep Disorders and Sleep Duration among Adults

Nutrients ◽  
2020 ◽  
Vol 12 (7) ◽  
pp. 2103 ◽  
Author(s):  
Jing Sun ◽  
Hong Jiang ◽  
Weijing Wang ◽  
Xue Dong ◽  
Dongfeng Zhang
Keyword(s):  
Sleep Disorders ◽  
Sleep Duration ◽  
Multinomial Logistic Regression ◽  
Cross Sectional Study ◽  
Current Evidence ◽  
Relative Risk Ratio ◽  
Cross Sectional ◽  
Short Sleep ◽  
Long Sleep ◽  
Normal Sleep

Current evidence on the relationship of phytoestrogens with sleep is limited and contradictory. In particular, studies on individual phytoestrogens and sleep have not been reported. Thus, this study aimed to appraise the associations of individual phytoestrogens with sleep disorders and sleep duration. This cross-sectional study comprising 4830 adults utilized data from the National Health and Nutrition Examination Survey 2005–2010. Phytoestrogens were tested in urine specimens. Sleep disorders and sleep duration were based on a self-reported doctor’s diagnosis and usual sleep duration. The main analyses utilized logistic and multinomial logistic regression models and a restricted cubic spline. In the fully adjusted model, compared with tertile 1 (lowest), the odds ratios (95% confidence intervals (CIs)) of sleep disorders for the highest tertile of urinary concentrations of enterolactone, enterodiol, and O-desmethylangolensin were 0.64 (0.41–1.00), 1.54 (1.07–2.21), and 1.89 (1.26–2.85), respectively. Linear inverse, approximatively linear positive, and inverted L-shaped concentration–response relationships were found between enterolactone, enterodiol, and O-desmethylangolensin and sleep disorders, respectively. Compared with normal sleep (7–8 h/night), the relative risk ratio (RRR) (95% CI) of very short sleep for enterolactone was 0.56 (0.36–0.86), and the RRR (95% CI) of long sleep risk for genistein was 0.62 (0.39–0.99). Furthermore, negative associations of genistein with sleep disorders and enterolactone with long sleep risk, as well as positive associations of enterodiol with both long and very short sleep, were observed in the stratified analysis by age or gender. Finally, a notable finding was that urinary O-desmethylangolensin concentration was positively related to sleep disorders in both females aged 40–59 years and non-Hispanic Whites but inversely associated with sleep disorders in both females aged 60 years or over and other Hispanics. Our findings suggested that enterolactone and genistein might be beneficial for preventing sleep disorders or non-normal sleep duration among adults, and enterodiol might be adverse toward this goal. However, the association of O-desmethylangolensin with sleep disorders might be discrepant in different races and females of different ages.

Insomnia and sleep state misperception: Clinical features, diagnosis, management and implications

2017 ◽  
Vol 41 (S1) ◽  
pp. s853-s853
Author(s):  
J. Isaac ◽  
C. Santos ◽  
A. Matos Pires
Keyword(s):  
Sleep Duration ◽  
Clinical Features ◽  
Mental Disease ◽  
Sleep Time ◽  
Sleep State ◽  
Short Sleep Duration ◽  
Short Sleep ◽  
Psychological Therapies ◽  
Normal Sleep ◽  
Competing Interest

BackgroundInsomnia is a highly prevalent complaint, largely associated with mental disease. Clinical evidence classifies insomnia in 2 subtypes: with sleep misperception (WSM) and without sleep misperception (wSM). That presents distinctive pathophysiologic pathways and different public health implications.ObjectivesDescribe the main differences between primary insomnia WSM and wSM regarding:– clinical features;– diagnosis;– management;– implications.MethodsWe conducted a systematic review. PubMed, Embase and PsycInfo were searched from 2000–2016. The reference lists of systematic reviews, narrative synthesis and some important articles were included. Following the inclusion criteria, we selected 25 studies from 59 articles.ResultsThe prevalence of sleep-state misperception in primary insomniacs (total sleep time > 6.5 h and sleep efficiency > 85%) is around 26%. Insomniacs with normal sleep duration showed a profile of high depression and anxiety and low ego strength, whereas insomniacs with short sleep duration showed a profile of a medical disorder.Cortical hyperarousal is higher in insomniacs and could be related to an alteration in sleep protection mechanisms. The sleep architecture was relatively normal for the WSM comparing with the group wSM. Risk of cardiometabolic, neurocognitive morbidity and mortality, and responses to treatment are different between these two insomnia phenotypes. Patients with short sleep duration may respond better to biological treatments, whereas insomnia with normal sleep duration may respond primarily to psychological therapies.ConclusionsThe clinical characteristics of patients with sleep-state misperception differed from those without this condition. Available research related to these conditions is expanding rapidly, but many questions remain unanswered.Disclosure of interestThe authors have not supplied their declaration of competing interest.

