Literature Riview : Penerapan Kompres Air Hangat Untuk Menurunkan Suhu Tubuh Pada Pasien Demam Thypoid

Typhoid fever is a systemic infection caused by salmonella enterica bacteria, especially its derivative variants, namely salmonella typhi, paratyphi A, paratyphi B, paratyphi C. These germs attack the digestive tract, especially in the stomach and intestines, nursing problems that often occur in patients with typhoid fever, namely hyperthermia. Hyperthermia is a condition in which an individual has an increase in body temperature above 37.8 C parrectal due to external factors. A warm compress is a procedure used to improve control of body heat loss through evaporation and conduction which is usually performed on patients who have a high fever. The purpose of scientific papers is to see an overview of the application of warm compresses to reduce body temperature in typhoid fever patients. The method is carried out by searching several research journals entitled about the application of warm water compresses to reduce body temperature in typhoid fever patients. The results obtained after the action of warm water compresses, body temperature decreased within normal limits. The conclusion of this scientific paper is that the action of warm water compresses can reduce body temperature in patients with typhoid fever. Suggestions for nurses are expected to apply warm compresses to reduce body temperature in typhoid fever patients.Keywords: Key words: typhoid fever, hyperthermia, warm water compress AbstrakDemam typhoid adalah infeksi sistemik yang di sebabkan oleh bakteri salmonella enterika, khususnya varian-varian turunannya, yaitu salmonella typhi, paratyphi A, paratyphi B, paratyphi C. Kuman-kuman tersebut menyerang saluran pencernaan, terutatama di perut dan usus masalah keperawatan yang sering terjadi pada pasien demam tifoid yaitu hipertermia . Hipertermi adalah suatu keadaan dimana seorang individu mengalami peningkatan suhu tubuh di atas 37,8⁰C parrektal karena factor eksternal. Kompres air hangat adalah prosedur yang di gunakan untuk meningkatkan control kehilangan panas tubuh melalui evaporasi dan konduksi yang biasanya di lakukan pada pasien yang mengalami demam tinggi. Tujuan dari karya tulis ilmiah adalah untuk mengetahui gambaran tentang penerapan kompres air hangat untuk menurunkan suhu tubuh pada pasien demam thypoid. Metode yang dilakukan dengan mencari beberapa jurnal penelitian berjudul tentang penerapan kompres air hangat untuk menurunkan suhu tubuh pada pasien demam thypoid. Hasil yang didapatkan setelah dilakukan tindakan kompres air hangat suhu tubuh mengalami penurunan dalam batas normal. Kesimpulan karya tulis ilmiah ini bahwa tindakan kompres air hangat dapat menurunkan suhu tubuh pada pasien demam thypoid. Saran bagi perawat diharapkan dapat menerapkan tindakan kompres air hangat untuk menurunkan suhu tubuh pada pasien demam thypoid.Kata kunci: Demam Thypoid, Hipertermi, Kompres air hangat

Garment-Integrated Thermoelectric Generator Arrays for Wearable Body Heat Harvesting

Wearable thermoelectric generator arrays have the potential to use waste body heat to power on-body sensors and create, for example, self-powered health monitoring systems. In this work, we demonstrate that a surface coating of a conducting polymer poly(3,4-ethylenedioxythiophene) (PEDOT-Cl), created on one face of a wool felt using a chemical vapor deposition method was able to manifest a Seebeck voltage when subjected to a temperature gradient. The wool felt devices can produce voltage outputs of up to 120 mV when measured on a human body. Herein, we present a strategy to create arrays of polymer-coated fabric thermopiles and to integrate such arrays into familiar garments that could become a part of a consumer’s daily wardrobe. Using wool felt as the substrate fabric onto which the conducting polymer coating is created allowed for a higher mass loading of the polymer on the fabric surface and shorter thermoelectric legs, as compared to our previous iteration. Six or eight of these PEDOT-Cl coated wool felt swatches were sewed onto a backing/support fabric and interconnected with silver threads to create a coupled array, which was then patched onto the collar of a commercial three-quarter zip jacket. The observed power output from a six-leg array while worn by a healthy person at room temperature (ΔT = 15 °C) was 2 µW, which is the highest value currently reported for a polymer thermoelectric device measured at room temperature.