scholarly journals 0356 Associations Between Ethnicity, Socioeconomic Deprivation and Preschoolers’ Sleep Health in Aotearoa/New Zealand

SLEEP ◽  
2020 ◽  
Vol 43 (Supplement_1) ◽  
pp. A136-A136
Author(s):  
D Muller ◽  
S Paine ◽  
L J Wu ◽  
T L Signal
Keyword(s):  
New Zealand ◽  
Sleep Duration ◽  
Socioeconomic Position ◽  
Large Problem ◽  
Sleep Health ◽  
Individual Level ◽  
Short Sleep ◽  
Aotearoa New Zealand ◽  
Falling Asleep ◽  
Deprived Areas

Abstract Introduction In Aotearoa/New Zealand (NZ) ethnic and socioeconomic inequities exist in adult sleep health but less is known about relationships between ethnicity, socioeconomic position and sleep in early childhood. Methods Maternally-completed questionnaire data from a pregnancy-birth cohort were analysed cross-sectionally. Log-binomial regression models investigated independent associations between ethnicity, socioeconomic position and sleep of 340 Māori (Indigenous) and 570 non-Māori 3-4 year olds. Independent variables included child ethnicity, gender, area-level deprivation (NZDep quintiles; 5=most deprived) and individual-level deprivation (NZiDep scores 1-5; 5=most deprived). Dependent variables included typical weekday and weekend sleep duration (&lt;10hrs/10-13hrs), difference in week/weekend sleep duration (&gt;1hr/≤1hr) and midsleep time (≥1hr/&lt;1hr), problems falling asleep and problematic sleep patterns (no vs. moderate/large problem). Results Māori preschoolers were more likely to have short sleep (weekdays: PR=2.23, 95% CI 1.31-3.82; weekends: PR=2.04, 95% CI 1.24-3.36), week/weekend sleep duration difference &gt;1hr (PR=2.47, 95% CI 1.59-3.84), week/weekend midsleep difference ≥1hr (PR=2.38, 95% CI 1.30-4.36) and a moderate/large problem falling asleep (PR=1.43, 95% CI 1.00-2.06) than non-Māori preschoolers. Preschoolers living in most deprived areas were more likely to have short sleep on weekdays (NZDep quintile 4: PR=3.91, 95% CI 1.43-10.72; NZDep quintile 5: PR=4.14, 95% CI 1.54-11.12) and week/weekend sleep duration difference &gt;1hr (NZDep quintile 4: PR=2.34, 95% CI 1.23-4.43) than preschoolers in least deprived areas. Children with higher individual-level deprivation scores were more likely to have short sleep on weekends (NZiDep 5: PR=2.38, 95% CI 1.21-4.67) and a moderate/large problem falling asleep (NZiDep 3: PR=1.72, 95% CI 1.10-2.67) compared to children with lowest scores. Conclusion Ethnic and socioeconomic sleep health inequities exist as early as 3 years of age in NZ. Socio-political drivers of social and economic disadvantage experienced by Māori children and children from families who hold low socioeconomic position must be addressed to achieve equitable sleep health early in the lifecourse. Support Funding support was provided by Massey University New Zealand (Massey University Strategic Innovation Fund; Massey University Research Fund; and Massey University Doctoral Scholarship) and the Health Research Council of New Zealand (HRC 09/233, 08/547).

Sleep health in U.S. adults before and during the COVID-19 pandemic

2020 ◽  
Author(s):  
Garrett Hisler ◽  
Jean Twenge
Keyword(s):  
Sleep Duration ◽  
Representative Sample ◽  
The United States ◽  
Future Research ◽  
Racial Groups ◽  
Sleep Health ◽  
The Past ◽  
Falling Asleep ◽  
Interview Survey ◽  
Nationally Representative

Understanding how health has changed in response to the COVID-19 pandemic is critical to reducing and recovering from the pandemic. This study focused on how sleep health in the United States may have been impacted by the COVID-19 pandemic. Sleep duration and number of days in the past week with difficulty falling asleep, difficulty staying asleep, and not feeling rested in a nationally representative sample of U.S. adults collected before the COVID-19 outbreak (2018 National Health Interview Survey, n = 19,433) was compared to the same measures in a nationally representative sample of U.S. adults collected during the COVID-19 outbreak (2020 Luc.id, n = 2,059). Sleep duration was slightly shorter in 2020 than 2018 (d = -.05). Moreover, the prevalence of insufficient sleep duration (RR = 1.20) and the number of days with difficulty falling asleep (d = .54), difficulty staying asleep (d =.36), and not feeling rested (d = .14) was greater in 2020 than 2018. Twice as many people in 2020 reported experiencing at least one night of difficulty falling asleep (RR = 1.95) or staying asleep (RR = 1.75). Adults younger than 60 and those who belonged to an Asian racial group had larger differences than other age and racial groups between 2018 and 2020. Thus, sleep health in U.S. adults was worse in 2020 than in 2018, particularly in younger and Asian adults. Findings highlight sleep as target in future research and interventions seeking to understand and reduce the effects of the spread of COVID-19.

Abstract 2455: Short Sleep Duration is an Independent Predictor of Cardiovascular Events in Hypertensive Patients

Circulation ◽  
2008 ◽  
Vol 118 (suppl_18) ◽  
Author(s):  
Kazuo Eguchi ◽  
Thomas G Pickering ◽  
Joseph E Schwartz ◽  
Satoshi Hoshide ◽  
Joji Ishikawa ◽  
...  
Keyword(s):  
Sleep Duration ◽  
Short Duration ◽  
Cardiovascular Events ◽  
Sleep Time ◽  
Cvd Risk ◽  
Short Sleep Duration ◽  
Hypertensive Patients ◽  
Short Sleep ◽  
Multivariable Analyses ◽  
Normal Sleep

We aimed this study to test the hypothesis that short duration of sleep is independently associated with incident cardiovascular diseases (CVD) in hypertensive patients. We performed ambulatory BP monitoring (ABPM) in 1255 subjects with hypertension (mean age: 70.4 ± 9.9 years) and they were followed for an average of 50 ± 23 months. Short sleep duration was defined as <7.5 hrs (20 th percentile). Multivariable Cox hazard models predicting CVD events were used to estimate the adjusted hazard ratio (HR) and 95% CI for short sleep duration. A riser pattern was defined when average nighttime SBP exceeded daytime SBP. The end point was cardiovascular events: stroke, fatal or non-fatal myocardial infarction (MI), and sudden cardiac death. In multivariable analyses, short duration of sleep (<7.5 hrs) was associated with incident CVD (HR=1.68; 1.06 –2.66, P=.03). A synergistic interaction was observed between short sleep duration and the riser pattern (P=.089). When subjects were categorized on the basis of their sleep time and riser/non-riser patterns, the shorter sleep+riser group had a highest incidence of CVD among the 4 groups ( Figure ), and substantially and significantly higher incidence of CVD than the predominant normal sleep+non-riser group (HR=4.43; 2.09 –9.39, P<0.001), independent of covariates. Short duration of sleep is associated with incident CVD risk, and the combination of riser pattern and short duration of sleep that is most strongly predictive of future CVD, independent of ambulatory BP levels. Physicians should inquire about sleep duration in the risk assessment of hypertensive patients.