Modeling and Simulation of Human Body Heat Transfer System Based on Air Space Values in 3D Clothing Model

Comfort can be considered as subjective feeling, which could be affected by the external ambient, by the physical activity, and by clothing. Considering the human body heat transfer system, it mainly depends on various parameters including clothing materials, external and internal environment, etc. The purpose of the current paper is to study and establish a quantitative relationship between one of the clothing parameters, ease allowance (air gap values) and the heat transfer through the human body to clothing materials and then to the environment. The study considered clothing which is integrated with the 3D ease allowance from the anthropometric and morphological data. Such incorporating of the clothing’s 3D ease control was essential to properly manage the air space between the body and the proposed clothing thermal regulation model. In the context of thermal comfort, a clothing system consisting of the human body, an ease allowance under clothing, a layer of textile materials, and a peripheral layer adjacent to the textile material was used. For the complete system, the heat transfer from the skin to the environment, which is influenced by thermoregulation of the human body, air gap, tissue, and environmental conditions were also considered. To model and predict the heat transfer between the human body and the temperature of skin and clothes, a 3D adaptive garment which could be adjusted with ease allowance was used. In the paper, a thermoregulatory model was developed and proposed to predict the temperature and heat within clothing material, skin, and air space. Based on the result, in general the main difference in the temperature of clothing and skin from segment to segment is due to the uneven distribution of air layers under the clothing.

Dysfunctional TRPM8 signalling in the vascular response to environmental cold in ageing

Ageing is associated with increased vulnerability to environmental cold exposure. Previously, we identified the role of the cold-sensitive transient receptor potential (TRP) A1, M8 receptors as vascular cold sensors in mouse skin. We hypothesised that this dynamic cold-sensor system may become dysfunctional in ageing. We show that behavioural and vascular responses to skin local environmental cooling are impaired with even moderate ageing, with reduced TRPM8 gene/protein expression especially. Pharmacological blockade of the residual TRPA1/TRPM8 component substantially diminished the response in aged, compared with young mice. This implies the reliance of the already reduced cold-induced vascular response in ageing mice on remaining TRP receptor activity. Moreover, sympathetic-induced vasoconstriction was reduced with downregulation of the α2c adrenoceptor expression in ageing. The cold-induced vascular response is important for sensing cold and retaining body heat and health. These findings reveal that cold sensors, essential for this neurovascular pathway, decline as ageing onsets.

Integrated cooling (i-Cool) textile of heat conduction and sweat transportation for personal perspiration management

Perspiration evaporation plays an indispensable role in human body heat dissipation. However, conventional textiles tend to focus on sweat removal and pay little attention to the basic thermoregulation function of sweat, showing limited evaporation ability and cooling efficiency in moderate/profuse perspiration scenarios. Here, we propose an integrated cooling (i-Cool) textile with unique functional structure design for personal perspiration management. By integrating heat conductive pathways and water transport channels decently, i-Cool exhibits enhanced evaporation ability and high sweat evaporative cooling efficiency, not merely liquid sweat wicking function. In the steady-state evaporation test, compared to cotton, up to over 100% reduction in water mass gain ratio, and 3 times higher skin power density increment for every unit of sweat evaporation are demonstrated. Besides, i-Cool shows about 3 °C cooling effect with greatly reduced sweat consumption than cotton in the artificial sweating skin test. The practical application feasibility of i-Cool design principles is well validated based on commercial fabrics. Owing to its exceptional personal perspiration management performance, we expect the i-Cool concept can provide promising design guidelines for next-generation perspiration management textiles.

Effect of chitosan on blood profile, inflammatory cytokines by activating TLR4/NF-κB signaling pathway in intestine of heat stressed mice

Heat stress can significantly affect the immune function of the animal body. Heat stress stimulates oxidative stress in intestinal tissue and suppresses the immune responses of mice. The protecting effects of chitosan on heat stress induced colitis have not been reported. Therefore, the aim of this study was to investigate the protective effects of chitosan on immune function in heat stressed mice. Mice were exposed to heat stress (40 °C per day for 4 h) for 14 consecutive days. The mice (C57BL/6J), were randomly divided into three groups including: control group, heat stress, Chitosan group (LD: group 300 mg/kg/day, MD: 600 mg/kg/day, HD: 1000 mg/kg/day). The results showed that tissue histology was improved in chitosan groups than heat stress group. The current study showed that the mice with oral administration of chitosan groups had improved body performance as compared with the heat stress group. The results also showed that in chitosan treated groups the production of HSP70, TLR4, p65, TNF-α, and IL-10 was suppressed on day 1, 7, and 14 as compared to the heat stress group. In addition Claudin-2, and Occludin mRNA levels were upregulated in mice receiving chitosan on day 1, 7, and 14 of heat stress. Furthermore, the IL-6, IL-10, and TNF-α plasma levels were down-regulated on day 1, 7, and 14 of heat stress in mice receiving the oral administration of chitosan. In conclusion, the results showed that chitosan has an anti-inflammatory ability to tolerate hot environmental conditions.