Abstract P183: Influence of Insufficient Sleep on Endothelial Fibrinolytic Capacity in Adults With Elevated Blood Pressure

Hypertension ◽  
2017 ◽  
Vol 70 (suppl_1) ◽  
Author(s):  
Anthony R Bain ◽  
Caitlin A Dow ◽  
Kyle J Diehl ◽  
Tyler D Bammert ◽  
Jared J Greiner ◽  
...  
Keyword(s):  
Sleep Duration ◽  
Tissue Type ◽  
Short Sleep Duration ◽  
Insufficient Sleep ◽  
Short Sleep ◽  
Type Plasminogen Activator ◽  
Increased Risk ◽  
Normal Sleep ◽  
Middle Aged Adults

The capacity of the endothelium to release tissue-type plasminogen activator (t-PA) is impaired in adults with elevated BP, leading to an increased risk of thrombotic events. Insufficient sleep is independently associated with elevated BP and impaired t-PA release. However, the compounded influence of insufficient sleep on t-PA release in adults with elevated BP is unknown. We tested the hypothesis that impairments in the capacity of the endothelium to release t-PA in adults with elevated BP is worse in those who sleep <7 h/night (short sleep duration) compared with those who sleep 7 to 9 h/night (normal sleep duration). We studied 38 sedentary, middle-aged adults: 10 with normal BP and normal nightly sleep duration (6M/4F; age: 55±2 yr; BP: 114/94±2/3 mmHg, sleep duration: 7.4±0.2 h); 14 with elevated BP and normal nightly sleep duration (8M/6F; 60±2 yr; 141/87±2/2 mmHg; 7.8±0.1 h); and 14 with elevated BP and short nightly sleep duration (10M/4F; 57±2 yr; 139/85±2/2 mmHg; 6.1±0.2 h). All subjects were free of overt metabolic and coronary disease. Net endothelial release of t-PA was determined, in vivo, in response to intra-brachial infusions of bradykinin (BK: 125-500 ng/min) and sodium nitroprusside (SNP: 2.0-8.0 μg/min). In the normal sleep groups, as expected, endothelial t-PA release in response to BK was significantly blunted (~30%) in the adults with elevated BP (from -1.2±0.8 to 50.2±4.8 ng/100mL tissue/min) compared with normal BP (from 0.9±3.4 to 73.0±8.0 ng/100mL tissue/min); and total t-PA release (area under the BK curve) was ~25% lower (p<0.05) in the adults with elevated (307±33 ng/100mL tissue) vs. normal (396±27 ng/100mL tissue) BP. Importantly, net endothelial release rate (from -1.5±1.0 to 40.6±4.3 ng/100mL tissue/min) and total amount of t-PA released (222±28 ng/100mL tissue) in response to BK were markedly lower (~25% and 30%, respectively, P<0.05) in the elevated BP and short sleep duration group compared with the elevated BP and normal sleep duration group. In the elevated BP population, sleep duration was positively correlated with total t-PA release (r=0.46, P<0.05). There was no effect of SNP on t-PA release in any group. In summary, insufficient sleep is associated with exacerbated impairments in t-PA release in adults with elevated BP.

Abstract 28: Regular Aerobic Exercise Counteracts Endothelial Vasomotor Dysfunction Associated With Insufficient Sleep

Circulation ◽  
2020 ◽  
Vol 141 (Suppl_1) ◽  
Author(s):  
Kelly A Stockelman ◽  
Anthony R Bain ◽  
Caitlin A Dow ◽  
Jared J Greiner ◽  
Brian L Stauffer ◽  
...  
Keyword(s):  
Exercise Training ◽  
Aerobic Exercise ◽  
Sleep Duration ◽  
Middle Aged ◽  
Short Sleep Duration ◽  
Insufficient Sleep ◽  
Short Sleep ◽  
Vasomotor Regulation ◽  
Normal Sleep ◽  
Middle Aged Adults

Insufficient sleep, defined as chronic short sleep duration (<7 h/night), is an independent risk factor for cardiovascular disease (CVD). We have previously demonstrated that insufficient sleep is associated with reduced endothelium-dependent vasodilation and enhanced endothelin (ET)-1-mediated vasoconstrictor tone. Impaired endothelial vasomotor regulation is thought to contribute mechanistically to the increased risk of atherosclerotic vascular disease incurred with chronic insufficient sleep. Regular aerobic exercise is an effective lifestyle strategy for improving endothelial function and, in turn reducing cardiovascular risk. It is currently unknown if regular aerobic exercise can counteract the negative impact of insufficient sleep on endothelial vasomotor regulation. We tested the hypotheses that regular aerobic exercise would: 1) improve endothelial vasodilation; and 2) decrease ET-1-mediated vasoconstrictor tone in middle-aged adults who chronically sleep less than 7 h/night. We studied 36 healthy, middle-aged adults: 16 with normal sleep duration (10M/6F; age: 57±2 yr; sleep duration: 7.4±0.1 h/night) and 20 with short sleep duration (11M/9F; 56±1 yr; sleep duration: 6.2±0.1 h/night). The 20 short sleepers completed a 3-month aerobic exercise training intervention. Forearm blood flow (FBF; plethysmography) was determined in response to intra-arterial doses of acetylcholine (ACh), sodium nitroprusside (SNP), BQ-123 (ET A receptor antagonist) and ACh + BQ-123 in both groups and after the exercise intervention in the short sleepers. As expected, forearm vasodilator responses to ACh were lower (20%; P<0.05) in the short (from 4.2±0.2 to 10.5±0.6 mL/100 mL tissue/min) vs normal (4.2±0.2 to 12.7±0.6 mL/100 mL tissue/min) sleepers. FBF responses to SNP were comparable between the groups. In response to BQ-123, short sleep group had a greater increase in resting FBF than normal sleep group (~25% vs ~8%; P< 0.05). ACh+BQ-123 resulted in an ~25% increase in the ACh-vasodilation in the short sleep group only. After exercise training, although nightly sleep duration was not affected (6.4±0.1 h/night), ACh-mediated vasodilation was ~20% higher (P<0.05), ET-1-mediated vasoconstriction was ~90% lower (P<0.05) and vasodilator response to ACh was not significantly increased with ET A receptor blockade. These results indicate that regular aerobic exercise can reverse the negative influence of insufficient sleep on endothelial vasomotor function, independent of changes in nightly sleep duration.

scholarly journals The association between sleep duration and excess body weight of the American adult population: a cross-sectional study of the national health and nutrition examination survey 2015–2016

2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Qing Li
Keyword(s):  
Sleep Duration ◽  
National Health ◽  
Subgroup Analysis ◽  
Overweight And Obesity ◽  
Nutrition Examination Survey ◽  
Short Sleep ◽  
Health And Nutrition ◽  
Long Sleep ◽  
Prevalence Of Obesity ◽  
Normal Sleep

Abstract Background We intend to explore whether sleep duration is associated with overweight and obesity among the adult American population. Furthermore, we stratified the study population by age and sex in the subgroup analysis to investigate the potential disparities between adults and older adults, and men and women. Methods In total, 2459 individuals from the 2015–2016 National Health and Nutrition Examination Survey cycle were included for analysis in this study. Sleep duration was assessed by the Sleep Disorders Questionnaire. Classification of the short-sleep, normal-sleep, and long-sleep group was based on the recommendation of the National Sleep Foundation. Bodyweight was measured during the physical examination. Multivariate logistic regression models were implemented. Results We observed a significantly higher overweight incidence in the short-sleep group compared to the normal-sleep group (OR = 1.825, 95%CI: 1.251–2.661, P = 0.004). Short-sleep (OR = 1.832, 95%CI: 1.215–2.762, P = 0.007) duration and long-sleep duration (OR = 1.370, 95%CI: 1.043–1.800, P = 0.027) were associated with higher prevalence of obesity. When stratified by age, short-sleep also increased the overweight and obese incidence 1.951 and 1.475 times in the adult group. In the sex-stratified subgroup analysis, the short-sleep group showed 2.49 times higher overweight incidence among females. The prevalence of obesity was 2.59 times higher in the short-sleep group and 1.698 times higher in the long-sleep group in the female population. Conclusions Sleep duration is associated with the occurrence of overweight and obesity, with sleep duration less than 7 h increase the overweight and obesity rate nearly 2 folds comparing to sleep 7–9 h.

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